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Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment

Oncogene, 2017-08, Vol.36 (33), p.4818-4818

2017

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Overcoming chemoresistance of non-small cell lung carcinoma through restoration of an AIF-dependent apoptotic pathway

Oncogene, 2008-03, Vol.27 (14), p.1981-1992

2008

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Caspase inhibition prevents cardiac dysfunction and heart apoptosis in a rat model of sepsis

American journal of respiratory and critical care medicine, 2001, Vol.163 (1), p.218-225

2001

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Epidermal differentiation does not involve the pro-apoptotic executioner caspases, but is associated with caspase-14 induction and processing

Cell death and differentiation, 2000-12, Vol.7 (12), p.1218-1224

2000

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Colon cancer cells escape 5FU chemotherapy-induced cell death by entering stemness and quiescence associated with the c-Yes/YAP axis

Clinical cancer research, 2014-02, Vol.20 (4), p.837-846

2014

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Progression of actinic keratosis to squamous cell carcinoma of the skin correlates with deletion of the 9p21 region encoding the p16(INK4a) tumor suppressor

Cancer letters, 2002-02, Vol.176 (2), p.205-214

2002

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Hepatocyte Nuclear Factor 4α Isoforms Originated from the P1 Promoter Are Expressed in Human Pancreatic β-Cells and Exhibit Stronger Transcriptional Potentials than P2 Promoter-Driven Isoforms

Endocrinology (Philadelphia), 2003-05, Vol.144 (5), p.1686-1694

2003

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Hepatocyte Nuclear Factor 4 Alpha Ligand Binding and F Domains Mediate Interaction and Transcriptional Synergy with the Pancreatic Islet LIM HD Transcription Factor Isl1

Journal of molecular biology, 2006-12, Vol.364 (4), p.567-581

2006

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Terminal differentiation of human epidermal keratinocytes involves mitochondria- and caspase-dependent cell death pathway

Cell death and differentiation, 2003-07, Vol.10 (7), p.850-852

2003

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Erratum: Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment

Oncogene, 2017-08, Vol.36 (33), p.4818-4818

2017

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Study of mitochondrial membrane potential, reactive oxygen species, DNA fragmentation and cell viability by flow cytometry in human sperm

Human reproduction (Oxford), 2002-05, Vol.17 (5), p.1257-1265

2002

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Essential role of mitochondria in apoptosis of cancer cells induced by the marine alkaloid Lamellarin D

Molecular cancer therapeutics, 2009-12, Vol.8 (12), p.3307-3317

2009

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Ventricular myocyte caspases are directly responsible for endotoxin-induced cardiac dysfunction

Circulation (New York, N.Y.), 2005-05, Vol.111 (20), p.2596-2604

2005

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Comparison of four fluorochromes for the detection of the inner mitochondrial membrane potential in human spermatozoa and their correlation with sperm motility

Human reproduction (Oxford), 2004-10, Vol.19 (10), p.2267-2276

2004

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Improvement of the gene splicing overlap (SOE) method

BioTechniques, 1995-08, Vol.19 (2), p.186

1995

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Recent Advances in Steroid Hormone Action

Moudgil, V. K

1, 2019

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Apoptosis and senescence are triggered by Lamellarin-D in cancer cells

European journal of cancer supplements, 2008, Vol.6 (9), p.98-99

2008

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Mécanismes proapoptotiques des agents anticancéreux : la mitochondrie, une cible pharmacologique potentielle ?

Annales de biologie clinique (Paris), 2002, Vol.60 (4), p.391-403

2002

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Retinoic Acid Receptors Inhibit AP1 Activation by Regulating Extracellular Signal-Regulated Kinase and CBP Recruitment to an AP1-Responsive Promoter

Molecular and Cellular Biology, 2002-07, Vol.22 (13), p.4522-4534

2002

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Functional properties of the R154X HNF-4α protein generated by a mutation associated with maturity-onset diabetes of the young, type 1

FEBS letters, 2000-08, Vol.479 (1), p.41-45

2000

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Apoptosis-inducing factor determines the chemoresistance of non-small-cell lung carcinomas

Oncogene, 2004-08, Vol.23 (37), p.6282-6291

2004

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Epidermal growth factor receptor distinguishes between stem and transient amplifying cell fate in squamous cell carcinoma cell line

European journal of cancer supplements, 2008, Vol.6 (9), p.22-22

2008

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Translational research and Cancer Plan

Bulletin du cancer, 2007-12, Vol.94 (12), p.1107

2007

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Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment

Oncogene, 2002-01, Vol.21 (1), p.65-77

2002

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Nouvelle approche globale en protéomique: les biopuces à protéines: Techniques actuelles et applications

Annales de biologie clinique (Paris), 2004-03, Vol.62 (2), p.139-148

2004

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Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment

Overcoming chemoresistance of non-small cell lung carcinoma through restoration of an AIF-dependent apoptotic pathway

Caspase inhibition prevents cardiac dysfunction and heart apoptosis in a rat model of sepsis

Epidermal differentiation does not involve the pro-apoptotic executioner caspases, but is associated with caspase-14 induction and processing

Colon cancer cells escape 5FU chemotherapy-induced cell death by entering stemness and quiescence associated with the c-Yes/YAP axis

Progression of actinic keratosis to squamous cell carcinoma of the skin correlates with deletion of the 9p21 region encoding the p16(INK4a) tumor suppressor

Hepatocyte Nuclear Factor 4α Isoforms Originated from the P1 Promoter Are Expressed in Human Pancreatic β-Cells and Exhibit Stronger Transcriptional Potentials than P2 Promoter-Driven Isoforms

Hepatocyte Nuclear Factor 4 Alpha Ligand Binding and F Domains Mediate Interaction and Transcriptional Synergy with the Pancreatic Islet LIM HD Transcription Factor Isl1

Terminal differentiation of human epidermal keratinocytes involves mitochondria- and caspase-dependent cell death pathway

Erratum: Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment

Study of mitochondrial membrane potential, reactive oxygen species, DNA fragmentation and cell viability by flow cytometry in human sperm

Essential role of mitochondria in apoptosis of cancer cells induced by the marine alkaloid Lamellarin D

Ventricular myocyte caspases are directly responsible for endotoxin-induced cardiac dysfunction

Comparison of four fluorochromes for the detection of the inner mitochondrial membrane potential in human spermatozoa and their correlation with sperm motility

Improvement of the gene splicing overlap (SOE) method

Recent Advances in Steroid Hormone Action

Apoptosis and senescence are triggered by Lamellarin-D in cancer cells

Mécanismes proapoptotiques des agents anticancéreux : la mitochondrie, une cible pharmacologique potentielle ?

Retinoic Acid Receptors Inhibit AP1 Activation by Regulating Extracellular Signal-Regulated Kinase and CBP Recruitment to an AP1-Responsive Promoter

Functional properties of the R154X HNF-4α protein generated by a mutation associated with maturity-onset diabetes of the young, type 1

Apoptosis-inducing factor determines the chemoresistance of non-small-cell lung carcinomas

Epidermal growth factor receptor distinguishes between stem and transient amplifying cell fate in squamous cell carcinoma cell line

Translational research and Cancer Plan

Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment

Nouvelle approche globale en protéomique: les biopuces à protéines: Techniques actuelles et applications

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