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Arrhythmogenic late Ca2+ sparks in failing heart cells and their control by action potential configuration

Proceedings of the National Academy of Sciences - PNAS, 2020-02, Vol.117 (5), p.2687-2692

2020

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Improved Ca2+ release synchrony following selective modification of Itof and phase 1 repolarization in normal and failing ventricular myocytes

Journal of molecular and cellular cardiology, 2022-11, Vol.172, p.52-62

2022

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Inducing Ito,f and phase 1 repolarization of the cardiac action potential with a Kv4.3/KChIP2.1 bicistronic transgene

Journal of molecular and cellular cardiology, 2022-03, Vol.164, p.29-41

2022

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Energy Metabolism in the Failing Right Ventricle: Limitations of Oxygen Delivery and the Creatine Kinase System

International journal of molecular sciences, 2019-04, Vol.20 (8), p.1805

2019

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Molecular, Subcellular, and Arrhythmogenic Mechanisms in Genetic RyR2 Disease

Biomolecules (Basel, Switzerland), 2022-07, Vol.12 (8), p.1030

2022

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Voluntary exercise delays heart failure onset in rats with pulmonary artery hypertension

American journal of physiology. Heart and circulatory physiology, 2015-08, Vol.309 (3), p.H421-H424

2015

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Diastolic dysfunction in pulmonary artery hypertension: Creatine kinase and the potential therapeutic benefit of beta‐blockers

Clinical and experimental pharmacology & physiology, 2018-04, Vol.45 (4), p.384-389

2018

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Beta1-adrenoceptor antagonist, metoprolol attenuates cardiac myocyte Ca2+ handling dysfunction in rats with pulmonary artery hypertension

Journal of molecular and cellular cardiology, 2018-07, Vol.120, p.74-83

2018

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Arrhythmogenic late Ca 2+ sparks in failing heart cells and their control by action potential configuration

Proceedings of the National Academy of Sciences - PNAS, 2020-02, Vol.117 (5), p.2687-2692

2020

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Synergistic role of ADP and Ca(2+) in diastolic myocardial stiffness

The Journal of physiology, 2015-09, Vol.593 (17), p.3899

2015

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Synergistic role of ADP and Ca2+ in diastolic myocardial stiffness

The Journal of physiology, 2015-09, Vol.593 (17), p.3899-3916

2015

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Late Ca2+ Sparks and Ripples During the Systolic Ca2+ Transient in Heart Muscle Cells

Circulation research, 2018-02, Vol.122 (3), p.473-478

2018

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Decreased creatine kinase is linked to diastolic dysfunction in rats with right heart failure induced by pulmonary artery hypertension

Journal of molecular and cellular cardiology, 2015-09, Vol.86, p.1-8

2015

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A tubulin alpha 8 mouse knockout model indicates a likely role in spermatogenesis but not in brain development

PloS one, 2017-04, Vol.12 (4), p.e0174264

2017

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Late Ca 2+ Sparks and Ripples During the Systolic Ca 2+ Transient in Heart Muscle Cells

Circulation research, 2018-02, Vol.122 (3), p.473-478

2018

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Editorial: Stretch and the heart: mechanoelectrical coupling and arrhythmias

Frontiers in physiology, 2023-08, Vol.14, p.1278561-1278561

2023

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Systems approach to the study of stretch and arrhythmias in right ventricular failure induced in rats by monocrotaline

Progress in biophysics and molecular biology, 2014-08, Vol.115 (2-3), p.162-172

2014

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Late Ca2+Spark Arrhythmogenesis in Failing Cardiac Myocytes

Biophysical journal, 2020-02, Vol.118 (3), p.569a-569a

2020

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Shaping Action Potential Repolarization Phase I by Stoichiometric Expression of Kv4.3/KChIP2.1

Biophysical journal, 2020-02, Vol.118 (3), p.101a-101a

2020

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The β1-Adrenergic Receptor Blocker, Metoprolol, Improves Survival and Electrical Remodeling in Rats with Pulmonary Artery Hypertension

Biophysical journal, 2016-02, Vol.110 (3), p.478a-478a

2016

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Metoprolol Reverses β-Adrenergic Remodeling in the Failing Right Ventricle of Pulmonary Artery Hypertensive (PAH) Rats

Biophysical journal, 2016-02, Vol.110 (3), p.89a-90a

2016

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Re-triggerability of Ca2+ Sparks Following Evoked Ca2+ Release

Biophysical journal, 2018-02, Vol.114 (3), p.289a-289a

2018

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Intensity-dependence of exercise and active recovery in high-intensity interval training

Journal of sports medicine and physical fitness, 2019-12, Vol.59 (12), p.1937

2019

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Improved Ca 2+ release synchrony following selective modification of I tof and phase 1 repolarization in normal and failing ventricular myocytes

Journal of molecular and cellular cardiology, 2022-11, Vol.172, p.52

2022

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Inducing I to,f and phase 1 repolarization of the cardiac action potential with a Kv4.3/KChIP2.1 bicistronic transgene

Journal of molecular and cellular cardiology, 2022-03, Vol.164, p.29

2022

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Arrhythmogenic late Ca2+ sparks in failing heart cells and their control by action potential configuration

Improved Ca2+ release synchrony following selective modification of Itof and phase 1 repolarization in normal and failing ventricular myocytes

Inducing Ito,f and phase 1 repolarization of the cardiac action potential with a Kv4.3/KChIP2.1 bicistronic transgene

Energy Metabolism in the Failing Right Ventricle: Limitations of Oxygen Delivery and the Creatine Kinase System

Molecular, Subcellular, and Arrhythmogenic Mechanisms in Genetic RyR2 Disease

Voluntary exercise delays heart failure onset in rats with pulmonary artery hypertension

Diastolic dysfunction in pulmonary artery hypertension: Creatine kinase and the potential therapeutic benefit of beta‐blockers

Beta1-adrenoceptor antagonist, metoprolol attenuates cardiac myocyte Ca2+ handling dysfunction in rats with pulmonary artery hypertension

Arrhythmogenic late Ca 2+ sparks in failing heart cells and their control by action potential configuration

Synergistic role of ADP and Ca(2+) in diastolic myocardial stiffness

Synergistic role of ADP and Ca2+ in diastolic myocardial stiffness

Late Ca2+ Sparks and Ripples During the Systolic Ca2+ Transient in Heart Muscle Cells

Decreased creatine kinase is linked to diastolic dysfunction in rats with right heart failure induced by pulmonary artery hypertension

A tubulin alpha 8 mouse knockout model indicates a likely role in spermatogenesis but not in brain development

Late Ca 2+ Sparks and Ripples During the Systolic Ca 2+ Transient in Heart Muscle Cells

Editorial: Stretch and the heart: mechanoelectrical coupling and arrhythmias

Systems approach to the study of stretch and arrhythmias in right ventricular failure induced in rats by monocrotaline

Late Ca2+Spark Arrhythmogenesis in Failing Cardiac Myocytes

Shaping Action Potential Repolarization Phase I by Stoichiometric Expression of Kv4.3/KChIP2.1

The β1-Adrenergic Receptor Blocker, Metoprolol, Improves Survival and Electrical Remodeling in Rats with Pulmonary Artery Hypertension

Metoprolol Reverses β-Adrenergic Remodeling in the Failing Right Ventricle of Pulmonary Artery Hypertensive (PAH) Rats

Re-triggerability of Ca2+ Sparks Following Evoked Ca2+ Release

Intensity-dependence of exercise and active recovery in high-intensity interval training

Improved Ca 2+ release synchrony following selective modification of I tof and phase 1 repolarization in normal and failing ventricular myocytes

Inducing I to,f and phase 1 repolarization of the cardiac action potential with a Kv4.3/KChIP2.1 bicistronic transgene

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