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Medical device‐induced thrombosis: what causes it and how can we prevent it?

Journal of thrombosis and haemostasis, 2015-06, Vol.13 (S1), p.S72-S81

2015

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Comparison of the ecarin chromogenic assay and diluted thrombin time for quantification of dabigatran concentrations

Journal of thrombosis and haemostasis, 2017-12, Vol.15 (12), p.2377-2387

2017

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A Protease TAMER: a nucleic acid‐based anticoagulant

Journal of thrombosis and haemostasis, 2012-05, Vol.10 (5), p.867-869

2012

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Zinc: an important cofactor in haemostasis and thrombosis

Thrombosis and haemostasis, 2013-03, Vol.109 (3), p.421

2013

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Mechanism of action of plasminogen activators

Thrombosis and haemostasis, 1999-08, Vol.82 (2), p.974

1999

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Effect of nonspecific binding to plasma proteins on the antithrombin activities of unfractionated heparin, low-molecular-weight heparin, and dermatan sulfate

Circulation (New York, N.Y.), 1997-01, Vol.95 (1), p.118-124

1997

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Vasoflux, a new anticoagulant with a novel mechanism of action

Circulation (New York, N.Y.), 1999-02, Vol.99 (5), p.682-689

1999

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Molecular Basis for the Susceptibility of Fibrin-bound Thrombin to Inactivation by Heparin Cofactor II in the Presence of Dermatan Sulfate but Not Heparin

The Journal of biological chemistry, 2001-06, Vol.276 (24), p.20959-20965

2001

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The activated protein C-mediated enhancement of tissue-type plasminogen activator-induced fibrinolysis in a cell-free system

The Journal of biological chemistry, 1990-10, Vol.265 (28), p.16948-16954

1990

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Lys-plasminogen is a significant intermediate in the activation of Glu-plasminogen during fibrinolysis in vitro

The Journal of biological chemistry, 1992-12, Vol.267 (36), p.26150-26156

1992

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The dissociation constants and stoichiometries of the interactions of Lys-plasminogen and chloromethyl ketone derivatives of tissue plasminogen activator and the variant delta FEIX with intact fibrin

The Journal of biological chemistry, 1990-12, Vol.265 (35), p.21541-21548

1990

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Evidence for Allosteric Linkage between Exosites 1 and 2 of Thrombin

The Journal of biological chemistry, 1997-10, Vol.272 (41), p.25493-25499

1997

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Exosites 1 and 2 Are Essential for Protection of Fibrin-bound Thrombin from Heparin-catalyzed Inhibition by Antithrombin and Heparin Cofactor II

The Journal of biological chemistry, 1999-03, Vol.274 (10), p.6226-6233

1999

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Comparison of Heparin- and Dermatan Sulfate-mediated Catalysis of Thrombin Inactivation by Heparin Cofactor II

The Journal of biological chemistry, 1999-09, Vol.274 (39), p.27597-27604

1999

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Only high levels of dabigatran attenuate catheter thrombosis in vitro and in rabbits

Thrombosis and haemostasis, 2014, Vol.112 (1), p.79

2014

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Identification of the Mechanism Responsible for the Increased Fibrin Specificity of TNK-Tissue Plasminogen Activator Relative to Tissue Plasminogen Activator

The Journal of biological chemistry, 2000-04, Vol.275 (14), p.10112-10120

2000

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Conformational Changes in Thrombin When Complexed by Serpins

The Journal of biological chemistry, 2001-11, Vol.276 (48), p.44828-44834

2001

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Characterization of the Interactions of Plasminogen and Tissue and Vampire Bat Plasminogen Activators with Fibrinogen, Fibrin, and the Complex of d-Dimer Noncovalently Linked to Fragment E

The Journal of biological chemistry, 1998-07, Vol.273 (29), p.18292-18299

1998

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HD1, a thrombin- and prothrombin-binding DNA aptamer, inhibits thrombin generation by attenuating prothrombin activation and thrombin feedback reactions

Thrombosis and haemostasis, 2010-01, Vol.103 (1), p.83

2010

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Hypersulfated Low Molecular Weight Heparin with Reduced Affinity for Antithrombin Acts as an Anticoagulant by Inhibiting Intrinsic Tenase and Prothrombinase

The Journal of biological chemistry, 2001-03, Vol.276 (13), p.9755-9761

2001

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Thrombin-activable fibrinolysis inhibitor attenuates (DD)E-mediated stimulation of plasminogen activation by reducing the affinity of (DD)E for tissue plasminogen activator. A potential mechanism for enhancing the fibrin specificity of tissue plasminogen activator

The Journal of biological chemistry, 2000-11, Vol.275 (47), p.36612

2000

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Localization of the Thrombin-binding Domain on Prothrombin Fragment 2

The Journal of biological chemistry, 1998-04, Vol.273 (15), p.8932-8939

1998

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Clot waveform analysis in patients with haemophilia A

Hämostaseologie, 2014-01, Vol.34 Suppl 1, p.S48

2014

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The Activation-resistant Conformation of Recombinant Human Plasminogen Is Stabilized by Basic Residues in the Amino-terminal Hinge Region (∗)

The Journal of biological chemistry, 1995-06, Vol.270 (26), p.15770-15776

1995

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Evidence That Both Exosites on Thrombin Participate in Its High Affinity Interaction with Fibrin

The Journal of biological chemistry, 2003-06, Vol.278 (24), p.21584-21591

2003

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Medical device‐induced thrombosis: what causes it and how can we prevent it?

Comparison of the ecarin chromogenic assay and diluted thrombin time for quantification of dabigatran concentrations

A Protease TAMER: a nucleic acid‐based anticoagulant

Zinc: an important cofactor in haemostasis and thrombosis

Mechanism of action of plasminogen activators

Effect of nonspecific binding to plasma proteins on the antithrombin activities of unfractionated heparin, low-molecular-weight heparin, and dermatan sulfate

Vasoflux, a new anticoagulant with a novel mechanism of action

Molecular Basis for the Susceptibility of Fibrin-bound Thrombin to Inactivation by Heparin Cofactor II in the Presence of Dermatan Sulfate but Not Heparin

The activated protein C-mediated enhancement of tissue-type plasminogen activator-induced fibrinolysis in a cell-free system

Lys-plasminogen is a significant intermediate in the activation of Glu-plasminogen during fibrinolysis in vitro

The dissociation constants and stoichiometries of the interactions of Lys-plasminogen and chloromethyl ketone derivatives of tissue plasminogen activator and the variant delta FEIX with intact fibrin

Evidence for Allosteric Linkage between Exosites 1 and 2 of Thrombin

Exosites 1 and 2 Are Essential for Protection of Fibrin-bound Thrombin from Heparin-catalyzed Inhibition by Antithrombin and Heparin Cofactor II

Comparison of Heparin- and Dermatan Sulfate-mediated Catalysis of Thrombin Inactivation by Heparin Cofactor II

Only high levels of dabigatran attenuate catheter thrombosis in vitro and in rabbits

Identification of the Mechanism Responsible for the Increased Fibrin Specificity of TNK-Tissue Plasminogen Activator Relative to Tissue Plasminogen Activator

Conformational Changes in Thrombin When Complexed by Serpins

Characterization of the Interactions of Plasminogen and Tissue and Vampire Bat Plasminogen Activators with Fibrinogen, Fibrin, and the Complex of d-Dimer Noncovalently Linked to Fragment E

HD1, a thrombin- and prothrombin-binding DNA aptamer, inhibits thrombin generation by attenuating prothrombin activation and thrombin feedback reactions

Hypersulfated Low Molecular Weight Heparin with Reduced Affinity for Antithrombin Acts as an Anticoagulant by Inhibiting Intrinsic Tenase and Prothrombinase

Thrombin-activable fibrinolysis inhibitor attenuates (DD)E-mediated stimulation of plasminogen activation by reducing the affinity of (DD)E for tissue plasminogen activator. A potential mechanism for enhancing the fibrin specificity of tissue plasminogen activator

Localization of the Thrombin-binding Domain on Prothrombin Fragment 2

Clot waveform analysis in patients with haemophilia A

The Activation-resistant Conformation of Recombinant Human Plasminogen Is Stabilized by Basic Residues in the Amino-terminal Hinge Region (∗)

Evidence That Both Exosites on Thrombin Participate in Its High Affinity Interaction with Fibrin

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