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Preferential induction of necrosis in human breast cancer cells by a p53 peptide derived from the MDM2 binding site

Oncogene, 2003-03, Vol.22 (10), p.1431-1444

2003

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Identification of 2-piperidone as a biomarker of CYP2E1 activity through metabolomic phenotyping

Toxicological sciences, 2013-09, Vol.135 (1), p.37-47

2013

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Molecular modeling of mammalian cytochrome P450s

Frontiers in bioscience, 2004-09, Vol.9 (1-3), p.2796

2004

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PNC‐28, a p53‐derived peptide that is cytotoxic to cancer cells, blocks pancreatic cancer cell growth in vivo

International journal of cancer, 2006-10, Vol.119 (7), p.1577-1585

2006

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Peptides from the Amino Terminal mdm-2-Binding Domain of p53, Designed from Conformational Analysis, are Selectively Cytotoxic to Transformed Cells

Proceedings of the National Academy of Sciences - PNAS, 2001-10, Vol.98 (22), p.12438-12443

2001

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Comparative inhibition of human cytochromes P450 1A1 and 1A2 by flavonoids

Drug metabolism and disposition, 1998-10, Vol.26 (10), p.989

1998

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Functional Interactions of Raf and MEK with Jun-N-Terminal Kinase (JNK) Result in a Positive Feedback Loop on the Oncogenic Ras Signaling Pathway

Biochemistry (Easton), 2005-08, Vol.44 (32), p.10784-10795

2005

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Two peptides derived from ras-p21 induce either phenotypic reversion or tumor cell necrosis of ras-transformed human cancer cells

Cancer chemotherapy and pharmacology, 2008-08, Vol.62 (3), p.491-498

2008

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Rapid Conformational Dynamics of Cytochrome P450 2E1 in a Natural Biological Membrane Environment

Biochemistry (Easton), 2006-12, Vol.45 (51), p.15617-15623

2006

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The dual-specificity kinases, TOPK and DYRK1A, are critical for oocyte maturation induced by wild-type--but not by oncogenic--ras-p21 protein

Frontiers in bioscience, 2007-09, Vol.12 (12), p.5089

2007

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Conformation-Dependent Phosphorylation of p53

Proceedings of the National Academy of Sciences - PNAS, 1997-03, Vol.94 (5), p.1686-1691

1997

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Site-specific phosphorylation of raf in cells containing oncogenic ras-p21 is likely mediated by jun-N-terminal kinase

Annals of clinical and laboratory science, 2008-12, Vol.38 (1), p.47-56

2008

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Peptides designed from molecular modeling studies of the ras-p21 protein induce phenotypic reversion of a pancreatic carcinoma cell line but have no effect on normal pancreatic acinar cell growth

Cancer chemotherapy and pharmacology, 2003-09, Vol.52 (3), p.202-208

2003

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Arginine to lysine 108 substitution in recombinant CYP1A2 abolishes methoxyresorufin metabolism in lymphoblastoid cells

British journal of pharmacology, 2002-06, Vol.136 (3), p.347-352

2002

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Drug-drug interactions: Effect of quinidine on nifedipine binding to human cytochrome P450 3A4

Biochemical pharmacology, 1997-02, Vol.53 (4), p.455-460

1997

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Conformational Modulation of Human Cytochrome P450 2E1 by Ethanol and Other Substrates: A CO Flash Photolysis Study

Biochemistry (Easton), 2000-05, Vol.39 (19), p.5731-5737

2000

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An effector peptide from glutathione-S-transferase-pi strongly and selectively blocks mitotic signaling by oncogenic ras-p21

The Protein Journal, 2004-04, Vol.23 (3), p.235-238

2004

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Heme Pocket Disorder in Myoglobin: Reversal by Acid-Induced Soft Refolding

Biochemistry (Easton), 2001-10, Vol.40 (39), p.11841-11850

2001

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Loop domain peptides from the SOS ras-guanine nucleotide exchange protein, identified from molecular dynamics calculations, strongly inhibit ras signaling

The Protein Journal, 2004-04, Vol.23 (3), p.229-234

2004

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Inhibition of human liver cytochrome P-450 1A2 by the class IB antiarrhythmics mexiletine, lidocaine, and tocainide

The Journal of pharmacology and experimental therapeutics, 1999-05, Vol.289 (2), p.853

1999

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Immunochemical Analysis of Chromatin

Methods in Enzymology, 2003, Vol.376, p.209-220

2003

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Inhibition of methoxyresorufin demethylase activity by flavonoids in human liver microsomes

Life sciences (1973), 1998, Vol.63 (8), p.PL119-PL123

1998

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Comparison of molecular dynamics averaged structures for complexes of normal and oncogenic ras-p21 with SOS nucleotide exchange protein, containing computed conformations for three crystallographically undefined domains, suggests a potential role of these domains in ras signaling

The Protein Journal, 2004-04, Vol.23 (3), p.217-228

2004

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Identification of a glutathione-S-transferase effector domain for inhibition of jun kinase, by molecular dynamics

Journal of Protein Chemistry, 1999-11, Vol.18 (8), p.859-866

1999

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Molecular modeling of cytochrome P450 2B1: mode of membrane insertion and substrate specificity

Journal of Protein Chemistry, 1998-02, Vol.17 (2), p.121-129

1998

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Preferential induction of necrosis in human breast cancer cells by a p53 peptide derived from the MDM2 binding site

Identification of 2-piperidone as a biomarker of CYP2E1 activity through metabolomic phenotyping

Molecular modeling of mammalian cytochrome P450s

PNC‐28, a p53‐derived peptide that is cytotoxic to cancer cells, blocks pancreatic cancer cell growth in vivo

Peptides from the Amino Terminal mdm-2-Binding Domain of p53, Designed from Conformational Analysis, are Selectively Cytotoxic to Transformed Cells

Comparative inhibition of human cytochromes P450 1A1 and 1A2 by flavonoids

Functional Interactions of Raf and MEK with Jun-N-Terminal Kinase (JNK) Result in a Positive Feedback Loop on the Oncogenic Ras Signaling Pathway

Two peptides derived from ras-p21 induce either phenotypic reversion or tumor cell necrosis of ras-transformed human cancer cells

Rapid Conformational Dynamics of Cytochrome P450 2E1 in a Natural Biological Membrane Environment

The dual-specificity kinases, TOPK and DYRK1A, are critical for oocyte maturation induced by wild-type--but not by oncogenic--ras-p21 protein

Conformation-Dependent Phosphorylation of p53

Site-specific phosphorylation of raf in cells containing oncogenic ras-p21 is likely mediated by jun-N-terminal kinase

Peptides designed from molecular modeling studies of the ras-p21 protein induce phenotypic reversion of a pancreatic carcinoma cell line but have no effect on normal pancreatic acinar cell growth

Arginine to lysine 108 substitution in recombinant CYP1A2 abolishes methoxyresorufin metabolism in lymphoblastoid cells

Drug-drug interactions: Effect of quinidine on nifedipine binding to human cytochrome P450 3A4

Conformational Modulation of Human Cytochrome P450 2E1 by Ethanol and Other Substrates: A CO Flash Photolysis Study

An effector peptide from glutathione-S-transferase-pi strongly and selectively blocks mitotic signaling by oncogenic ras-p21

Heme Pocket Disorder in Myoglobin: Reversal by Acid-Induced Soft Refolding

Loop domain peptides from the SOS ras-guanine nucleotide exchange protein, identified from molecular dynamics calculations, strongly inhibit ras signaling

Inhibition of human liver cytochrome P-450 1A2 by the class IB antiarrhythmics mexiletine, lidocaine, and tocainide

Immunochemical Analysis of Chromatin

Inhibition of methoxyresorufin demethylase activity by flavonoids in human liver microsomes

Comparison of molecular dynamics averaged structures for complexes of normal and oncogenic ras-p21 with SOS nucleotide exchange protein, containing computed conformations for three crystallographically undefined domains, suggests a potential role of these domains in ras signaling

Identification of a glutathione-S-transferase effector domain for inhibition of jun kinase, by molecular dynamics

Molecular modeling of cytochrome P450 2B1: mode of membrane insertion and substrate specificity

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