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Effects of cytokine treatment on angiotensin II type 1A receptor transcription and splicing in rat cardiac fibroblasts

American journal of physiology. Heart and circulatory physiology, 2005-09, Vol.289 (3), p.H1176-H1183

2005

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Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects

American journal of physiology. Heart and circulatory physiology, 2005-12, Vol.289 (6), p.H2356-H2363

2005

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IL-1β and TNF-α upregulate angiotensin II type 1 (AT1) receptors on cardiac fibroblasts and are associated with increased AT1 density in the post-MI heart

Journal of molecular and cellular cardiology, 2005-03, Vol.38 (3), p.505-515

2005

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Tumor necrosis factor-alpha-induced AT(1) receptor upregulation enhances angiotensin II-mediated cardiac fibroblast responses that favor fibrosis

Circulation research, 2002-12, Vol.91 (12), p.1119

2002

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Tumor Necrosis Factor-α–Induced AT1 Receptor Upregulation Enhances Angiotensin II–Mediated Cardiac Fibroblast Responses That Favor Fibrosis

Circulation research, 2002-12, Vol.91 (12), p.1119-1126

2002

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Transcription Factor NF-κB Is Necessary for Up-regulation of Type 1 Angiotensin II Receptor mRNA in Rat Cardiac Fibroblasts Treated with Tumor Necrosis Factor-α or Interleukin-1β

The Journal of biological chemistry, 2002-02, Vol.277 (8), p.5719-5724

2002

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Tumor Necrosis Factor-α Upregulates Angiotensin II Type 1 Receptors on Cardiac Fibroblasts

Circulation research, 1999-08, Vol.85 (3), p.272-279

1999

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Etanercept or intravenous immunoglobulin attenuates expression of genes involved in post-myocardial infarction remodeling

Cardiovascular research, 2005-07, Vol.67 (1), p.106-115

2005

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Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects

American journal of physiology. Heart and circulatory physiology, 2005-12, Vol.58 (6), p.H2356-H2363

2005

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Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects

American Journal of Physiology: Cell Physiology, 2005-12, Vol.289 (6), p.H2356-H2363

2005

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IL-1beta and TNF-alpha upregulate angiotensin II type 1 (AT1) receptors on cardiac fibroblasts and are associated with increased AT1 density in the post-MI heart

Journal of molecular and cellular cardiology, 2005-03, Vol.38 (3), p.505-515

2005

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Effects of cytokine treatment on angiotensin II type 1A receptor transcription and splicing in rat cardiac fibroblasts

American journal of physiology. Heart and circulatory physiology, 2005-09, Vol.58 (3), p.H1176-H1183

2005

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IL-1β and TNF-α upregulate angiotensin II type 1 (AT 1) receptors on cardiac fibroblasts and are associated with increased AT 1 density in the post-MI heart

Journal of molecular and cellular cardiology, 2005, Vol.38 (3), p.505-515

2005

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Tumor Necrosis Factor-α–Induced AT 1 Receptor Upregulation Enhances Angiotensin II–Mediated Cardiac Fibroblast Responses That Favor Fibrosis

Circulation research, 2002-12, Vol.91 (12), p.1119-1126

2002

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Transcription Factor NF-ÎºB Is Necessary for Up-regulation of Type 1 Angiotensin II Receptor mRNA in Rat Cardiac Fibroblasts Treated with Tumor Necrosis Factor-Î± or Interleukin-1Î

The Journal of biological chemistry, 2002-02, Vol.277 (8), p.5719

2002

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Temporal Regulation of Shaker- and Shab-Like Potassium Channel Gene Expression in Single Embryonic Spinal Neurons during K+ Current Development

The Journal of neuroscience, 1996-05, Vol.16 (10), p.3287-3295

1996

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Sustained upregulation in embryonic spinal neurons of a Kv3.1 potassium channel gene encoding a delayed rectifier current

Journal of neurobiology, 2000-02, Vol.42 (3), p.347-356

2000

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Growth factor regulation of type 1 angiotensin II receptor (AT 1R) expression in cardiac fibroblasts

Journal of cardiac failure, 1998-09, Vol.4 (3), p.8-8

1998

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Changes in the number, function, and regulation of nicotinic acetylcholine receptors during neuronal development

Developmental biology, 1989-10, Vol.135 (2), p.326-339

1989

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Temporal Regulation of Shaker- andShab-Like Potassium Channel Gene Expression in Single Embryonic Spinal Neurons during K+ Current Development

The Journal of neuroscience, 1996-05, Vol.16 (10), p.3287-3295

1996

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VIP modulates neuronal nicotinic acetylcholine receptor function by a cyclic AMP-dependent mechanism

The Journal of neuroscience, 1994-06, Vol.14 (6), p.3540-3547

1994

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Regulation of the neuronal nicotinic acetylcholine receptor function

Gurantz, Devorah

1991

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Hypercholeresis induced by unconjugated bile acid infusion correlates with recovery in bile of unconjugated bile acids

Hepatology (Baltimore, Md.), 1991-03, Vol.13 (3), p.540-550

1991

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Modulation by albumin of neuronal cholinergic sensitivity

Molecular pharmacology, 1993-05, Vol.43 (5), p.807-812

1993

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Inhibition of Na+/H+ exchange in the rat is associated with decreased ursodeoxycholate hypercholeresis, decreased secretion of unconjugated ursodeoxycholate, and increased ursodeoxycholate glucuronidation

Gastroenterology (New York, N.Y. 1943), 1988-08, Vol.95 (2), p.454-463

1988

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Effects of cytokine treatment on angiotensin II type 1A receptor transcription and splicing in rat cardiac fibroblasts

Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects

IL-1β and TNF-α upregulate angiotensin II type 1 (AT1) receptors on cardiac fibroblasts and are associated with increased AT1 density in the post-MI heart

Tumor necrosis factor-alpha-induced AT(1) receptor upregulation enhances angiotensin II-mediated cardiac fibroblast responses that favor fibrosis

Tumor Necrosis Factor-α–Induced AT1 Receptor Upregulation Enhances Angiotensin II–Mediated Cardiac Fibroblast Responses That Favor Fibrosis

Transcription Factor NF-κB Is Necessary for Up-regulation of Type 1 Angiotensin II Receptor mRNA in Rat Cardiac Fibroblasts Treated with Tumor Necrosis Factor-α or Interleukin-1β

Tumor Necrosis Factor-α Upregulates Angiotensin II Type 1 Receptors on Cardiac Fibroblasts

Etanercept or intravenous immunoglobulin attenuates expression of genes involved in post-myocardial infarction remodeling

Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects

Angiotensin-(1-7) binds to specific receptors on cardiac fibroblasts to initiate antifibrotic and antitrophic effects

IL-1beta and TNF-alpha upregulate angiotensin II type 1 (AT1) receptors on cardiac fibroblasts and are associated with increased AT1 density in the post-MI heart

Effects of cytokine treatment on angiotensin II type 1A receptor transcription and splicing in rat cardiac fibroblasts

IL-1β and TNF-α upregulate angiotensin II type 1 (AT 1) receptors on cardiac fibroblasts and are associated with increased AT 1 density in the post-MI heart

Tumor Necrosis Factor-α–Induced AT 1 Receptor Upregulation Enhances Angiotensin II–Mediated Cardiac Fibroblast Responses That Favor Fibrosis

Transcription Factor NF-ÎºB Is Necessary for Up-regulation of Type 1 Angiotensin II Receptor mRNA in Rat Cardiac Fibroblasts Treated with Tumor Necrosis Factor-Î± or Interleukin-1Î

Temporal Regulation of Shaker- and Shab-Like Potassium Channel Gene Expression in Single Embryonic Spinal Neurons during K+ Current Development

Sustained upregulation in embryonic spinal neurons of a Kv3.1 potassium channel gene encoding a delayed rectifier current

Growth factor regulation of type 1 angiotensin II receptor (AT 1R) expression in cardiac fibroblasts

Changes in the number, function, and regulation of nicotinic acetylcholine receptors during neuronal development

Temporal Regulation of Shaker- andShab-Like Potassium Channel Gene Expression in Single Embryonic Spinal Neurons during K+ Current Development

VIP modulates neuronal nicotinic acetylcholine receptor function by a cyclic AMP-dependent mechanism

Regulation of the neuronal nicotinic acetylcholine receptor function

Hypercholeresis induced by unconjugated bile acid infusion correlates with recovery in bile of unconjugated bile acids

Modulation by albumin of neuronal cholinergic sensitivity

Inhibition of Na+/H+ exchange in the rat is associated with decreased ursodeoxycholate hypercholeresis, decreased secretion of unconjugated ursodeoxycholate, and increased ursodeoxycholate glucuronidation

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