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Dynamic and diverse changes in the functional properties of vascular smooth muscle cells in pulmonary hypertension

Cardiovascular research, 2018-03, Vol.114 (4), p.551-564

2018

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Metabolic and Proliferative State of Vascular Adventitial Fibroblasts in Pulmonary Hypertension Is Regulated Through a MicroRNA-124/PTBP1 (Polypyrimidine Tract Binding Protein 1)/Pyruvate Kinase Muscle Axis

Circulation (New York, N.Y.), 2017-12, Vol.136 (25), p.2468-2485

2017

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The adventitia: essential regulator of vascular wall structure and function

Annual review of physiology, 2013-01, Vol.75 (1), p.23-47

2013

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Identification of MicroRNA-124 as a Major Regulator of Enhanced Endothelial Cell Glycolysis in Pulmonary Arterial Hypertension via PTBP1 (Polypyrimidine Tract Binding Protein) and Pyruvate Kinase M2

Circulation (New York, N.Y.), 2017-12, Vol.136 (25), p.2451-2467

2017

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Hypoxia-Induced Pulmonary Vascular Remodeling: Cellular and Molecular Mechanisms

Circulation research, 2006-09, Vol.99 (7), p.675-691

2006

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MicroRNA-124 Controls the Proliferative, Migratory, and Inflammatory Phenotype of Pulmonary Vascular Fibroblasts

Circulation research, 2014-01, Vol.114 (1), p.67-78

2014

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Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension

The Journal of immunology (1950), 2014-07, Vol.193 (2), p.597-609

2014

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Suppression of HIF2 signalling attenuates the initiation of hypoxia-induced pulmonary hypertension

The European respiratory journal, 2019-12, Vol.54 (6), p.1900378

2019

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MicroRNA-143 Activation Regulates Smooth Muscle and Endothelial Cell Crosstalk in Pulmonary Arterial Hypertension

Circulation research, 2015-10, Vol.117 (10), p.870-883

2015

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The zinc transporter ZIP12 regulates the pulmonary vascular response to chronic hypoxia

Nature (London), 2015-08, Vol.524 (7565), p.356-360

2015

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Immunoglobulin-driven Complement Activation Regulates Proinflammatory Remodeling in Pulmonary Hypertension

American journal of respiratory and critical care medicine, 2020-01, Vol.201 (2), p.224-239

2020

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Potential long-term effects of SARS-CoV-2 infection on the pulmonary vasculature: a global perspective

Nature reviews cardiology, 2022-05, Vol.19 (5), p.314

2022

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Varicella-zoster virus vasculopathy: immune characteristics of virus-infected arteries

Neurology, 2013-01, Vol.80 (1), p.62

2013

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A Time- and Compartment-Specific Activation of Lung Macrophages in Hypoxic Pulmonary Hypertension

The Journal of immunology (1950), 2017-06, Vol.198 (12), p.4802-4812

2017

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Metabolic Reprogramming Regulates the Proliferative and Inflammatory Phenotype of Adventitial Fibroblasts in Pulmonary Hypertension Through the Transcriptional Corepressor C-Terminal Binding Protein-1

Circulation (New York, N.Y.), 2016-10, Vol.134 (15), p.1105-1121

2016

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Sustained hypoxia promotes the development of a pulmonary artery-specific chronic inflammatory microenvironment

American journal of physiology. Lung cellular and molecular physiology, 2009-08, Vol.297 (2), p.L238-L250

2009

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Local Complement Contributes to Pathogenic Activation of Lung Endothelial Cells in SARS-CoV-2 Infection

American journal of respiratory cell and molecular biology, 2023-08, Vol.69 (2), p.210-219

2023

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Microenvironmental Regulation of Macrophage Transcriptomic and Metabolomic Profiles in Pulmonary Hypertension

Frontiers in immunology, 2021-03, Vol.12, p.640718

2021

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Emergence of fibroblasts with a proinflammatory epigenetically altered phenotype in severe hypoxic pulmonary hypertension

The Journal of immunology (1950), 2011-09, Vol.187 (5), p.2711-2722

2011

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Pulmonary Arterial Stiffness: Toward a New Paradigm in Pulmonary Arterial Hypertension Pathophysiology and Assessment

Current Hypertension Reports, 2016, Vol.18 (1), p.4-4, Article 4

2016

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Mechanisms of SARS-CoV-2-induced lung vascular disease: potential role of complement

Pulmonary Circulation, 2021-04, Vol.11 (2), p.1-14

2021

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Development of an electrospun biomimetic polyurea scaffold suitable for vascular grafting

Journal of biomedical materials research. Part B, Applied biomaterials, 2018-01, Vol.106 (1), p.278-290

2018

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Functional and molecular determinants of right ventricular response to severe pulmonary hypertension in a large animal model

American journal of physiology. Heart and circulatory physiology, 2023-06, Vol.324 (6), p.H804-H820

2023

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Hypoxia-Induced Pulmonary Vascular Remodeling Requires Recruitment of Circulating Mesenchymal Precursors of a Monocyte/Macrophage Lineage

The American journal of pathology, 2006-02, Vol.168 (2), p.659-669

2006

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Osteopontin is an endogenous modulator of the constitutively activated phenotype of pulmonary adventitial fibroblasts in hypoxic pulmonary hypertension

American journal of physiology. Lung cellular and molecular physiology, 2012-07, Vol.303 (1), p.L1-L11

2012

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Dynamic and diverse changes in the functional properties of vascular smooth muscle cells in pulmonary hypertension

Metabolic and Proliferative State of Vascular Adventitial Fibroblasts in Pulmonary Hypertension Is Regulated Through a MicroRNA-124/PTBP1 (Polypyrimidine Tract Binding Protein 1)/Pyruvate Kinase Muscle Axis

The adventitia: essential regulator of vascular wall structure and function

Identification of MicroRNA-124 as a Major Regulator of Enhanced Endothelial Cell Glycolysis in Pulmonary Arterial Hypertension via PTBP1 (Polypyrimidine Tract Binding Protein) and Pyruvate Kinase M2

Hypoxia-Induced Pulmonary Vascular Remodeling: Cellular and Molecular Mechanisms

MicroRNA-124 Controls the Proliferative, Migratory, and Inflammatory Phenotype of Pulmonary Vascular Fibroblasts

Adventitial fibroblasts induce a distinct proinflammatory/profibrotic macrophage phenotype in pulmonary hypertension

Suppression of HIF2 signalling attenuates the initiation of hypoxia-induced pulmonary hypertension

MicroRNA-143 Activation Regulates Smooth Muscle and Endothelial Cell Crosstalk in Pulmonary Arterial Hypertension

The zinc transporter ZIP12 regulates the pulmonary vascular response to chronic hypoxia

Immunoglobulin-driven Complement Activation Regulates Proinflammatory Remodeling in Pulmonary Hypertension

Potential long-term effects of SARS-CoV-2 infection on the pulmonary vasculature: a global perspective

Varicella-zoster virus vasculopathy: immune characteristics of virus-infected arteries

A Time- and Compartment-Specific Activation of Lung Macrophages in Hypoxic Pulmonary Hypertension

Metabolic Reprogramming Regulates the Proliferative and Inflammatory Phenotype of Adventitial Fibroblasts in Pulmonary Hypertension Through the Transcriptional Corepressor C-Terminal Binding Protein-1

Sustained hypoxia promotes the development of a pulmonary artery-specific chronic inflammatory microenvironment

Local Complement Contributes to Pathogenic Activation of Lung Endothelial Cells in SARS-CoV-2 Infection

Microenvironmental Regulation of Macrophage Transcriptomic and Metabolomic Profiles in Pulmonary Hypertension

Emergence of fibroblasts with a proinflammatory epigenetically altered phenotype in severe hypoxic pulmonary hypertension

Pulmonary Arterial Stiffness: Toward a New Paradigm in Pulmonary Arterial Hypertension Pathophysiology and Assessment

Mechanisms of SARS-CoV-2-induced lung vascular disease: potential role of complement

Development of an electrospun biomimetic polyurea scaffold suitable for vascular grafting

Functional and molecular determinants of right ventricular response to severe pulmonary hypertension in a large animal model

Hypoxia-Induced Pulmonary Vascular Remodeling Requires Recruitment of Circulating Mesenchymal Precursors of a Monocyte/Macrophage Lineage

Osteopontin is an endogenous modulator of the constitutively activated phenotype of pulmonary adventitial fibroblasts in hypoxic pulmonary hypertension

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