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Cholesterol esterification and atherogenic index of plasma correlate with lipoprotein size and findings on coronary angiography

Journal of lipid research, 2011-03, Vol.52 (3), p.566-571

2011

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Does ENHANCE Diminish Confidence in Lowering LDL or in Ezetimibe?

The New England journal of medicine, 2008-04, Vol.358 (14), p.1504-1507

2008

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Nicotinic Acid, Alone and in Combinations, for Reduction of Cardiovascular Risk

The American journal of cardiology, 2008-04, Vol.101 (8), p.S58-S62

2008

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Levels of cholesterol in small LDL particles predict atherosclerosis progression and incident CHD in the HDL-Atherosclerosis Treatment Study (HATS)

PloS one, 2013-02, Vol.8 (2), p.e56782-e56782

2013

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Simvastatin and Niacin, Antioxidant Vitamins, or the Combination for the Prevention of Coronary Disease

The New England journal of medicine, 2001-11, Vol.345 (22), p.1583-1592

2001

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Lipid-Altering Efficacy and Safety of Ezetimibe/Simvastatin Coadministered With Extended-Release Niacin in Patients With Type IIa or Type IIb Hyperlipidemia

Journal of the American College of Cardiology, 2008-04, Vol.51 (16), p.1564-1572

2008

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Effects of Niacin Combination Therapy With Statin or Bile Acid Resin on Lipoproteins and Cardiovascular Disease

The American journal of cardiology, 2014-05, Vol.113 (9), p.1494-1498

2014

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Effects of Niacin on Glucose Levels, Coronary Stenosis Progression, and Clinical Events in Subjects With Normal Baseline Glucose Levels (<100 mg/dl): A Combined Analysis of the Familial Atherosclerosis Treatment Study (FATS), HDL-Atherosclerosis Treatment Study (HATS), Armed Forces Regression Study (AFREGS), and Carotid Plaque Composition by MRI During Lipid-Lowering (CPC) Study

The American journal of cardiology, 2013-02, Vol.111 (3), p.352-355

2013

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Effects of Combination Lipid Therapy on Coronary Stenosis Progression and Clinical Cardiovascular Events in Coronary Disease Patients With Metabolic Syndrome: A Combined Analysis of the Familial Atherosclerosis Treatment Study (FATS), the HDL-Atherosclerosis Treatment Study (HATS), and the Armed Forces Regression Study (AFREGS)

The American journal of cardiology, 2009-12, Vol.104 (11), p.1457-1464

2009

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Is Intravascular Ultrasound the Gold Standard Surrogate for Clinically Relevant Atherosclerosis Progression?

Journal of the American College of Cardiology, 2007-03, Vol.49 (9), p.933-938

2007

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Comparison of four methods of analysis of lipoprotein particle subfractions for their association with angiographic progression of coronary artery disease

Atherosclerosis, 2014-04, Vol.233 (2), p.713-720

2014

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Mortality reduction in patients treated with long-term intensive lipid therapy: 25-year follow-up of the Familial Atherosclerosis Treatment Study—Observational Study

Journal of clinical lipidology, 2016-09, Vol.10 (5), p.1091-1097

2016

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Prolonged combination lipid therapy is associated with reduced carotid intima-media thickness: A case-control study of the 20-year Familial Atherosclerosis Treatment - Observational Study (FATS-OS)

Journal of clinical lipidology, 2014-09, Vol.8 (5), p.489-493

2014

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Effect of intensive compared with moderate lipid-lowering therapy on progression of coronary atherosclerosis: a randomized controlled trial

JAMA : the journal of the American Medical Association, 2004-03, Vol.291 (9), p.1071

2004

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Simultaneous low-density lipoprotein-C lowering and high-density lipoprotein-C elevation for optimum cardiovascular disease prevention with various drug classes, and their combinations: a meta-analysis of 23 randomized lipid trials

Current opinion in lipidology, 2006-12, Vol.17 (6), p.631-636

2006

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Expert Commentary: Niacin Safety

The American journal of cardiology, 2007-03, Vol.99 (6), p.S32-S34

Brown, B. Greg, MD, PhD

2007

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Effects of lowering elevated LDL cholesterol on the cardiovascular risk of lipoprotein(a)

JAMA : the journal of the American Medical Association, 1995-12, Vol.274 (22), p.1771

1995

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Niacin plus simvastatin reduces coronary stenosis progression among patients with metabolic syndrome despite a modest increase in insulin resistance: A subgroup analysis of the HDL-Atherosclerosis treatment study

Journal of clinical lipidology, 2007-07, Vol.1 (3), p.203-210

2007

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Should both HDL-C and LDL-C be targets for lipid therapy? A review of current evidence

Journal of clinical lipidology, 2007-03, Vol.1 (1), p.88-94

2007

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Is there any hope for vitamin E?

JAMA : the journal of the American Medical Association, 2005-03, Vol.293 (11), p.1387

2005

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Apolipoprotein L-I is positively associated with hyperglycemia and plasma triglycerides in CAD patients with low HDL

Journal of lipid research, 2005-03, Vol.46 (3), p.469-474

2005

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Effects of prolonged intensive lipid-lowering therapy on the characteristics of carotid atherosclerotic plaques in vivo by MRI: a case-control study

Arteriosclerosis, thrombosis, and vascular biology, 2001-10, Vol.21 (10), p.1623-1629

2001

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A direct comparison of intravascular ultrasound and quantitative coronary arteriography: implications for measures of atherosclerosis as clinical surrogates

Circulation (New York, N.Y.), 2007-04, Vol.115 (14), p.1824-1826

Brown, B Greg

2007

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Evidence for a new pathophysiological mechanism for coronary artery disease regression : Hepatic lipase-mediated changes in LDL density

Circulation (New York, N.Y.), 1999-04, Vol.99 (15), p.1959-1964

1999

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Antioxidant supplements block the response of HDL to simvastatin-niacin therapy in patients with coronary artery disease and low HDL

Arteriosclerosis, thrombosis, and vascular biology, 2001-08, Vol.21 (8), p.1320-1326

2001

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Cholesterol esterification and atherogenic index of plasma correlate with lipoprotein size and findings on coronary angiography

Does ENHANCE Diminish Confidence in Lowering LDL or in Ezetimibe?

Nicotinic Acid, Alone and in Combinations, for Reduction of Cardiovascular Risk

Levels of cholesterol in small LDL particles predict atherosclerosis progression and incident CHD in the HDL-Atherosclerosis Treatment Study (HATS)

Simvastatin and Niacin, Antioxidant Vitamins, or the Combination for the Prevention of Coronary Disease

Lipid-Altering Efficacy and Safety of Ezetimibe/Simvastatin Coadministered With Extended-Release Niacin in Patients With Type IIa or Type IIb Hyperlipidemia

Effects of Niacin Combination Therapy With Statin or Bile Acid Resin on Lipoproteins and Cardiovascular Disease

Is Intravascular Ultrasound the Gold Standard Surrogate for Clinically Relevant Atherosclerosis Progression?

Comparison of four methods of analysis of lipoprotein particle subfractions for their association with angiographic progression of coronary artery disease

Mortality reduction in patients treated with long-term intensive lipid therapy: 25-year follow-up of the Familial Atherosclerosis Treatment Study—Observational Study

Prolonged combination lipid therapy is associated with reduced carotid intima-media thickness: A case-control study of the 20-year Familial Atherosclerosis Treatment - Observational Study (FATS-OS)

Effect of intensive compared with moderate lipid-lowering therapy on progression of coronary atherosclerosis: a randomized controlled trial

Simultaneous low-density lipoprotein-C lowering and high-density lipoprotein-C elevation for optimum cardiovascular disease prevention with various drug classes, and their combinations: a meta-analysis of 23 randomized lipid trials

Expert Commentary: Niacin Safety

Effects of lowering elevated LDL cholesterol on the cardiovascular risk of lipoprotein(a)

Niacin plus simvastatin reduces coronary stenosis progression among patients with metabolic syndrome despite a modest increase in insulin resistance: A subgroup analysis of the HDL-Atherosclerosis treatment study

Should both HDL-C and LDL-C be targets for lipid therapy? A review of current evidence

Is there any hope for vitamin E?

Apolipoprotein L-I is positively associated with hyperglycemia and plasma triglycerides in CAD patients with low HDL

Effects of prolonged intensive lipid-lowering therapy on the characteristics of carotid atherosclerotic plaques in vivo by MRI: a case-control study

A direct comparison of intravascular ultrasound and quantitative coronary arteriography: implications for measures of atherosclerosis as clinical surrogates

Evidence for a new pathophysiological mechanism for coronary artery disease regression : Hepatic lipase-mediated changes in LDL density

Antioxidant supplements block the response of HDL to simvastatin-niacin therapy in patients with coronary artery disease and low HDL

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