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Cholesterol Efflux Pathways Suppress Inflammasome Activation, NETosis, and Atherogenesis

Circulation (New York, N.Y.), 2018-08, Vol.138 (9), p.898-912

2018

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Anti-Inflammatory Effects of HDL (High-Density Lipoprotein) in Macrophages Predominate Over Proinflammatory Effects in Atherosclerotic Plaques

Arteriosclerosis, thrombosis, and vascular biology, 2019-12, Vol.39 (12), p.e253

2019

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Cholesterol Accumulation in Dendritic Cells Links the Inflammasome to Acquired Immunity

Cell metabolism, 2017-06, Vol.25 (6), p.1294-1304.e6

2017

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Deficiency of ATP-Binding Cassette Transporters A1 and G1 in Endothelial Cells Accelerates Atherosclerosis in Mice

Arteriosclerosis, thrombosis, and vascular biology, 2016-07, Vol.36 (7), p.1328

2016

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HDL-apoA-I induces the expression of angiopoietin like 4 (ANGPTL4) in endothelial cells via a PI3K/AKT/FOXO1 signaling pathway

Metabolism, clinical and experimental, 2018-10, Vol.87, p.36-47

2018

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LXR Suppresses Inflammatory Gene Expression and Neutrophil Migration through cis-Repression and Cholesterol Efflux

Cell reports (Cambridge), 2018-12, Vol.25 (13), p.3774-3785.e4

2018

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Reconstituted HDL-apoE3 promotes endothelial cell migration through ID1 and its downstream kinases ERK1/2, AKT and p38 MAPK

Metabolism, clinical and experimental, 2022-02, Vol.127, p.154954-154954, Article 154954

2022

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Role of apolipoproteins, ABCA1 and LCAT in the biogenesis of normal and aberrant high density lipoproteins

Journal of biomedical research, 2017-01, Vol.31 (6), p.471-485

2017

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ApoA-IV promotes the biogenesis of apoA-IV-containing HDL particles with the participation of ABCA1 and LCAT[S]

Journal of lipid research, 2013-01, Vol.54 (1), p.107-115

2013

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apoE3[K146N/R147W] acts as a dominant negative apoE form that prevents remnant clearance and inhibits the biogenesis of HDL[S]

Journal of lipid research, 2014-07, Vol.55 (7), p.1310-1323

2014

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Significance of the hydrophobic residues 225–230 of apoA-I for the biogenesis of HDL1[S]

Journal of lipid research, 2013-12, Vol.54 (12), p.3293-3302

2013

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Role of the hydrophobic and charged residues in the 218–226 region of apoA-I in the biogenesis of HDL1[S]

Journal of lipid research, 2013-12, Vol.54 (12), p.3281-3292

2013

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Cholesterol accumulation in macrophages drives NETosis in atherosclerotic plaques via IL-1β secretion

Cardiovascular research, 2023-05, Vol.119 (4), p.969-981

2023

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HDL biogenesis, remodeling, and catabolism

Handbook of experimental pharmacology, 2015, Vol.224, p.53

2015

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Response by Fotakis et al to Letter Regarding Article, "Anti-Inflammatory Effects of HDL (High-Density Lipoprotein) in Macrophages Predominate Over Proinflammatory Effects in Atherosclerotic Plaques"

Arteriosclerosis, thrombosis, and vascular biology, 2020-02, Vol.40 (2), p.e33

2020

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TTC39B destabilizes retinoblastoma protein promoting hepatic lipogenesis in a sex-specific fashion

Journal of hepatology, 2022-02, Vol.76 (2), p.383-393

2022

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The Effect of Natural LCAT Mutations on the Biogenesis of HDL

Biochemistry (Easton), 2015-06, Vol.54 (21), p.3348-3359

2015

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Natural human apoA-I mutations L141RPisa and L159RFIN alter HDL structure and functionality and promote atherosclerosis development in mice

Atherosclerosis, 2015-11, Vol.243 (1), p.77-85

2015

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Natural human apoA-I mutations L141R Pisa and L159R FIN alter HDL structure and functionality and promote atherosclerosis development in mice

Atherosclerosis, 2015-11, Vol.243 (1), p.77-85

2015

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Tissue Uptake Mechanisms Involved in the Clearance of Non-Protein Nanoparticles that Mimic LDL Composition: A Study with Knockout and Transgenic Mice

Lipids, 2017-12, Vol.52 (12), p.991-998

2017

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Significance of the hydrophobic residues 225-230 of apoA-I for the biogenesis of HDL

Journal of lipid research, 2013-12, Vol.54 (12), p.3293-3302

2013

Volltextzugriff (PDF)

Role of the hydrophobic and charged residues in the 218-226 region of apoA-I in the biogenesis of HDL

Journal of lipid research, 2013-12, Vol.54 (12), p.3281-3292

2013

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Insights into intrauterine growth restriction based on maternal and umbilical cord blood metabolomics

Scientific reports, 2021-04, Vol.11 (1), p.7824-7824, Article 7824

2021

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Biomimetic Artificial Surfaces Quantitatively Reproduce the Water Repellency of a Lotus Leaf

Advanced materials (Weinheim), 2008-11, Vol.20 (21), p.4049-4054

2008

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Metric-Distortion Bounds under Limited Information

The Journal of artificial intelligence research, 2022-01, Vol.74, p.1449-1483

2022

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Cholesterol Efflux Pathways Suppress Inflammasome Activation, NETosis, and Atherogenesis

Anti-Inflammatory Effects of HDL (High-Density Lipoprotein) in Macrophages Predominate Over Proinflammatory Effects in Atherosclerotic Plaques

Cholesterol Accumulation in Dendritic Cells Links the Inflammasome to Acquired Immunity

Deficiency of ATP-Binding Cassette Transporters A1 and G1 in Endothelial Cells Accelerates Atherosclerosis in Mice

HDL-apoA-I induces the expression of angiopoietin like 4 (ANGPTL4) in endothelial cells via a PI3K/AKT/FOXO1 signaling pathway

LXR Suppresses Inflammatory Gene Expression and Neutrophil Migration through cis-Repression and Cholesterol Efflux

Reconstituted HDL-apoE3 promotes endothelial cell migration through ID1 and its downstream kinases ERK1/2, AKT and p38 MAPK

Role of apolipoproteins, ABCA1 and LCAT in the biogenesis of normal and aberrant high density lipoproteins

ApoA-IV promotes the biogenesis of apoA-IV-containing HDL particles with the participation of ABCA1 and LCAT[S]

apoE3[K146N/R147W] acts as a dominant negative apoE form that prevents remnant clearance and inhibits the biogenesis of HDL[S]

Significance of the hydrophobic residues 225–230 of apoA-I for the biogenesis of HDL1[S]

Role of the hydrophobic and charged residues in the 218–226 region of apoA-I in the biogenesis of HDL1[S]

Cholesterol accumulation in macrophages drives NETosis in atherosclerotic plaques via IL-1β secretion

HDL biogenesis, remodeling, and catabolism

Response by Fotakis et al to Letter Regarding Article, "Anti-Inflammatory Effects of HDL (High-Density Lipoprotein) in Macrophages Predominate Over Proinflammatory Effects in Atherosclerotic Plaques"

TTC39B destabilizes retinoblastoma protein promoting hepatic lipogenesis in a sex-specific fashion

The Effect of Natural LCAT Mutations on the Biogenesis of HDL

Natural human apoA-I mutations L141RPisa and L159RFIN alter HDL structure and functionality and promote atherosclerosis development in mice

Natural human apoA-I mutations L141R Pisa and L159R FIN alter HDL structure and functionality and promote atherosclerosis development in mice

Tissue Uptake Mechanisms Involved in the Clearance of Non-Protein Nanoparticles that Mimic LDL Composition: A Study with Knockout and Transgenic Mice

Significance of the hydrophobic residues 225-230 of apoA-I for the biogenesis of HDL

Role of the hydrophobic and charged residues in the 218-226 region of apoA-I in the biogenesis of HDL

Insights into intrauterine growth restriction based on maternal and umbilical cord blood metabolomics

Biomimetic Artificial Surfaces Quantitatively Reproduce the Water Repellency of a Lotus Leaf

Metric-Distortion Bounds under Limited Information

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