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From basic research to clinical development of MEK1/2 inhibitors for cancer therapy

Journal of hematology and oncology, 2010-02, Vol.3 (1), p.8-8, Article 8

2010

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Phosphoproteome dynamics reveal novel ERK1/2 MAP kinase substrates with broad spectrum of functions

Molecular systems biology, 2013-05, Vol.9 (1), p.669-n/a

2013

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Genetic Demonstration of a Redundant Role of Extracellular Signal-Regulated Kinase 1 (ERK1) and ERK2 Mitogen-Activated Protein Kinases in Promoting Fibroblast Proliferation

Molecular and Cellular Biology, 2010-06, Vol.30 (12), p.2918-2932

2010

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Fibrillar collagen scoring by second harmonic microscopy: A new tool in the assessment of liver fibrosis

Journal of hepatology, 2010-03, Vol.52 (3), p.398-406

2010

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Functional Redundancy of ERK1 and ERK2 MAP Kinases during Development

Cell reports (Cambridge), 2015-08, Vol.12 (6), p.913-921

2015

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Redundancy in the World of MAP Kinases: All for One

Frontiers in cell and developmental biology, 2016-06, Vol.4, p.67-67

2016

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RNAi‐mediated MEK1 knock‐down prevents ERK1/2 activation and abolishes human hepatocarcinoma growth in vitro and in vivo

International journal of cancer, 2010-03, Vol.126 (6), p.1367-1377

2010

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Multiple division cycles and long‐term survival of hepatocytes are distinctly regulated by extracellular signal‐regulated kinases ERK1 and ERK2

Hepatology (Baltimore, Md.), 2009-03, Vol.49 (3), p.930-939

2009

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The complexity of ERK1 and ERK2 MAPKs in multiple hepatocyte fate responses

Journal of cellular physiology, 2012-01, Vol.227 (1), p.59-69

2012

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Isolation of Mouse Embryonic Stem Cell Lines in the Study of ERK1/2 MAP Kinase Signaling

Methods in molecular biology (Clifton, N.J.), 2017-01, Vol.1487, p.243

2017

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The MAPK MEK1/2-ERK1/2 Pathway and Its Implication in Hepatocyte Cell Cycle Control

International journal of hepatology (New York), 2012-01, Vol.2012, p.328372-13

2012

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ERK2 but not ERK1 plays a key role in hepatocyte replication: An RNAi‐mediated ERK2 knockdown approach in wild‐type and ERK1 null hepatocytes

Hepatology (Baltimore, Md.), 2007-04, Vol.45 (4), p.1035-1045

2007

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MEK/ERK-dependent uPAR expression is required for motility via phosphorylation of P70S6K in human hepatocarcinoma cells

Journal of cellular physiology, 2007-08, Vol.212 (2), p.526-536

2007

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RNAi-mediated ERK2 knockdown inhibits growth of tumor cells in vitro and in vivo

Oncogene, 2008-09, Vol.27 (40), p.5315-5325

2008

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The knock-down of ERCC1 but not of XPF causes multinucleation

DNA repair, 2011-09, Vol.10 (9), p.978-990

2011

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An MLCK‐dependent window in late G1 controls S phase entry of proliferating rodent hepatocytes via ERK‐p70S6K pathway

Hepatology (Baltimore, Md.), 2006-07, Vol.44 (1), p.152-163

2006

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ERK2 but not ERK1 plays a key role in hepatocyte replication: an RNAi-mediated ERK2 knockdown approach in wild-type and ERK1 null hepatocytes

Hepatology (Baltimore, Md.), 2007-04, Vol.45 (4), p.1035-45

2007

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From basic research to clinical development of MEK1/2 inhibitors for cancer therapy

Phosphoproteome dynamics reveal novel ERK1/2 MAP kinase substrates with broad spectrum of functions

Genetic Demonstration of a Redundant Role of Extracellular Signal-Regulated Kinase 1 (ERK1) and ERK2 Mitogen-Activated Protein Kinases in Promoting Fibroblast Proliferation

Fibrillar collagen scoring by second harmonic microscopy: A new tool in the assessment of liver fibrosis

Functional Redundancy of ERK1 and ERK2 MAP Kinases during Development

Redundancy in the World of MAP Kinases: All for One

RNAi‐mediated MEK1 knock‐down prevents ERK1/2 activation and abolishes human hepatocarcinoma growth in vitro and in vivo

Multiple division cycles and long‐term survival of hepatocytes are distinctly regulated by extracellular signal‐regulated kinases ERK1 and ERK2

The complexity of ERK1 and ERK2 MAPKs in multiple hepatocyte fate responses

Isolation of Mouse Embryonic Stem Cell Lines in the Study of ERK1/2 MAP Kinase Signaling

The MAPK MEK1/2-ERK1/2 Pathway and Its Implication in Hepatocyte Cell Cycle Control

ERK2 but not ERK1 plays a key role in hepatocyte replication: An RNAi‐mediated ERK2 knockdown approach in wild‐type and ERK1 null hepatocytes

MEK/ERK-dependent uPAR expression is required for motility via phosphorylation of P70S6K in human hepatocarcinoma cells

RNAi-mediated ERK2 knockdown inhibits growth of tumor cells in vitro and in vivo

The knock-down of ERCC1 but not of XPF causes multinucleation

An MLCK‐dependent window in late G1 controls S phase entry of proliferating rodent hepatocytes via ERK‐p70S6K pathway

ERK2 but not ERK1 plays a key role in hepatocyte replication: an RNAi-mediated ERK2 knockdown approach in wild-type and ERK1 null hepatocytes

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