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Kainic acid-induced seizures produce necrotic, not apoptotic, neurons with internucleosomal DNA cleavage: implications for programmed cell death mechanisms

Neuroscience, 2000-01, Vol.98 (1), p.41-53

2000

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The temporal evolution of neuronal damage from pilocarpine-induced status epilepticus

Brain research, 1996-06, Vol.725 (1), p.11-22

Fujikawa, Denson G.

1996

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Neuroprotective Effect of Ketamine Administered After Status Epilepticus Onset

Epilepsia (Copenhagen), 1995-02, Vol.36 (2), p.186-195

Fujikawa, Denson G.

1995

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Status Epilepticus–Induced Neuronal Loss in Humans Without Systemic Complications or Epilepsy

Epilepsia (Copenhagen), 2000-08, Vol.41 (8), p.981-991

2000

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Pathophysiological Mechanisms of Brain Damage from Status Epilepticus

Epilepsia (Copenhagen), 1993-01, Vol.34 (s1), p.S37-S53

1993

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Seizure‐Induced Neuronal Necrosis: Implications for Programmed Cell Death Mechanisms

Epilepsia (Copenhagen), 2000-01, Vol.41 (s6), p.S9-S13

2000

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Lithium-pilocarpine-induced status epilepticus produces necrotic neurons with internucleosomal DNA fragmentation in adult rats

The European journal of neuroscience, 1999-05, Vol.11 (5), p.1605-1614

1999

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Confusion between neuronal apoptosis and activation of programmed cell death mechanisms in acute necrotic insults

Trends in neurosciences (Regular ed.), 2000-09, Vol.23 (9), p.410-411

Fujikawa, Denson G

2000

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Nonconvulsive status epilepticus with frontal features: quantitating severity of subclinical epileptiform discharges provides a marker for treatment efficacy, recurrence and outcome

Epilepsy research, 2002-09, Vol.51 (1), p.13-21

2002

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The competitive NMDA receptor antagonist CGP 40116 protects against status epilepticus-induced neuronal damage

Epilepsy research, 1994-03, Vol.17 (3), p.207-219

1994

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Hypoxic Neuronal Necrosis: Protein Synthesis-Independent Activation of a Cell Death Program

Proceedings of the National Academy of Sciences - PNAS, 2003-03, Vol.100 (5), p.2825-2830

2003

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Posthypoxic treatment with MK-801 reduces hypoxic-ischemic damage in the neonatal rat

Neurology, 1989-05, Vol.39 (5), p.713-713

1989

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In vivo elevation of extracellular potassium in the rat amygdala increases extracellular glutamate and aspartate and damages neurons

Neuroscience, 1996-10, Vol.74 (3), p.695-706

1996

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An electroencephalographic study comparing maximum blink rates in schizophrenic and nonschizophrenic psychiatric patients and nonpsychiatric control subjects

Biological psychiatry (1969), 1998-02, Vol.43 (4), p.282-287

1998

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Effects of N-methyl- d-aspartate-receptor blockade on high-potassium-induced neuronal death and glutamate release

Neuroscience letters, 1997-04, Vol.226 (1), p.25-28

Fujikawa, Denson G

1997

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Prolonged seizures and cellular injury: Understanding the connection

Epilepsy & behavior, 2005-12, Vol.7, p.3-11

Fujikawa, Denson G.

2005

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Caspase‐3 is not activated in seizure‐induced neuronal necrosis with internucleosomal DNA cleavage

Journal of neurochemistry, 2002-10, Vol.83 (1), p.229-240

2002

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364 SEIZURE-INDUCED NEURONAL NECROSIS IS A CASPASE-INDEPENDENT PROCESS

Journal of investigative medicine, 2006-01, Vol.54 (1), p.S143-S143

2006

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Effects of AMPA-receptor and voltage-sensitive sodium channel blockade on high potassium-induced glutamate release and neuronal death in vivo

Brain research, 2002-08, Vol.946 (1), p.119-129

2002

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Local cerebral glucose utilization during status epilepticus in newborn primates

American Journal of Physiology: Cell Physiology, 1989-06, Vol.256 (6), p.C1160-C1167

1989

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Caspase-dependent programmed cell death pathways are not activated in generalized seizure-induced neuronal death

Brain research, 2007-03, Vol.1135 (1), p.206-218

2007

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Generalized seizures deplete brain energy reserves in normoxemic newborn monkeys

Brain research, 1988-06, Vol.454 (1), p.51-59

1988

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Neuropathological changes during generalized seizures in newborn monkeys

Epilepsy research, 1992-09, Vol.12 (3), p.243-251

1992

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Single-dose phenytoin infusion

Annals of internal medicine, 1990-02, Vol.112 (3), p.235

Fujikawa, D G

1990

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Anticonvulsants for eclampsia

The Lancet (British edition), 1995-08, Vol.346 (8973), p.501

Fujikawa, D G

1995

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Kainic acid-induced seizures produce necrotic, not apoptotic, neurons with internucleosomal DNA cleavage: implications for programmed cell death mechanisms

The temporal evolution of neuronal damage from pilocarpine-induced status epilepticus

Neuroprotective Effect of Ketamine Administered After Status Epilepticus Onset

Status Epilepticus–Induced Neuronal Loss in Humans Without Systemic Complications or Epilepsy

Pathophysiological Mechanisms of Brain Damage from Status Epilepticus

Seizure‐Induced Neuronal Necrosis: Implications for Programmed Cell Death Mechanisms

Lithium-pilocarpine-induced status epilepticus produces necrotic neurons with internucleosomal DNA fragmentation in adult rats

Confusion between neuronal apoptosis and activation of programmed cell death mechanisms in acute necrotic insults

Nonconvulsive status epilepticus with frontal features: quantitating severity of subclinical epileptiform discharges provides a marker for treatment efficacy, recurrence and outcome

The competitive NMDA receptor antagonist CGP 40116 protects against status epilepticus-induced neuronal damage

Hypoxic Neuronal Necrosis: Protein Synthesis-Independent Activation of a Cell Death Program

Posthypoxic treatment with MK-801 reduces hypoxic-ischemic damage in the neonatal rat

In vivo elevation of extracellular potassium in the rat amygdala increases extracellular glutamate and aspartate and damages neurons

An electroencephalographic study comparing maximum blink rates in schizophrenic and nonschizophrenic psychiatric patients and nonpsychiatric control subjects

Effects of N-methyl- d-aspartate-receptor blockade on high-potassium-induced neuronal death and glutamate release

Prolonged seizures and cellular injury: Understanding the connection

Caspase‐3 is not activated in seizure‐induced neuronal necrosis with internucleosomal DNA cleavage

364 SEIZURE-INDUCED NEURONAL NECROSIS IS A CASPASE-INDEPENDENT PROCESS

Effects of AMPA-receptor and voltage-sensitive sodium channel blockade on high potassium-induced glutamate release and neuronal death in vivo

Local cerebral glucose utilization during status epilepticus in newborn primates

Caspase-dependent programmed cell death pathways are not activated in generalized seizure-induced neuronal death

Generalized seizures deplete brain energy reserves in normoxemic newborn monkeys

Neuropathological changes during generalized seizures in newborn monkeys

Single-dose phenytoin infusion

Anticonvulsants for eclampsia

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