UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

ERK Pathway in Activated, Myofibroblast-Like, Hepatic Stellate Cells: A Critical Signaling Crossroad Sustaining Liver Fibrosis

International journal of molecular sciences, 2019-06, Vol.20 (11), p.2700

2019

Volltextzugriff (PDF)

Hepatocellular Carcinoma: Latest Research in Pathogenesis, Detection and Treatment

International journal of molecular sciences, 2023-07, Vol.24 (15), p.12224

2023

Volltextzugriff (PDF)

Metabolic Reprogramming of HCC: A New Microenvironment for Immune Responses

International journal of molecular sciences, 2023-04, Vol.24 (8), p.7463

2023

Volltextzugriff (PDF)

Hypoxia, Hypoxia-Inducible Factors and Liver Fibrosis

Cells (Basel, Switzerland), 2021-07, Vol.10 (7), p.1764

2021

Volltextzugriff (PDF)

Microvesicles released from fat-laden cells promote activation of hepatocellular NLRP3 inflammasome: A pro-inflammatory link between lipotoxicity and non-alcoholic steatohepatitis

PloS one, 2017-03, Vol.12 (3), p.e0172575-e0172575

2017

Volltextzugriff (PDF)

Histidine-rich glycoprotein in metabolic dysfunction-associated steatohepatitis-related disease progression and liver carcinogenesis

Frontiers in immunology, 2024-02, Vol.15, p.1342404-1342404

2024

Volltextzugriff (PDF)

Hepatic Myofibroblasts: A Heterogeneous and Redox-Modulated Cell Population in Liver Fibrogenesis

Antioxidants, 2022-06, Vol.11 (7), p.1278

2022

Volltextzugriff (PDF)

SerpinB3 as a Pro-Inflammatory Mediator in the Progression of Experimental Non-Alcoholic Fatty Liver Disease

Frontiers in immunology, 2022-07, Vol.13, p.910526-910526

2022

Volltextzugriff (PDF)

Of FACT complex and oxidative stress response: a KEAP1/NRF2-dependent novel mechanism sustaining hepatocellular carcinoma progression

Gut, 2020-02, Vol.69 (2), p.195-196

2020

Volltextzugriff (PDF)

Liver fibrogenesis: un update on established and emerging basic concepts

Archives of biochemistry and biophysics, 2020-08, Vol.689, p.108445-108445, Article 108445

2020

Volltextzugriff (PDF)

FRI-479-YI The role of OSM/OSMRβ axis in shaping the immunemicroenvironment favouring MASLD-related HCC immune evasion

Journal of hepatology, 2024-06, Vol.80, p.S421-S421

2024

Volltextzugriff (PDF)

Hypoxia‐inducible factor 2α drives nonalcoholic fatty liver progression by triggering hepatocyte release of histidine‐rich glycoprotein

Hepatology (Baltimore, Md.), 2018-06, Vol.67 (6), p.2196-2214

2018

Volltextzugriff (PDF)

Oncostatin M promotes a pro-tumorigenic inflammatory response in NASH-related HCC

Journal of hepatology, 2023-06, Vol.78, p.S526-S527

2023

Volltextzugriff (PDF)

SAT-211 - Oncostatin M promotes a pro-tumorigenic inflammatory response in NASH-related HCC

Journal of hepatology, 2023-06, Vol.78, p.S526-S527

2023

Volltextzugriff (PDF)

Oncostatin M is overexpressed in NASH‐related hepatocellular carcinoma and promotes cancer cell invasiveness and angiogenesis

The Journal of pathology, 2022-05, Vol.257 (1), p.82-95

2022

Volltextzugriff (PDF)

Oncostatin M, A Profibrogenic Mediator Overexpressed in Non-Alcoholic Fatty Liver Disease, Stimulates Migration of Hepatic Myofibroblasts

Cells (Basel, Switzerland), 2019-12, Vol.9 (1), p.28

2019

Volltextzugriff (PDF)

Hepatocyte-Specific Deletion of HIF2α Prevents NASH-Related Liver Carcinogenesis by Decreasing Cancer Cell Proliferation

Cellular and molecular gastroenterology and hepatology, 2022-01, Vol.13 (2), p.459-482

2022

Volltextzugriff (PDF)

THU-464-NASH-related liver carcinogenesis is critically affected by hypoxia-inducible factor 2 alpha

Journal of hepatology, 2019-04, Vol.70 (1), p.e363-e364

2019

Volltextzugriff (PDF)

Nrf2 Mutation/Activation Is Dispensable for the Development of Chemically Induced Mouse HCC

Cellular and molecular gastroenterology and hepatology, 2022-01, Vol.13 (1), p.113-127

2022

Volltextzugriff (PDF)

Circulating Zonulin is Related to Hepatic Necroinflammation in Patients with Non Alcoholic Fatty Liver Disease

Clinical laboratory (Heidelberg), 2020-01, Vol.66 (4)

2020

Volltextzugriff (PDF)

Hepatocyte-Specific Deletion of HIF2α Prevents NASH-Related Liver Carcinogenesis by Decreasing Cancer Cell ProliferationSummary

Cellular and molecular gastroenterology and hepatology, 2022-01, Vol.13 (2), p.459-482

2022

Volltextzugriff (PDF)

Nrf2 Mutation/Activation Is Dispensable for the Development of Chemically Induced Mouse HCCSummary

Cellular and molecular gastroenterology and hepatology, 2022-01, Vol.13 (1), p.113-127

2022

Volltextzugriff (PDF)

COVID-19 and hospital management costs: the Italian experience

BMC health services research, 2022-08, Vol.22 (1), p.991-10, Article 991

2022

Volltextzugriff (PDF)

Comparison of Two Dosing Regimens of Miltefosine, Both in Combination With Allopurinol, on Clinical and Parasitological Findings of Dogs With Leishmaniosis: A Pilot Study

Frontiers in veterinary science, 2020-12, Vol.7, p.577395-577395

2020

Volltextzugriff (PDF)

1 – 25 von 29

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(29)
Mit Volltextzugriff
(26)

Publikationsform

Artikel
(29)

Sprache

Englisch
(29)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

ERK Pathway in Activated, Myofibroblast-Like, Hepatic Stellate Cells: A Critical Signaling Crossroad Sustaining Liver Fibrosis

Hepatocellular Carcinoma: Latest Research in Pathogenesis, Detection and Treatment

Metabolic Reprogramming of HCC: A New Microenvironment for Immune Responses

Hypoxia, Hypoxia-Inducible Factors and Liver Fibrosis

Microvesicles released from fat-laden cells promote activation of hepatocellular NLRP3 inflammasome: A pro-inflammatory link between lipotoxicity and non-alcoholic steatohepatitis

Histidine-rich glycoprotein in metabolic dysfunction-associated steatohepatitis-related disease progression and liver carcinogenesis

Hepatic Myofibroblasts: A Heterogeneous and Redox-Modulated Cell Population in Liver Fibrogenesis

SerpinB3 as a Pro-Inflammatory Mediator in the Progression of Experimental Non-Alcoholic Fatty Liver Disease

Of FACT complex and oxidative stress response: a KEAP1/NRF2-dependent novel mechanism sustaining hepatocellular carcinoma progression

Liver fibrogenesis: un update on established and emerging basic concepts

FRI-479-YI The role of OSM/OSMRβ axis in shaping the immunemicroenvironment favouring MASLD-related HCC immune evasion

Hypoxia‐inducible factor 2α drives nonalcoholic fatty liver progression by triggering hepatocyte release of histidine‐rich glycoprotein

Oncostatin M promotes a pro-tumorigenic inflammatory response in NASH-related HCC

SAT-211 - Oncostatin M promotes a pro-tumorigenic inflammatory response in NASH-related HCC

Oncostatin M is overexpressed in NASH‐related hepatocellular carcinoma and promotes cancer cell invasiveness and angiogenesis

Oncostatin M, A Profibrogenic Mediator Overexpressed in Non-Alcoholic Fatty Liver Disease, Stimulates Migration of Hepatic Myofibroblasts

Hepatocyte-Specific Deletion of HIF2α Prevents NASH-Related Liver Carcinogenesis by Decreasing Cancer Cell Proliferation

THU-464-NASH-related liver carcinogenesis is critically affected by hypoxia-inducible factor 2 alpha

SerpinB3 Differently Up-Regulates Hypoxia Inducible Factors -1α and -2α in Hepatocellular Carcinoma: Mechanisms Revealing Novel Potential Therapeutic Targets

Nrf2 Mutation/Activation Is Dispensable for the Development of Chemically Induced Mouse HCC

Circulating Zonulin is Related to Hepatic Necroinflammation in Patients with Non Alcoholic Fatty Liver Disease

Hepatocyte-Specific Deletion of HIF2α Prevents NASH-Related Liver Carcinogenesis by Decreasing Cancer Cell ProliferationSummary

Nrf2 Mutation/Activation Is Dispensable for the Development of Chemically Induced Mouse HCCSummary

COVID-19 and hospital management costs: the Italian experience

Comparison of Two Dosing Regimens of Miltefosine, Both in Combination With Allopurinol, on Clinical and Parasitological Findings of Dogs With Leishmaniosis: A Pilot Study

Suchergebnisse filtern