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Chemokines and tissue injury

The American journal of pathology, 1995-06, Vol.146 (6), p.1287-1301

1995

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The adhesion molecules used by monocytes for migration across endothelium include CD11a/CD18, CD11b/CD18, and VLA-4 on monocytes and ICAM-1, VCAM-1, and other ligands on endothelium

The Journal of immunology (1950), 1995-04, Vol.154 (8), p.4099

1995

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Borrelia burgdorferi binds plasminogen, resulting in enhanced penetration of endothelial monolayers

Infection and Immunity, 1995-07, Vol.63 (7), p.2478-2484

1995

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Borrelia burgdorferi upregulates expression of adhesion molecules on endothelial cells and promotes transendothelial migration of neutrophils in vitro

Infection and Immunity, 1995-11, Vol.63 (11), p.4439-4447

1995

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Monocytes use either CD11/CD18 or VLA-4 to migrate across human endothelium in vitro

The Journal of immunology (1950), 1994-02, Vol.152 (4), p.1915

1994

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Outer surface lipoproteins of Borrelia burgdorferi activate vascular endothelium in vitro

Infection and Immunity, 1996-08, Vol.64 (8), p.3180-3187

1996

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Monoclonal Antibodies to Leukocyte Integrins CD11a/CD18 and CD11b/CD18 or Intercellular Adhesion Molecule-1 Inhibit Chemoattractant-Stimulated Neutrophil Transendothelial Migration In Vitro

Blood, 1991-10, Vol.78 (8), p.2089-2097

1991

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Activation of Endothelium by Borrelia burgdorferi In Vitro Enhances Transmigration of Specific Subsets of T Lymphocytes

Infection and Immunity, 2001-04, Vol.69 (4), p.2190-2197

2001

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The live vaccine strain of Francisella tularensis replicates in human and murine macrophages but induces only the human cells to secrete proinflammatory cytokines

Journal of leukocyte biology, 2005-06, Vol.77 (6), p.893-897

2005

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Migration of neutrophils across endothelial monolayers is stimulated by treatment of the monolayers with interleukin-1 or tumor necrosis factor-alpha

The Journal of immunology (1950), 1989-11, Vol.143 (10), p.3309

1989

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Borrelia burgdorferi and Interleukin-1 Promote the Transendothelial Migration of Monocytes In Vitro by Different Mechanisms

Infection and Immunity, 1998-10, Vol.66 (10), p.4875-4883

1998

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Production of interleukin-8 (IL-8) by cultured endothelial cells in response to Borrelia burgdorferi occurs independently of secretion IL-1 and tumor necrosis factor alpha and is required for subsequent transendothelial migration of neutrophils

Infection and immunity, 1997-04, Vol.65 (4), p.1217-1222

1997

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Macrovascular and microvascular endothelium during long-term hypoxia: Alterations in cell growth, monolayer permeability, and cell surface coagulant properties

Journal of cellular physiology, 1991-01, Vol.146 (1), p.8-17

1991

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Francisella tularensis Selectively Induces Proinflammatory Changes in Endothelial Cells

The Journal of immunology (1950), 2003-09, Vol.171 (5), p.2563-2570

2003

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Interleukin-10 inhibits proinflammatory activation of endothelium in response to Borrelia burgdorferi or lipopolysaccharide but not interleukin-1β or tumor necrosis factor α

Journal of leukocyte biology, 2002-09, Vol.72 (3), p.503-511

2002

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Cultured Endothelial Cell Monolayers That Restrict the Transendothelial Passage of Macromolecules and Electrical Current

The Journal of cell biology, 1984-03, Vol.98 (3), p.1033-1041

1984

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A soluble gradient of endogenous monocyte chemoattractant protein-1 promotes the transendothelial migration of monocytes in vitro

The Journal of immunology (1950), 1995-10, Vol.155 (7), p.3610

1995

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Hypoxia-Mediated Induction of Acidic/Basic Fibroblast Growth Factor and Platelet-Derived Growth Factor in Mononuclear Phagocytes Stimulates Growth of Hypoxic Endothelial Cells

Proceedings of the National Academy of Sciences - PNAS, 1995-05, Vol.92 (10), p.4606-4610

1995

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Francisella tularensis Has a Significant Extracellular Phase in Infected Mice

The Journal of infectious diseases, 2007-07, Vol.196 (1), p.134-137

2007

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Migration of neutrophils across monolayers of cultured microvascular endothelial cells. An in vitro model of leucocyte extravasation

Journal of cell science, 1987-09, Vol.88 ( Pt 2) (2), p.161-175

1987

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Populations of Human T Lymphocytes That Traverse the Vascular Endothelium Stimulated by Borrelia burgdorferi Are Enriched with Cells That Secrete Gamma Interferon

Infection and Immunity, 2004-03, Vol.72 (3), p.1530-1536

2004

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The Formation of Fenestrations and Channels by Capillary Endothelium in vitro

Proceedings of the National Academy of Sciences - PNAS, 1985-09, Vol.82 (18), p.6181-6185

1985

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Extracts of smokeless tobacco induce pro-inflammatory changes in cultured human vascular endothelial cells

Immunopharmacology, 2000-04, Vol.47 (1), p.13-23

2000

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Effects of human neutrophil chemotaxis across human endothelial cell monolayers on the permeability of these monolayers to ions and macromolecules

Journal of cellular physiology, 1988-06, Vol.135 (3), p.355-366

1988

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Mononuclear phagocytes egress from an in vitro model of the vascular wall by migrating across endothelium in the basal to apical direction: role of intercellular adhesion molecule 1 and the CD11/CD18 integrins

The Journal of experimental medicine, 1996-02, Vol.183 (2), p.451-462

1996

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Chemokines and tissue injury

The adhesion molecules used by monocytes for migration across endothelium include CD11a/CD18, CD11b/CD18, and VLA-4 on monocytes and ICAM-1, VCAM-1, and other ligands on endothelium

Borrelia burgdorferi binds plasminogen, resulting in enhanced penetration of endothelial monolayers

Borrelia burgdorferi upregulates expression of adhesion molecules on endothelial cells and promotes transendothelial migration of neutrophils in vitro

Monocytes use either CD11/CD18 or VLA-4 to migrate across human endothelium in vitro

Outer surface lipoproteins of Borrelia burgdorferi activate vascular endothelium in vitro

Monoclonal Antibodies to Leukocyte Integrins CD11a/CD18 and CD11b/CD18 or Intercellular Adhesion Molecule-1 Inhibit Chemoattractant-Stimulated Neutrophil Transendothelial Migration In Vitro

Activation of Endothelium by Borrelia burgdorferi In Vitro Enhances Transmigration of Specific Subsets of T Lymphocytes

The live vaccine strain of Francisella tularensis replicates in human and murine macrophages but induces only the human cells to secrete proinflammatory cytokines

Migration of neutrophils across endothelial monolayers is stimulated by treatment of the monolayers with interleukin-1 or tumor necrosis factor-alpha

Borrelia burgdorferi and Interleukin-1 Promote the Transendothelial Migration of Monocytes In Vitro by Different Mechanisms

Production of interleukin-8 (IL-8) by cultured endothelial cells in response to Borrelia burgdorferi occurs independently of secretion IL-1 and tumor necrosis factor alpha and is required for subsequent transendothelial migration of neutrophils

Macrovascular and microvascular endothelium during long-term hypoxia: Alterations in cell growth, monolayer permeability, and cell surface coagulant properties

Francisella tularensis Selectively Induces Proinflammatory Changes in Endothelial Cells

Interleukin-10 inhibits proinflammatory activation of endothelium in response to Borrelia burgdorferi or lipopolysaccharide but not interleukin-1β or tumor necrosis factor α

Cultured Endothelial Cell Monolayers That Restrict the Transendothelial Passage of Macromolecules and Electrical Current

A soluble gradient of endogenous monocyte chemoattractant protein-1 promotes the transendothelial migration of monocytes in vitro

Hypoxia-Mediated Induction of Acidic/Basic Fibroblast Growth Factor and Platelet-Derived Growth Factor in Mononuclear Phagocytes Stimulates Growth of Hypoxic Endothelial Cells

Francisella tularensis Has a Significant Extracellular Phase in Infected Mice

Migration of neutrophils across monolayers of cultured microvascular endothelial cells. An in vitro model of leucocyte extravasation

Populations of Human T Lymphocytes That Traverse the Vascular Endothelium Stimulated by Borrelia burgdorferi Are Enriched with Cells That Secrete Gamma Interferon

The Formation of Fenestrations and Channels by Capillary Endothelium in vitro

Extracts of smokeless tobacco induce pro-inflammatory changes in cultured human vascular endothelial cells

Effects of human neutrophil chemotaxis across human endothelial cell monolayers on the permeability of these monolayers to ions and macromolecules

Mononuclear phagocytes egress from an in vitro model of the vascular wall by migrating across endothelium in the basal to apical direction: role of intercellular adhesion molecule 1 and the CD11/CD18 integrins

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