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358 - Post-Transplantation Cyclophosphamide Uniquely Restrains Alloreactive CD4+ T-Cell Activation, Proliferation, and Differentiation after Murine MHC-Haploidentical Hematopoietic Cell Transplantation

Transplantation and cellular therapy, 2022-03, Vol.28 (3), p.S277-S278

2022

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Abstract 1256: Non-steroidal anti-inflammatory drugs induce ER stress and have an immunomodulatory role in the suppression of colorectal tumorigenesis

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.1256-1256

2017

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BET Inhibitors Potentiate Chemotherapy and Killing of SPOP -Mutant Colon Cancer Cells via Induction of DR5

Cancer research (Chicago, Ill.), 2019-03, Vol.79 (6), p.1191-1203

2019

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Colorectal cancer prevention: Immune modulation taking the stage

Biochimica et biophysica acta. Reviews on cancer, 2018-04, Vol.1869 (2), p.138-148

2018

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Direct Integration of Anti-CD19 Chimeric-Antigen-Receptor (CAR) T Cells Early after Post-Transplantation Cyclophosphamide (PTCy) in Murine MHC-Haploidentical Hematopoietic Cell Transplantation (HCT) Exerts Anti-Tumor Effects without Causing Graft-Versus-Host Disease (GVHD)

Biology of blood and marrow transplantation, 2020-03, Vol.26 (3), p.S170-S170

2020

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Efflux capacity and aldehyde dehydrogenase both contribute to CD8+ T-cell resistance to posttransplant cyclophosphamide

Blood advances, 2022-09, Vol.6 (17), p.4994-5008

2022

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Modulation of the Gut Microbiota By Post-Transplantation Cyclophosphamide after Murine MHC-Haploidentical Hematopoietic Cell Transplantation

Transplantation and cellular therapy, 2021-03, Vol.27 (3), p.S254

2021

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Murine allogeneic CAR T cells integrated before or early after posttransplant cyclophosphamide exert antitumor effects

Blood, 2023-02, Vol.141 (6), p.659-672

2023

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Non-steroidal anti-inflammatory drugs induce immunogenic cell death in suppressing colorectal tumorigenesis

Oncogene, 2021-03, Vol.40 (11), p.2035-2050

2021

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Phenotypic Myeloid-Derived Suppressor Cells (MDSCs) are expanded after Post-Transplantation Cyclophosphamide (PTCy), but are not necessary for Graft-Versus-Host Disease Prevention by PTCy

Biology of blood and marrow transplantation, 2020-03, Vol.26 (3), p.S169-S170

2020

Volltextzugriff (PDF)

Post-Transplantation Cyclophosphamide Uniquely Restrains Alloreactive CD4+ T-Cell Activation, Proliferation, and Differentiation after Murine MHC-Haploidentical Hematopoietic Cell Transplantation

Transplantation and cellular therapy, 2022-03, Vol.28 (3), p.S277-S278

2022

Volltextzugriff (PDF)

Post-Transplantation Cyclophosphamide Uniquely Restrains Alloreactive CD4 + T-Cell Proliferation and Differentiation After Murine MHC-Haploidentical Hematopoietic Cell Transplantation

Frontiers in immunology, 2022-02, Vol.13, p.796349

2022

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Posttransplantation cyclophosphamide expands functional myeloid-derived suppressor cells and indirectly influences Tregs

Blood advances, 2023-04, Vol.7 (7), p.1117-1129

2023

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Prospective Study of a Novel, Radiation-Free, Reduced-Intensity Bone Marrow Transplantation Platform for Primary Immunodeficiency Diseases

Biology of blood and marrow transplantation, 2020-01, Vol.26 (1), p.94-106

2020

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The Role of NSAID-Induced ER Stress and Immunomodulation in the Suppression of Colorectal Tumorigenesis

Fletcher, Rochelle E

2018

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Synthesis and pharmacological evaluation of newly detected synthetic cannabinoid receptor agonists AB-4CN-BUTICA, MMB-4CN-BUTINACA, MDMB-4F-BUTICA, MDMB-4F-BUTINACA and their analogs

Frontiers in psychiatry, 2022-09, Vol.13, p.1010501-1010501

2022

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358 - Post-Transplantation Cyclophosphamide Uniquely Restrains Alloreactive CD4+ T-Cell Activation, Proliferation, and Differentiation after Murine MHC-Haploidentical Hematopoietic Cell Transplantation

Abstract 1256: Non-steroidal anti-inflammatory drugs induce ER stress and have an immunomodulatory role in the suppression of colorectal tumorigenesis

BET Inhibitors Potentiate Chemotherapy and Killing of SPOP -Mutant Colon Cancer Cells via Induction of DR5

Colorectal cancer prevention: Immune modulation taking the stage

Direct Integration of Anti-CD19 Chimeric-Antigen-Receptor (CAR) T Cells Early after Post-Transplantation Cyclophosphamide (PTCy) in Murine MHC-Haploidentical Hematopoietic Cell Transplantation (HCT) Exerts Anti-Tumor Effects without Causing Graft-Versus-Host Disease (GVHD)

Efflux capacity and aldehyde dehydrogenase both contribute to CD8+ T-cell resistance to posttransplant cyclophosphamide

Modulation of the Gut Microbiota By Post-Transplantation Cyclophosphamide after Murine MHC-Haploidentical Hematopoietic Cell Transplantation

Murine allogeneic CAR T cells integrated before or early after posttransplant cyclophosphamide exert antitumor effects

Non-steroidal anti-inflammatory drugs induce immunogenic cell death in suppressing colorectal tumorigenesis

Phenotypic Myeloid-Derived Suppressor Cells (MDSCs) are expanded after Post-Transplantation Cyclophosphamide (PTCy), but are not necessary for Graft-Versus-Host Disease Prevention by PTCy

Post-Transplantation Cyclophosphamide Uniquely Restrains Alloreactive CD4+ T-Cell Activation, Proliferation, and Differentiation after Murine MHC-Haploidentical Hematopoietic Cell Transplantation

Post-Transplantation Cyclophosphamide Uniquely Restrains Alloreactive CD4 + T-Cell Proliferation and Differentiation After Murine MHC-Haploidentical Hematopoietic Cell Transplantation

Posttransplantation cyclophosphamide expands functional myeloid-derived suppressor cells and indirectly influences Tregs

Prospective Study of a Novel, Radiation-Free, Reduced-Intensity Bone Marrow Transplantation Platform for Primary Immunodeficiency Diseases

The Role of NSAID-Induced ER Stress and Immunomodulation in the Suppression of Colorectal Tumorigenesis

Synthesis and pharmacological evaluation of newly detected synthetic cannabinoid receptor agonists AB-4CN-BUTICA, MMB-4CN-BUTINACA, MDMB-4F-BUTICA, MDMB-4F-BUTINACA and their analogs

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