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Amyloid beta-protein dimers rapidly form stable synaptotoxic protofibrils

The Journal of neuroscience, 2010-10, Vol.30 (43), p.14411-14419

2010

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Interaction between prion protein and toxic amyloid β assemblies can be therapeutically targeted at multiple sites

Nature communications, 2011-06, Vol.2 (1), p.336, Article 336

2011

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Amyloid-β nanotubes are associated with prion protein-dependent synaptotoxicity

Nature communications, 2013-09, Vol.4 (1), p.2416, Article 2416

2013

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Aβ oligomers inhibit synapse remodelling necessary for memory consolidation

Neurobiology of aging, 2011-12, Vol.32 (12), p.2211-2218

2011

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The Familial British Dementia Mutation Promotes Formation of Neurotoxic Cystine Cross-linked Amyloid Bri (ABri) Oligomers

The Journal of biological chemistry, 2015-07, Vol.290 (27), p.16502-16516

2015

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Amyloid β peptide as a physiological modulator of neuronal ‘A’-type K + current

Neurobiology of aging, 2006-11, Vol.27 (11), p.1673-1683

2006

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Inhibition of LTP in vivo by beta-amyloid peptide in different conformational states

Brain research, 2008-03, Vol.1197, p.135-142

2008

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Inhibition of l-type voltage dependent calcium channels causes impairment of long-term potentiation in the hippocampal CA1 region in vivo

Brain research, 2003-03, Vol.967 (1), p.27-36

2003

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Assessment of the adequacy of counselling regarding reproductive-related issues in women of childbearing age on anti-epileptic drugs

QJM : An International Journal of Medicine, 2021-07, Vol.114 (4), p.233-237

2021

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Blockade of long-term potentiation by beta-amyloid peptides in the CA1 region of the rat hippocampus in vivo

Journal of neurophysiology, 2001-02, Vol.85 (2), p.708-713

2001

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A[beta] oligomers inhibit synapse remodelling necessary for memory consolidation

Neurobiology of aging, 2011-12, Vol.32 (12), p.2211-2218

2011

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A Delta b oligomers inhibit synapse remodelling necessary for memory consolidation

Neurobiology of aging, 2011-12, Vol.32 (12), p.2211-2218

2011

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Amyloid b-Protein Dimers Rapidly Form Stable Synaptotoxic Protofibrils

The Journal of neuroscience, 2010-10, Vol.30 (43), p.14411-14419

2010

Volltextzugriff (PDF)

Inhibition of LTP in uiuo by beta-amyloid peptide in different conformational states

Brain research, 2008, Vol.1197, p.135-142

2008

Volltextzugriff (PDF)

Amyloid beta peptide as a physiological modulator of neuronal A-type K super(+) current

Neurobiology of aging, 2006-11, Vol.27 (11), p.1673-1683

2006

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Amyloid-[beta] nanotubes are associated with prion protein-dependent synaptotoxicity

Nature communications, 2013-09, Vol.4, p.2416

2013

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Blockade of Long-Term Potentiation by {beta}-Amyloid Peptides in the CA1 Region of the Rat Hippocampus In Vivo

Journal of neurophysiology, 2001-02, Vol.85 (2), p.708

2001

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Interaction between prion protein and toxic amyloid [beta] assemblies can be therapeutically targeted at multiple sites

Nature communications, 2011-06, Vol.2, p.336

2011

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Amyloid beta-protein dimers rapidly form stable synaptotoxic protofibrils

Interaction between prion protein and toxic amyloid β assemblies can be therapeutically targeted at multiple sites

Amyloid-β nanotubes are associated with prion protein-dependent synaptotoxicity

Aβ oligomers inhibit synapse remodelling necessary for memory consolidation

The Familial British Dementia Mutation Promotes Formation of Neurotoxic Cystine Cross-linked Amyloid Bri (ABri) Oligomers

Amyloid β peptide as a physiological modulator of neuronal ‘A’-type K + current

Inhibition of LTP in vivo by beta-amyloid peptide in different conformational states

Inhibition of l-type voltage dependent calcium channels causes impairment of long-term potentiation in the hippocampal CA1 region in vivo

Assessment of the adequacy of counselling regarding reproductive-related issues in women of childbearing age on anti-epileptic drugs

Blockade of long-term potentiation by beta-amyloid peptides in the CA1 region of the rat hippocampus in vivo

A[beta] oligomers inhibit synapse remodelling necessary for memory consolidation

A Delta b oligomers inhibit synapse remodelling necessary for memory consolidation

Amyloid b-Protein Dimers Rapidly Form Stable Synaptotoxic Protofibrils

Inhibition of LTP in uiuo by beta-amyloid peptide in different conformational states

Amyloid beta peptide as a physiological modulator of neuronal A-type K super(+) current

Amyloid-[beta] nanotubes are associated with prion protein-dependent synaptotoxicity

Blockade of Long-Term Potentiation by {beta}-Amyloid Peptides in the CA1 Region of the Rat Hippocampus In Vivo

Interaction between prion protein and toxic amyloid [beta] assemblies can be therapeutically targeted at multiple sites

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