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Anti-apoptotic function of a microRNA encoded by the HSV-1 latency-associated transcript

Nature, 2006-07, Vol.442 (7098), p.82-85

2006

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During Lytic Infection Herpes Simplex Virus Type 1 Is Associated with Histones Bearing Modifications That Correlate with Active Transcription

Journal of Virology, 2004-09, Vol.78 (18), p.10178-10186

2004

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Long-term AAV vector gene and protein expression in mouse brain from a small pan-cellular promoter is similar to neural cell promoters

Gene therapy, 2009-07, Vol.16 (7), p.927-932

2009

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Regions of the Herpes Simplex Virus Type 1 Latency-Associated Transcript That Protect Cells from Apoptosis In Vitro and Protect Neuronal Cells In Vivo

Journal of Virology, 2002-01, Vol.76 (2), p.717-729

2002

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Anti-apoptotic function of a microRNA encoded by the HSV-1 latency-associated transcript

Nature (London), 2008-01, Vol.451 (7178), p.600-600

2008

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During latency, herpes simplex virus type 1 DNA is associated with nucleosomes in a chromatin structure

Journal of Virology, 1989-02, Vol.63 (2), p.943-947

1989

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The cellular response to herpes simplex virus type 1 (HSV-1) during latency and reactivation

Journal of neurovirology, 2005-08, Vol.11 (4), p.376-383

2005

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Stable Levels of Long-Term Transgene Expression Driven by the Latency-Associated Transcript Promoter in a Herpes Simplex Virus Type 1 Vector

Molecular therapy, 2005-12, Vol.12 (6), p.1111-1119

2005

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Detection of herpes simplex virus type 1 transcripts during latent infection in mice

Journal of Virology, 1987-12, Vol.61 (12), p.3841-3847

1987

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Herpes Simplex Virus Type 1 2-Kilobase Latency-Associated Transcript Intron Associates with Ribosomal Proteins and Splicing Factors

Journal of Virology, 2001-12, Vol.75 (24), p.12070-12080

2001

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Therapy of experimental human brain tumors using a neuroattenuated herpes simplex virus mutant

Laboratory investigation, 1995-11, Vol.73 (5), p.636

1995

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Gene expression during reactivation of herpes simplex virus type 1 from latency in the peripheral nervous system is different from that during lytic infection of tissue cultures

Journal of Virology, 1997-07, Vol.71 (7), p.5268-5276

1997

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The herpes simplex virus type 1 2.0-kilobase latency-associated transcript is a stable intron which branches at a guanosine

Journal of Virology, 1997-06, Vol.71 (6), p.4199-4208

1997

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Use of a replication-restricted herpes virus to treat experimental human malignant mesothelioma

Cancer research (Chicago, Ill.), 1997-02, Vol.57 (3), p.466-471

1997

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RNA from an Immediate Early Region of the Type 1 Herpes Simplex Virus Genome is Present in the Trigeminal Ganglia of Latently Infected Mice

Proceedings of the National Academy of Sciences - PNAS, 1987-05, Vol.84 (10), p.3204-3208

1987

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The 2-Kilobase Intron of the Herpes Simplex Virus Type 1 Latency-Associated Transcript Has a Half-Life of Approximately 24 Hours in SY5Y and COS-1 Cells

Journal of Virology, 2002-01, Vol.76 (2), p.532-540

2002

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Expression of herpes simplex virus type 1 latency-associated transcripts in the trigeminal ganglia of mice during acute infection and reactivation of latent infection

Journal of Virology, 1988-05, Vol.62 (5), p.1479-1485

1988

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Herpes simplex virus type 1 latency‐associated transcripts are evidently not essential for latent infection

The EMBO journal, 1989-02, Vol.8 (2), p.505-511

1989

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Analysis of Protein Expression from within the Region Encoding the 2.0-Kilobase Latency-Associated Transcript of Herpes Simplex Virus Type 1

Journal of Virology, 2001-04, Vol.75 (7), p.3413-3426

2001

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The latency-associated transcripts of herpes simplex virus: RNA in search of function

Virology, 1992-11, Vol.191 (1), p.1-8

1992

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Induction of cellular transcription factors in trigeminal ganglia of mice by corneal scarification, herpes simplex virus type 1 infection, and explantation of trigeminal ganglia

Journal of Virology, 1991-08, Vol.65 (8), p.4142-4152

1991

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Transfectable and Transplantable Postmitotic Human Neurons: A Potential “Platform” for Gene Therapy of Nervous System Diseases

Experimental neurology, 1997-03, Vol.144 (1), p.92-97

1997

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Selection of a nonconsensus branch point is influenced by an RNA stem-loop structure and is important to confer stability to the herpes simplex virus 2-kilobase latency-associated transcript

Journal of Virology, 1997-08, Vol.71 (8), p.5849-5860

1997

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Physical state of the latent herpes simplex virus genome in a mouse model system: Evidence suggesting an episomal state

Virology (New York, N.Y.), 1987-06, Vol.158 (2), p.265-275

1987

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Treatment of Experimental Subcutaneous Human Melanoma with a Replication-Restricted Herpes Simplex Virus Mutant

Journal of investigative dermatology, 1997-06, Vol.108 (6), p.933-937

1997

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Anti-apoptotic function of a microRNA encoded by the HSV-1 latency-associated transcript

During Lytic Infection Herpes Simplex Virus Type 1 Is Associated with Histones Bearing Modifications That Correlate with Active Transcription

Long-term AAV vector gene and protein expression in mouse brain from a small pan-cellular promoter is similar to neural cell promoters

Regions of the Herpes Simplex Virus Type 1 Latency-Associated Transcript That Protect Cells from Apoptosis In Vitro and Protect Neuronal Cells In Vivo

Anti-apoptotic function of a microRNA encoded by the HSV-1 latency-associated transcript

During latency, herpes simplex virus type 1 DNA is associated with nucleosomes in a chromatin structure

The cellular response to herpes simplex virus type 1 (HSV-1) during latency and reactivation

Stable Levels of Long-Term Transgene Expression Driven by the Latency-Associated Transcript Promoter in a Herpes Simplex Virus Type 1 Vector

Detection of herpes simplex virus type 1 transcripts during latent infection in mice

Herpes Simplex Virus Type 1 2-Kilobase Latency-Associated Transcript Intron Associates with Ribosomal Proteins and Splicing Factors

Therapy of experimental human brain tumors using a neuroattenuated herpes simplex virus mutant

Gene expression during reactivation of herpes simplex virus type 1 from latency in the peripheral nervous system is different from that during lytic infection of tissue cultures

The herpes simplex virus type 1 2.0-kilobase latency-associated transcript is a stable intron which branches at a guanosine

Use of a replication-restricted herpes virus to treat experimental human malignant mesothelioma

RNA from an Immediate Early Region of the Type 1 Herpes Simplex Virus Genome is Present in the Trigeminal Ganglia of Latently Infected Mice

The 2-Kilobase Intron of the Herpes Simplex Virus Type 1 Latency-Associated Transcript Has a Half-Life of Approximately 24 Hours in SY5Y and COS-1 Cells

Expression of herpes simplex virus type 1 latency-associated transcripts in the trigeminal ganglia of mice during acute infection and reactivation of latent infection

Herpes simplex virus type 1 latency‐associated transcripts are evidently not essential for latent infection

Analysis of Protein Expression from within the Region Encoding the 2.0-Kilobase Latency-Associated Transcript of Herpes Simplex Virus Type 1

The latency-associated transcripts of herpes simplex virus: RNA in search of function

Induction of cellular transcription factors in trigeminal ganglia of mice by corneal scarification, herpes simplex virus type 1 infection, and explantation of trigeminal ganglia

Transfectable and Transplantable Postmitotic Human Neurons: A Potential “Platform” for Gene Therapy of Nervous System Diseases

Selection of a nonconsensus branch point is influenced by an RNA stem-loop structure and is important to confer stability to the herpes simplex virus 2-kilobase latency-associated transcript

Physical state of the latent herpes simplex virus genome in a mouse model system: Evidence suggesting an episomal state

Treatment of Experimental Subcutaneous Human Melanoma with a Replication-Restricted Herpes Simplex Virus Mutant

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