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Skeletal muscle CaV1.1 channelopathies

Pflügers Archiv, 2020-07, Vol.472 (7), p.739-754

Flucher, Bernhard E.

2020

Volltextzugriff (PDF)

The Role of Auxiliary Subunits for the Functional Diversity of Voltage-Gated Calcium Channels

Journal of cellular physiology, 2015-09, Vol.230 (9), p.2019-2031

2015

Volltextzugriff (PDF)

Molecular Interactions in the Voltage Sensor Controlling Gating Properties of CaV Calcium Channels

Structure (London), 2016-02, Vol.24 (2), p.261-271

2016

Volltextzugriff (PDF)

Postsynaptic CaV1.1-driven calcium signaling coordinates presynaptic differentiation at the developing neuromuscular junction

Scientific reports, 2019-12, Vol.9 (1), p.1-15, Article 18450

2019

Volltextzugriff (PDF)

Retrograde Coupling: Muscle’s Orphan Signaling Pathway?

Biophysical journal, 2016-02, Vol.110 (4), p.870-871

Flucher, Bernhard E.

2016

Volltextzugriff (PDF)

CaV1.1 voltage-sensing domain III exclusively controls skeletal muscle excitation-contraction coupling

Nature communications, 2024-08, Vol.15 (1), p.7440-13, Article 7440

2024

Volltextzugriff (PDF)

A homozygous missense variant in CACNB4 encoding the auxiliary calcium channel beta4 subunit causes a severe neurodevelopmental disorder and impairs channel and non-channel functions

PLoS genetics, 2020-03, Vol.16 (3), p.e1008625-e1008625

2020

Volltextzugriff (PDF)

Molecular Interactions in the Voltage Sensor Controlling Gating Properties of Ca V Calcium Channels

Structure (London), 2016-02, Vol.24 (2), p.261-271

2016

Volltextzugriff (PDF)

How is SR calcium release in muscle modulated by PIP(4,5)2?

The Journal of general physiology, 2015-05, Vol.145 (5), p.361-364

Flucher, Bernhard E

2015

Volltextzugriff (PDF)

Restricting calcium currents is required for correct fiber type specification in skeletal muscle

Development (Cambridge), 2016-05, Vol.143 (9), p.1547-1559

2016

Volltextzugriff (PDF)

Structural determinants of voltage-gating properties in calcium channels

eLife, 2021-03, Vol.10

2021

Volltextzugriff (PDF)

Physiological and Pharmacological Modulation of the Embryonic Skeletal Muscle Calcium Channel Splice Variant CaV1.1e

Biophysical journal, 2015-03, Vol.108 (5), p.1072-1080

2015

Volltextzugriff (PDF)

Counteractive and cooperative actions of muscle β-catenin and CaV1.1 during early neuromuscular synapse formation

iScience, 2022-04, Vol.25 (4), p.104025-104025, Article 104025

2022

Volltextzugriff (PDF)

Biophysical classification of a CACNA1D de novo mutation as a high-risk mutation for a severe neurodevelopmental disorder

Molecular autism, 2020-01, Vol.11 (1), p.4-18, Article 4

2020

Volltextzugriff (PDF)

Calcium Influx and Release Cooperatively Regulate AChR Patterning and Motor Axon Outgrowth during Neuromuscular Junction Formation

Cell reports (Cambridge), 2018-06, Vol.23 (13), p.3891-3904

2018

Volltextzugriff (PDF)

STAC proteins: The missing link in skeletal muscle EC coupling and new regulators of calcium channel function

Biochimica et biophysica acta. Molecular cell research, 2019-07, Vol.1866 (7), p.1101-1110

2019

Volltextzugriff (PDF)

Spatial transcriptomics in embryonic mouse diaphragm muscle reveals regional gradients and subdomains of developmental gene expression

iScience, 2024-06, Vol.27 (6), p.110018-110018, Article 110018

2024

Volltextzugriff (PDF)

A Polybasic Plasma Membrane Binding Motif in the I-II Linker Stabilizes Voltage-gated CaV1.2 Calcium Channel Function

The Journal of biological chemistry, 2015-08, Vol.290 (34), p.21086-21100

2015

Volltextzugriff (PDF)

How and why are calcium currents curtailed in the skeletal muscle voltage‐gated calcium channels?

The Journal of physiology, 2017-03, Vol.595 (5), p.1451-1463

2017

Volltextzugriff (PDF)

STAC3 stably interacts through its C1 domain with CaV1.1 in skeletal muscle triads

Scientific reports, 2017-01, Vol.7 (1), p.41003-41003, Article 41003

2017

Volltextzugriff (PDF)

STAC3 stably interacts through its C1 domain with Ca V 1.1 in skeletal muscle triads

Scientific reports, 2017-01, Vol.7, p.41003

2017

Volltextzugriff (PDF)

STAC3 determines the slow activation kinetics of Ca V 1.1 currents and inhibits its voltage-dependent inactivation

Journal of cellular physiology, 2022-11, Vol.237 (11), p.4197-4214

2022

Volltextzugriff (PDF)

A new L-type calcium channel isoform required for normal patterning of the developing neuromuscular junction

Channels (Austin, Tex.), 2011-11, Vol.5 (6), p.518-524

2011

Volltextzugriff (PDF)

Specific contributions of the four voltage-sensing domains in L-type calcium channels to gating and modulation

The Journal of general physiology, 2016-08, Vol.148 (2), p.91-95

Flucher, Bernhard E

2016

Volltextzugriff (PDF)

Correction: Specific contributions of the four voltage-sensing domains in L-type calcium channels to gating and modulation

The Journal of general physiology, 2016-10, Vol.148 (4), p.357-357

Flucher, Bernhard E

2016

Volltextzugriff (PDF)

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Skeletal muscle CaV1.1 channelopathies

The Role of Auxiliary Subunits for the Functional Diversity of Voltage-Gated Calcium Channels

Molecular Interactions in the Voltage Sensor Controlling Gating Properties of CaV Calcium Channels

Postsynaptic CaV1.1-driven calcium signaling coordinates presynaptic differentiation at the developing neuromuscular junction

Retrograde Coupling: Muscle’s Orphan Signaling Pathway?

CaV1.1 voltage-sensing domain III exclusively controls skeletal muscle excitation-contraction coupling

A homozygous missense variant in CACNB4 encoding the auxiliary calcium channel beta4 subunit causes a severe neurodevelopmental disorder and impairs channel and non-channel functions

Molecular Interactions in the Voltage Sensor Controlling Gating Properties of Ca V Calcium Channels

How is SR calcium release in muscle modulated by PIP(4,5)2?

Restricting calcium currents is required for correct fiber type specification in skeletal muscle

Structural determinants of voltage-gating properties in calcium channels

Physiological and Pharmacological Modulation of the Embryonic Skeletal Muscle Calcium Channel Splice Variant CaV1.1e

Counteractive and cooperative actions of muscle β-catenin and CaV1.1 during early neuromuscular synapse formation

Biophysical classification of a CACNA1D de novo mutation as a high-risk mutation for a severe neurodevelopmental disorder

Calcium Influx and Release Cooperatively Regulate AChR Patterning and Motor Axon Outgrowth during Neuromuscular Junction Formation

STAC proteins: The missing link in skeletal muscle EC coupling and new regulators of calcium channel function

Spatial transcriptomics in embryonic mouse diaphragm muscle reveals regional gradients and subdomains of developmental gene expression

A Polybasic Plasma Membrane Binding Motif in the I-II Linker Stabilizes Voltage-gated CaV1.2 Calcium Channel Function

How and why are calcium currents curtailed in the skeletal muscle voltage‐gated calcium channels?

STAC3 stably interacts through its C1 domain with CaV1.1 in skeletal muscle triads

STAC3 stably interacts through its C1 domain with Ca V 1.1 in skeletal muscle triads

STAC3 determines the slow activation kinetics of Ca V 1.1 currents and inhibits its voltage-dependent inactivation

A new L-type calcium channel isoform required for normal patterning of the developing neuromuscular junction

Specific contributions of the four voltage-sensing domains in L-type calcium channels to gating and modulation

Correction: Specific contributions of the four voltage-sensing domains in L-type calcium channels to gating and modulation

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