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Connexins and gap junctions in the EDHF phenomenon and conducted vasomotor responses

Pflügers Archiv, 2010-05, Vol.459 (6), p.897-914

2010

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Endothelium‐dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?

British journal of pharmacology, 2004-03, Vol.141 (6), p.881-903

Griffith, Tudor M

2004

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Ascorbic acid and tetrahydrobiopterin potentiate the EDHF phenomenon by generating hydrogen peroxide

Cardiovascular research, 2009-11, Vol.84 (2), p.218-226

2009

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cAMP Facilitates EDHF-Type Relaxations in Conduit Arteries by Enhancing Electrotonic Conduction via Gap Junctions

Proceedings of the National Academy of Sciences - PNAS, 2002-04, Vol.99 (9), p.6392-6397

2002

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Gap junctional communication underpins EDHF-type relaxations evoked by ACh in the rat hepatic artery

American journal of physiology. Heart and circulatory physiology, 2001-06, Vol.280 (6), p.H2441-H2450

2001

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5-Methyltetrahydrofolate and Tetrahydrobiopterin Can Modulate Electrotonically Mediated Endothelium-Dependent Vascular Relaxation

Proceedings of the National Academy of Sciences - PNAS, 2005-05, Vol.102 (19), p.7008-7013

2005

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Distinct hyperpolarizing and relaxant roles for gap junctions and endothelium-derived H2O2 in NO-independent relaxations of rabbit arteries

Proceedings of the National Academy of Sciences - PNAS, 2003-12, Vol.100 (25), p.15212-15217

2003

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The effect of inorganic arsenic on endothelium-dependent relaxation: Role of NADPH oxidase and hydrogen peroxide

Toxicology (Amsterdam), 2013-04, Vol.306, p.50-58

2013

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Effects of connexin‐mimetic peptides on gap junction functionality and connexin expression in cultured vascular cells

British journal of pharmacology, 2005-03, Vol.144 (5), p.617-627

2005

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Gap junction-dependent and -independent EDHF-type relaxations may involve smooth muscle cAMP accumulation

American journal of physiology. Heart and circulatory physiology, 2002-04, Vol.282 (4), p.H1548-H1555

2002

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Which Connexins Connect?

Circulation research, 2007-12, Vol.101 (12), p.1219-1221

Griffith, Tudor M

2007

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Deterministic nonlinear features of cutaneous perfusion are lost in diabetic subjects with neuropathy

Microvascular research, 2011-07, Vol.82 (1), p.42-51

2011

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Central role of intracellular calcium stores in acute flow‐ and agonist‐evoked endothelial nitric oxide release

British journal of pharmacology, 1997-09, Vol.122 (1), p.117-125

1997

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Connexin‐mimetic peptides dissociate electrotonic EDHF‐type signalling via myoendothelial and smooth muscle gap junctions in the rabbit iliac artery

British journal of pharmacology, 2005-01, Vol.144 (1), p.108-114

2005

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Analysis of effects of connexin-mimetic peptides in rat mesenteric small arteries

American journal of physiology. Heart and circulatory physiology, 2006-07, Vol.291 (1), p.H357-H367

2006

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The obligatory link: role of gap junctional communication in endothelium-dependent smooth muscle hyperpolarization

Pharmacological research, 2004-06, Vol.49 (6), p.551-564

2004

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Attenuated store-operated Ca2+ entry underpins the dual inhibition of nitric oxide and EDHF-type relaxations by iodinated contrast media

Cardiovascular research, 2009-12, Vol.84 (3), p.470-478

2009

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Inhibition of the gap junctional component of endothelium‐dependent relaxations in rabbit iliac artery by 18‐α glycyrrhetinic acid

British journal of pharmacology, 1998-09, Vol.125 (1), p.1-3

1998

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Endothelial control of vascular tone by nitric oxide and gap junctions: a haemodynamic perspective

Biorheology (Oxford), 2002, Vol.39 (3-4), p.307

Griffith, Tudor M

2002

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Nitric Oxide-Independent Relaxations to Acetylcholine and A23187 Involve Different Routes of Heterocellular Communication: Role of Gap Junctions and Phospholipase A2

Circulation research, 1999-01, Vol.84 (1), p.53-63

1999

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Endogenous Nitric Oxide Synthesis Differentially Modulates Pressure-Flow and Pressure-Conductance Relationships in the Internal and External Carotid Artery Circulations of the Rat

Neurologia medico-chirurgica, 2002, Vol.42(12), pp.527-535

2002

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Essential role of gap junctions in NO- and prostanoid-independent relaxations evoked by acetylcholine in rabbit intracerebral arteries

Stroke (1970), 2003-02, Vol.34 (2), p.544-550

2003

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Spatial heterogeneity in the mechanisms contributing to acetylcholine‐induced dilatation in the rabbit isolated ear

British journal of pharmacology, 1998-07, Vol.124 (6), p.1245-1253

1998

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Relative Contributions of NO and Gap Junctional Communication to Endothelium-Dependent Relaxations of Rabbit Resistance Arteries Vary with Vessel Size

Microvascular research, 2002-01, Vol.63 (1), p.115-128

2002

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Modulation of Gap Junction-Dependent Arterial Relaxation by Ascorbic Acid

Journal of vascular research, 2007-01, Vol.44 (5), p.410-422

2007

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Connexins and gap junctions in the EDHF phenomenon and conducted vasomotor responses

Endothelium‐dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?

Ascorbic acid and tetrahydrobiopterin potentiate the EDHF phenomenon by generating hydrogen peroxide

cAMP Facilitates EDHF-Type Relaxations in Conduit Arteries by Enhancing Electrotonic Conduction via Gap Junctions

Gap junctional communication underpins EDHF-type relaxations evoked by ACh in the rat hepatic artery

5-Methyltetrahydrofolate and Tetrahydrobiopterin Can Modulate Electrotonically Mediated Endothelium-Dependent Vascular Relaxation

Distinct hyperpolarizing and relaxant roles for gap junctions and endothelium-derived H2O2 in NO-independent relaxations of rabbit arteries

The effect of inorganic arsenic on endothelium-dependent relaxation: Role of NADPH oxidase and hydrogen peroxide

Effects of connexin‐mimetic peptides on gap junction functionality and connexin expression in cultured vascular cells

Gap junction-dependent and -independent EDHF-type relaxations may involve smooth muscle cAMP accumulation

Which Connexins Connect?

Deterministic nonlinear features of cutaneous perfusion are lost in diabetic subjects with neuropathy

Central role of intracellular calcium stores in acute flow‐ and agonist‐evoked endothelial nitric oxide release

Connexin‐mimetic peptides dissociate electrotonic EDHF‐type signalling via myoendothelial and smooth muscle gap junctions in the rabbit iliac artery

Analysis of effects of connexin-mimetic peptides in rat mesenteric small arteries

The obligatory link: role of gap junctional communication in endothelium-dependent smooth muscle hyperpolarization

Attenuated store-operated Ca2+ entry underpins the dual inhibition of nitric oxide and EDHF-type relaxations by iodinated contrast media

Inhibition of the gap junctional component of endothelium‐dependent relaxations in rabbit iliac artery by 18‐α glycyrrhetinic acid

Endothelial control of vascular tone by nitric oxide and gap junctions: a haemodynamic perspective

Nitric Oxide-Independent Relaxations to Acetylcholine and A23187 Involve Different Routes of Heterocellular Communication: Role of Gap Junctions and Phospholipase A2

Endogenous Nitric Oxide Synthesis Differentially Modulates Pressure-Flow and Pressure-Conductance Relationships in the Internal and External Carotid Artery Circulations of the Rat

Essential role of gap junctions in NO- and prostanoid-independent relaxations evoked by acetylcholine in rabbit intracerebral arteries

Spatial heterogeneity in the mechanisms contributing to acetylcholine‐induced dilatation in the rabbit isolated ear

Relative Contributions of NO and Gap Junctional Communication to Endothelium-Dependent Relaxations of Rabbit Resistance Arteries Vary with Vessel Size

Modulation of Gap Junction-Dependent Arterial Relaxation by Ascorbic Acid

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