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The roles of calcium and ATP in the physiology and pathology of the exocrine pancreas

Physiological reviews, 2021-10, Vol.101 (4), p.1691-1744

2021

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The role of Ca2+ in the pathophysiology of pancreatitis

The Journal of physiology, 2014-01, Vol.592 (2), p.269-280

2014

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CRAC channel inhibitors in pancreatic pathologies

The Journal of physiology, 2022-04, Vol.600 (7), p.1597-1598

2022

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Ca²⁺ release-activated Ca²⁺ channel blockade as a potential tool in antipancreatitis therapy

Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (32), p.13186-13191

2013

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The role of CFTR in diabetes‐induced pancreatic ductal dysfunction

The Journal of physiology, 2024-03, Vol.602 (6), p.993-994

2024

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The role of Ca2+ signalling in the pathology of exocrine pancreas

Cell calcium (Edinburgh), 2023-06, Vol.112, p.102740-102740, Article 102740

2023

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Activation of pancreatic stellate cells attenuates intracellular Ca2+ signals due to downregulation of TRPA1 and protects against cell death induced by alcohol metabolites

Cell death & disease, 2022-08, Vol.13 (8), p.1-14, Article 744

2022

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Ca2+ signalling underlying pancreatitis

Cell calcium (Edinburgh), 2018-03, Vol.70, p.95-101

2018

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Calcium Elevation in Mitochondria Is the Main Ca2+ Requirement for Mitochondrial Permeability Transition Pore (mPTP) Opening

The Journal of biological chemistry, 2009-07, Vol.284 (31), p.20796-20803

2009

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Ca2+ signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca2+ channel blockade

The Journal of physiology, 2016-01, Vol.594 (2), p.281-293

2016

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Calcium signalling in the acinar environment of the exocrine pancreas: physiology and pathophysiology

The Journal of physiology, 2018-07, Vol.596 (14), p.2663-2678

2018

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A Novel Role for Bcl-2 in Regulation of Cellular Calcium Extrusion

Current biology, 2012-07, Vol.22 (13), p.1241-1246

2012

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Ca(2+) signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca(2+) channel blockade

The Journal of physiology, 2016-01, Vol.594 (2), p.281-293

2016

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Both RyRs and TPCs are required for NAADP-induced intracellular Ca2+ release

Cell calcium (Edinburgh), 2015-09, Vol.58 (3), p.237-245

2015

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NAADP, cADPR and IP3 all release Ca2+ from the endoplasmic reticulum and an acidic store in the secretory granule area

Journal of cell science, 2006-01, Vol.119 (Pt 2), p.226-238

2006

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Galactose protects against cell damage in mouse models of acute pancreatitis

The Journal of clinical investigation, 2018-09, Vol.128 (9), p.3769-3778

2018

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Calcium and adenosine triphosphate control of cellular pathology: asparaginase-induced pancreatitis elicited via protease-activated receptor 2

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2016-08, Vol.371 (1700)

2016

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Pancreatic protease activation by alcohol metabolite depends on Ca²⁺ release via acid store IP₃ receptors

Proceedings of the National Academy of Sciences - PNAS, 2009-06, Vol.106 (26), p.10758-10763

2009

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Bile acids induce necrosis in pancreatic stellate cells dependent on calcium entry and sodium‐driven bile uptake

The Journal of physiology, 2016-11, Vol.594 (21), p.6147-6164

2016

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Endocytic uptake of SARS-CoV-2: the critical roles of pH, Ca2+, and NAADP

Function (Oxford, England), 2020-01, Vol.1 (1), p.zqaa003-zqaa003

2020

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Bile Acids Induce Ca2+ Release from Both the Endoplasmic Reticulum and Acidic Intracellular Calcium Stores through Activation of Inositol Trisphosphate Receptors and Ryanodine Receptors

The Journal of biological chemistry, 2006-12, Vol.281 (52), p.40154-40163

2006

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Nitric oxide signals are interlinked with calcium signals in normal pancreatic stellate cells upon oxidative stress and inflammation

Open biology, 2016-08, Vol.6 (8), p.160149

2016

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Direct Activation of Cytosolic Ca2+ Signaling and Enzyme Secretion by Cholecystokinin in Human Pancreatic Acinar Cells

Gastroenterology (New York, N.Y. 1943), 2008-08, Vol.135 (2), p.632-641

2008

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Calmodulin protects against alcohol-induced pancreatic trypsinogen activation elicited via Ca²⁺ release through IP₃ receptors

Proceedings of the National Academy of Sciences - PNAS, 2011-04, Vol.108 (14), p.5873-5878

2011

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Elastic and inelastic particles scattering by dust acoustic soliton. A new oscillatory process in dusty plasma

New journal of physics, 2021-09, Vol.23 (9), p.93016

2021

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The roles of calcium and ATP in the physiology and pathology of the exocrine pancreas

The role of Ca2+ in the pathophysiology of pancreatitis

CRAC channel inhibitors in pancreatic pathologies

Ca²⁺ release-activated Ca²⁺ channel blockade as a potential tool in antipancreatitis therapy

The role of CFTR in diabetes‐induced pancreatic ductal dysfunction

The role of Ca2+ signalling in the pathology of exocrine pancreas

Activation of pancreatic stellate cells attenuates intracellular Ca2+ signals due to downregulation of TRPA1 and protects against cell death induced by alcohol metabolites

Ca2+ signalling underlying pancreatitis

Calcium Elevation in Mitochondria Is the Main Ca2+ Requirement for Mitochondrial Permeability Transition Pore (mPTP) Opening

Ca2+ signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca2+ channel blockade

Calcium signalling in the acinar environment of the exocrine pancreas: physiology and pathophysiology

A Novel Role for Bcl-2 in Regulation of Cellular Calcium Extrusion

Ca(2+) signals mediated by bradykinin type 2 receptors in normal pancreatic stellate cells can be inhibited by specific Ca(2+) channel blockade

Both RyRs and TPCs are required for NAADP-induced intracellular Ca2+ release

NAADP, cADPR and IP3 all release Ca2+ from the endoplasmic reticulum and an acidic store in the secretory granule area

Galactose protects against cell damage in mouse models of acute pancreatitis

Calcium and adenosine triphosphate control of cellular pathology: asparaginase-induced pancreatitis elicited via protease-activated receptor 2

Pancreatic protease activation by alcohol metabolite depends on Ca²⁺ release via acid store IP₃ receptors

Bile acids induce necrosis in pancreatic stellate cells dependent on calcium entry and sodium‐driven bile uptake

Endocytic uptake of SARS-CoV-2: the critical roles of pH, Ca2+, and NAADP

Bile Acids Induce Ca2+ Release from Both the Endoplasmic Reticulum and Acidic Intracellular Calcium Stores through Activation of Inositol Trisphosphate Receptors and Ryanodine Receptors

Nitric oxide signals are interlinked with calcium signals in normal pancreatic stellate cells upon oxidative stress and inflammation

Direct Activation of Cytosolic Ca2+ Signaling and Enzyme Secretion by Cholecystokinin in Human Pancreatic Acinar Cells

Calmodulin protects against alcohol-induced pancreatic trypsinogen activation elicited via Ca²⁺ release through IP₃ receptors

Elastic and inelastic particles scattering by dust acoustic soliton. A new oscillatory process in dusty plasma

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