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An Epithelial―Mesenchymal Transition Gene Signature Predicts Resistance to EGFR and PI3K Inhibitors and Identifies Axl as a Therapeutic Target for Overcoming EGFR Inhibitor Resistance

Clinical cancer research, 2013-01, Vol.19 (1), p.279-290

2013

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Inhibition of nonsense-mediated decay rescues p53β/γ isoform expression and activates the p53 pathway in MDM2-overexpressing and select p53-mutant cancers

The Journal of biological chemistry, 2021-11, Vol.297 (5), p.101163-101163, Article 101163

2021

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RAD50 Expression Is Associated with Poor Clinical Outcomes after Radiotherapy for Resected Non-small Cell Lung Cancer

Clinical cancer research, 2018-01, Vol.24 (2), p.341-350

2018

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An alternative branch of the nonsense-mediated decay pathway

The EMBO journal, 2007-04, Vol.26 (7), p.1820-1830

2007

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Nonsense Codons Trigger an RNA Partitioning Shift

The Journal of biological chemistry, 2009-02, Vol.284 (7), p.4062-4072

2009

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RNA splicing promotes translation and RNA surveillance

Nature structural & molecular biology, 2005-09, Vol.12 (9), p.801-809

2005

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Boundary-independent polar nonsense-mediated decay

EMBO reports, 2002-03, Vol.3 (3), p.274-279

2002

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T-cell receptor sequences that elicit strong down-regulation of premature termination codon-bearing transcripts

The EMBO journal, 2002-01, Vol.21 (1-2), p.125-134

2002

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Co-occurring genomic alterations define major subsets of KRAS-mutant lung adenocarcinoma with distinct biology, immune profiles, and therapeutic vulnerabilities

Cancer discovery, 2015-08, Vol.5 (8), p.860-877

2015

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Enhanced Vulnerability of LKB1-Deficient NSCLC to Disruption of ATP Pools and Redox Homeostasis by 8-Cl-Ado

Molecular cancer research, 2022-02, Vol.20 (2), p.280-292

2022

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Abstract 1719: Superior efficacy of neoadjuvant compared to adjuvant immune checkpoint blockade in non-small cell lung cancer

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.1719-1719

2018

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Reprogramming of cell signaling in response to MEK inhibition in non-small cell cancer (NSCLC)

Journal of clinical oncology, 2016-05, Vol.34 (15_suppl), p.e23222-e23222

2016

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KDR Amplification Is Associated with VEGF-Induced Activation of the mTOR and Invasion Pathways but does not Predict Clinical Benefit to the VEGFR TKI Vandetanib

Clinical cancer research, 2016-04, Vol.22 (8), p.1940-1950

2016

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Abstract 5480: NSCLC cells with LKB1 mutation are vulnerable to energetic and oxidative stresses induced by 8-Chloroadenosine

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19_Supplement), p.5480-5480

2014

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Abstract 2118: Nonsense-mediated decay regulates the expression of tumor suppressor transcripts and cancer pathways in non-small cell lung cancer cell lines

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.2118-2118

2015

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Abstract 5733: Targeting nonsense-mediated decay restores p53 function in HPV-associated head and neck cancers

Cancer research (Chicago, Ill.), 2022-06, Vol.82 (12_Supplement), p.5733-5733

2022

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Abstract 1589: LKB1 overexpression regulates DNA repair pathway and sensitivity to radiation in LKB1 mutant non-small cell lung cancer cell lines

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.1589-1589

2013

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Abstract 374: KDR amplification in NSCLC is associated with sensitivity to VEGFR tyrosine kinase inhibitors

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.374-374

2013

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Use of proteomic analysis of LKB1/AMPK/mTOR pathways to identify IGF-1R pathway upregulation with LKB1 loss or mTOR inhibition in NSCLC: Implications for targeted combinations

Journal of clinical oncology, 2012-05, Vol.30 (15_suppl), p.10612-10612

2012

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Abstract 968: Co-occurring genomic alterations define major subsets of KRAS -mutant lung adenocarcinoma (LUAC) with distinct biology and therapeutic vulnerabilities

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.968-968

2015

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Abstract 2498: Proteomic analysis reveals Src/Stat and EGFR/MAPK pathways as potential mechanism of resistance to PI3K inhibitors in lung cancer

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.2498-2498

2013

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Abstract 1045: Proteomic analysis of effects of MEK inhibition with BAY86-9766 on LKB1/AMPK and mTOR pathway in lung cancer cell lines

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.1045-1045

2013

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Nonsense Codons Trigger an RNA Partitioning ShiftS

The Journal of biological chemistry, 2009-02, Vol.284 (7), p.4062-4072

2009

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Characterization of ribonuclease P isolated from rat liver cytosol

Archives of biochemistry and biophysics, 1992-07, Vol.296 (1), p.264-270

1992

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Multiple, large deletions in rat mitochondrial DNA: evidence for a major hot spot

Mutation research, 1996-01, Vol.349 (1), p.95-107

1996

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An Epithelial―Mesenchymal Transition Gene Signature Predicts Resistance to EGFR and PI3K Inhibitors and Identifies Axl as a Therapeutic Target for Overcoming EGFR Inhibitor Resistance

Inhibition of nonsense-mediated decay rescues p53β/γ isoform expression and activates the p53 pathway in MDM2-overexpressing and select p53-mutant cancers

RAD50 Expression Is Associated with Poor Clinical Outcomes after Radiotherapy for Resected Non-small Cell Lung Cancer

An alternative branch of the nonsense-mediated decay pathway

Nonsense Codons Trigger an RNA Partitioning Shift

RNA splicing promotes translation and RNA surveillance

Boundary-independent polar nonsense-mediated decay

T-cell receptor sequences that elicit strong down-regulation of premature termination codon-bearing transcripts

Co-occurring genomic alterations define major subsets of KRAS-mutant lung adenocarcinoma with distinct biology, immune profiles, and therapeutic vulnerabilities

Enhanced Vulnerability of LKB1-Deficient NSCLC to Disruption of ATP Pools and Redox Homeostasis by 8-Cl-Ado

Abstract 1719: Superior efficacy of neoadjuvant compared to adjuvant immune checkpoint blockade in non-small cell lung cancer

Reprogramming of cell signaling in response to MEK inhibition in non-small cell cancer (NSCLC)

KDR Amplification Is Associated with VEGF-Induced Activation of the mTOR and Invasion Pathways but does not Predict Clinical Benefit to the VEGFR TKI Vandetanib

Abstract 5480: NSCLC cells with LKB1 mutation are vulnerable to energetic and oxidative stresses induced by 8-Chloroadenosine

Abstract 2118: Nonsense-mediated decay regulates the expression of tumor suppressor transcripts and cancer pathways in non-small cell lung cancer cell lines

Abstract 5733: Targeting nonsense-mediated decay restores p53 function in HPV-associated head and neck cancers

Abstract 1589: LKB1 overexpression regulates DNA repair pathway and sensitivity to radiation in LKB1 mutant non-small cell lung cancer cell lines

Abstract 374: KDR amplification in NSCLC is associated with sensitivity to VEGFR tyrosine kinase inhibitors

Use of proteomic analysis of LKB1/AMPK/mTOR pathways to identify IGF-1R pathway upregulation with LKB1 loss or mTOR inhibition in NSCLC: Implications for targeted combinations

Abstract 968: Co-occurring genomic alterations define major subsets of KRAS -mutant lung adenocarcinoma (LUAC) with distinct biology and therapeutic vulnerabilities

Abstract 2498: Proteomic analysis reveals Src/Stat and EGFR/MAPK pathways as potential mechanism of resistance to PI3K inhibitors in lung cancer

Abstract 1045: Proteomic analysis of effects of MEK inhibition with BAY86-9766 on LKB1/AMPK and mTOR pathway in lung cancer cell lines

Nonsense Codons Trigger an RNA Partitioning ShiftS

Characterization of ribonuclease P isolated from rat liver cytosol

Multiple, large deletions in rat mitochondrial DNA: evidence for a major hot spot

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