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Significance of Development and Reversion of Collaterals on MRI in Early Neurologic Improvement and Long-Term Functional Outcome after Intravenous Thrombolysis for Ischemic Stroke

American journal of neuroradiology : AJNR, 2015-10, Vol.36 (10), p.1839-1845

2015

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SUMOylation substrates in neuronal intranuclear inclusion disease

Neuropathology and applied neurobiology, 2006-02, Vol.32 (1), p.92-100

2006

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CAG repeat expansion in the TATA box-binding protein gene causes autosomal dominant cerebellar ataxia

Brain (London, England : 1878), 2001-10, Vol.124 (10), p.1939-1947

2001

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Histone deacetylase (HDAC) 4 involvement in both Lewy and Marinesco bodies

Neuropathology and applied neurobiology, 2006-10, Vol.32 (5), p.562-566

2006

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Cytoplasmic and nuclear polyglutamine aggregates in SCA6 Purkinje cells

Neurology, 2001-06, Vol.56 (12), p.1753-1756

2001

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Similarities between spinocerebellar ataxia type 7 (SCA7) cell models and human brain: proteins recruited in inclusions and activation of caspase-3

Human molecular genetics, 2001-10, Vol.10 (22), p.2569-2579

2001

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PML nuclear bodies and neuronal intranuclear inclusion in polyglutamine diseases

Neurobiology of disease, 2003-08, Vol.13 (3), p.230-237

2003

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Amyotrophic lateral sclerosis with neuronal intranuclear protein inclusions

Acta neuropathologica, 2004-07, Vol.108 (1), p.81-87

2004

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A simple non-invasive method to measure the spontaneous swallowing frequency by a laryngeal microphone in Parkinson’s disease patients

Journal of the neurological sciences, 2017-10, Vol.381, p.584-584

2017

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Neuronal intranuclear hyaline inclusion disease with polyglutamine-immunoreactive inclusions

Acta neuropathologica, 2000-05, Vol.99 (5), p.589-594

2000

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Preferential recruitment of ataxin-3 independent of expanded polyglutamine: an immunohistochemical study on Marinesco bodies

Journal of neurology, neurosurgery and psychiatry, 2001-10, Vol.71 (4), p.518-520

2001

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Huntington’s disease‐like phenotype due to trinucleotide repeat expansions in the TBP and JPH3 genes

Brain (London, England : 1878), 2003-07, Vol.126 (7), p.1599-1603

2003

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Abundant Expression and Cytoplasmic Aggregations of α1A Voltage-Dependent Calcium Channel Protein Associated with Neurodegeneration in Spinocerebellar Ataxia Type 6

Human molecular genetics, 1999-07, Vol.8 (7), p.1185-1193

1999

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Neuronal intranuclear inclusions in spinocerebellar ataxia type 2: triple-labeling immunofluorescent study

Neuroscience letters, 1999-10, Vol.273 (2), p.117-120

1999

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Non-expanded polyglutamine proteins in intranuclear inclusions of hereditary ataxias - triple-labeling immunofluorescence study

Acta neuropathologica, 2001-08, Vol.102 (2), p.149-152

2001

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Spinocerebellar Ataxia Type 6 Mutation Alters P-type Calcium Channel Function

The Journal of biological chemistry, 2000-04, Vol.275 (15), p.10893-10898

2000

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Abundant expression and cytoplasmic aggregations of alpha 1A voltage-dependent calcium channel protein associated with neurodegeneration in spinocerebellar ataxia type 6

Human molecular genetics, 1999-07, Vol.8 (7), p.1185-1193

1999

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A clinical and genetic study in a large cohort of patients with spinocerebellar ataxia type 6

Journal of human genetics, 2004-05, Vol.49 (5), p.256-264

2004

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Ataxin-3 Is Translocated into the Nucleus for the Formation of Intranuclear Inclusions in Normal and Machado–Joseph Disease Brains

Experimental neurology, 2000-10, Vol.165 (2), p.248-256

2000

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Recruitment of Nonexpanded Polyglutamine Proteins to Intranuclear Aggregates in Neuronal Intranuclear Hyaline Inclusion Disease

Journal of neuropathology and experimental neurology, 2001-04, Vol.60 (4), p.369-376

2001

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SCA12 is a rare locus for autosomal dominant cerebellar ataxia: A study of an Indian family

Annals of neurology, 2001-01, Vol.49 (1), p.117-121

2001

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Two populations of neuronal intranuclear inclusions in SCA7 differ in size and promyelocytic leukaemia protein content

Brain (London, England : 1878), 2002-07, Vol.125 (7), p.1534-1543

2002

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Spastic paraparesis and mutations in the prion protein gene

Journal of the neurological sciences, 1995-12, Vol.134 (1), p.215-216

1995

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Spastic tetraplegia as an initial manifestation of familial Alzheimer's disease

Journal of neurology, neurosurgery and psychiatry, 1995-10, Vol.59 (4), p.395-399

1995

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Significance of Development and Reversion of Collaterals on MRI in Early Neurologic Improvement and Long-Term Functional Outcome after Intravenous Thrombolysis for Ischemic Stroke

SUMOylation substrates in neuronal intranuclear inclusion disease

CAG repeat expansion in the TATA box-binding protein gene causes autosomal dominant cerebellar ataxia

Histone deacetylase (HDAC) 4 involvement in both Lewy and Marinesco bodies

Cytoplasmic and nuclear polyglutamine aggregates in SCA6 Purkinje cells

Similarities between spinocerebellar ataxia type 7 (SCA7) cell models and human brain: proteins recruited in inclusions and activation of caspase-3

PML nuclear bodies and neuronal intranuclear inclusion in polyglutamine diseases

Amyotrophic lateral sclerosis with neuronal intranuclear protein inclusions

A simple non-invasive method to measure the spontaneous swallowing frequency by a laryngeal microphone in Parkinson’s disease patients

Neuronal intranuclear hyaline inclusion disease with polyglutamine-immunoreactive inclusions

Preferential recruitment of ataxin-3 independent of expanded polyglutamine: an immunohistochemical study on Marinesco bodies

An Autosomal Dominant Cerebellar Ataxia Linked to Chromosome 16q22.1 Is Associated with a Single-Nucleotide Substitution in the 5′ Untranslated Region of the Gene Encoding a Protein with Spectrin Repeat and Rho Guanine-Nucleotide Exchange-Factor Domains

Huntington’s disease‐like phenotype due to trinucleotide repeat expansions in the TBP and JPH3 genes

Abundant Expression and Cytoplasmic Aggregations of α1A Voltage-Dependent Calcium Channel Protein Associated with Neurodegeneration in Spinocerebellar Ataxia Type 6

Neuronal intranuclear inclusions in spinocerebellar ataxia type 2: triple-labeling immunofluorescent study

Non-expanded polyglutamine proteins in intranuclear inclusions of hereditary ataxias - triple-labeling immunofluorescence study

Spinocerebellar Ataxia Type 6 Mutation Alters P-type Calcium Channel Function

Abundant expression and cytoplasmic aggregations of alpha 1A voltage-dependent calcium channel protein associated with neurodegeneration in spinocerebellar ataxia type 6

A clinical and genetic study in a large cohort of patients with spinocerebellar ataxia type 6

Ataxin-3 Is Translocated into the Nucleus for the Formation of Intranuclear Inclusions in Normal and Machado–Joseph Disease Brains

Recruitment of Nonexpanded Polyglutamine Proteins to Intranuclear Aggregates in Neuronal Intranuclear Hyaline Inclusion Disease

SCA12 is a rare locus for autosomal dominant cerebellar ataxia: A study of an Indian family

Two populations of neuronal intranuclear inclusions in SCA7 differ in size and promyelocytic leukaemia protein content

Spastic paraparesis and mutations in the prion protein gene

Spastic tetraplegia as an initial manifestation of familial Alzheimer's disease

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