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Nitroglycerin Use in Myocardial Infarction Patients: Risks and Benefits

Circulation Journal, 2012, Vol.76(1), pp.15-21

2012

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pH-Gated Succinate Secretion Regulates Muscle Remodeling in Response to Exercise

Cell, 2020-10, Vol.183 (1), p.62-75.e17

2020

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Mitochondria as a source of reactive oxygen and nitrogen species: from molecular mechanisms to human health

Antioxidants & redox signaling, 2013-06, Vol.18 (16), p.2029-2074

2013

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A Personalized Medicine Approach for Asian Americans with the Aldehyde Dehydrogenase 22 Variant

Annual review of pharmacology and toxicology, 2015-01, Vol.55 (1), p.107-127

2015

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Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction

Journal of the American Heart Association, 2013-10, Vol.2 (5), p.e000461-e000461

2013

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Impact of exercise training on redox signaling in cardiovascular diseases

Food and chemical toxicology, 2013-12, Vol.62, p.107-119

2013

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Novel and prevalent non-East Asian ALDH2 variants; Implications for global susceptibility to aldehydes’ toxicity

EBioMedicine, 2020-05, Vol.55, p.102753, Article 102753

2020

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Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: Benefits of Alda-1

International journal of cardiology, 2015-01, Vol.179, p.129-138

2015

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Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3

International journal of cardiology, 2018-12, Vol.272, p.194-201

2018

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Comment on: “Aldehyde dehydrogenases contribute to skeletal muscle homeostasis in healthy, aging, and Duchenne muscular dystrophy patients” by Etienne et al

Journal of cachexia, sarcopenia and muscle, 2020-12, Vol.11 (6), p.1858-1859

2020

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Pharmacological inhibition of βIIPKC is cardioprotective in late-stage hypertrophy

Journal of molecular and cellular cardiology, 2011-12, Vol.51 (6), p.980-987

2011

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Intracellular mechanisms of specific β-adrenoceptor antagonists involved in improved cardiac function and survival in a genetic model of heart failure

Journal of molecular and cellular cardiology, 2008-08, Vol.45 (2), p.240-249

2008

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Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives

Trends in pharmacological sciences (Regular ed.), 2021-10, Vol.42 (10), p.829-844

2021

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Mitochondrially-targeted treatment strategies

Molecular aspects of medicine, 2020-02, Vol.71, p.100836-100836, Article 100836

2020

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Glyceraldehyde-3-phosphate Dehydrogenase (GAPDH) Phosphorylation by Protein Kinase Cδ (PKCδ) Inhibits Mitochondria Elimination by Lysosomal-like Structures following Ischemia and Reoxygenation-induced Injury

The Journal of biological chemistry, 2013-06, Vol.288 (26), p.18947-18960

2013

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A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats

Nature communications, 2019-01, Vol.10 (1), p.329-329, Article 329

2019

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Ultra-sensitive Quantification of Carnosine-4-Hydroxy-2-hexenal Adduct in Mice Heart Using On-line Liquid Chromatography-Electrospray Tandem Mass Spectrometry Assay

Free radical biology & medicine, 2023-03, Vol.198, p.S13-S14

2023

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Mitofusin 1 and 2 deletion results in extensive mitochondria-ER/SR remodeling and compromises skeletal muscle function

Biochimica et biophysica acta. Bioenergetics, 2024-09, Vol.1865, p.149361, Article 149361

2024

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RETRACTED: Angiotensin receptor blockade improves the net balance of cardiac Ca2+ handling-related proteins in sympathetic hyperactivity-induced heart failure

Life sciences (1973), 2011-03, Vol.88 (13-14), p.578-585

2011

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Mitochondrial Unfolded Protein Response (UPRmt) Activation in Cardiac Diseases

Journal of the American College of Cardiology, 2019-08, Vol.74 (7), p.1011-1012

2019

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Glyceraldehyde-3-Phosphate Dehydrogenase (GAPDH) Protein-Protein Interaction Inhibitor Reveals a Non-catalytic Role for GAPDH Oligomerization in Cell Death

The Journal of biological chemistry, 2016-06, Vol.291 (26), p.13608-13621

2016

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Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure

Autophagy, 2017-08, Vol.13 (8), p.1304-1317

2017

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ALDH2 and Cardiovascular Disease

Advances in experimental medicine and biology, 2019, Vol.1193, p.53-67

2019

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Mild mitochondrial impairment enhances innate immunity and longevity through ATFS-1 and p38 signaling

EMBO reports, 2021-12, Vol.22 (12), p.e52964-n/a

2021

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Aldehyde dehydrogenase 2 activation in heart failure restores mitochondrial function and improves ventricular function and remodelling

Cardiovascular research, 2014-09, Vol.103 (4), p.498-508

2014

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Nitroglycerin Use in Myocardial Infarction Patients: Risks and Benefits

pH-Gated Succinate Secretion Regulates Muscle Remodeling in Response to Exercise

Mitochondria as a source of reactive oxygen and nitrogen species: from molecular mechanisms to human health

A Personalized Medicine Approach for Asian Americans with the Aldehyde Dehydrogenase 22 Variant

Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction

Impact of exercise training on redox signaling in cardiovascular diseases

Novel and prevalent non-East Asian ALDH2 variants; Implications for global susceptibility to aldehydes’ toxicity

Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: Benefits of Alda-1

Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3

Comment on: “Aldehyde dehydrogenases contribute to skeletal muscle homeostasis in healthy, aging, and Duchenne muscular dystrophy patients” by Etienne et al

Pharmacological inhibition of βIIPKC is cardioprotective in late-stage hypertrophy

Intracellular mechanisms of specific β-adrenoceptor antagonists involved in improved cardiac function and survival in a genetic model of heart failure

Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives

Mitochondrially-targeted treatment strategies

Glyceraldehyde-3-phosphate Dehydrogenase (GAPDH) Phosphorylation by Protein Kinase Cδ (PKCδ) Inhibits Mitochondria Elimination by Lysosomal-like Structures following Ischemia and Reoxygenation-induced Injury

A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats

Ultra-sensitive Quantification of Carnosine-4-Hydroxy-2-hexenal Adduct in Mice Heart Using On-line Liquid Chromatography-Electrospray Tandem Mass Spectrometry Assay

Mitofusin 1 and 2 deletion results in extensive mitochondria-ER/SR remodeling and compromises skeletal muscle function

RETRACTED: Angiotensin receptor blockade improves the net balance of cardiac Ca2+ handling-related proteins in sympathetic hyperactivity-induced heart failure

Mitochondrial Unfolded Protein Response (UPRmt) Activation in Cardiac Diseases

Glyceraldehyde-3-Phosphate Dehydrogenase (GAPDH) Protein-Protein Interaction Inhibitor Reveals a Non-catalytic Role for GAPDH Oligomerization in Cell Death

Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure

ALDH2 and Cardiovascular Disease

Mild mitochondrial impairment enhances innate immunity and longevity through ATFS-1 and p38 signaling

Aldehyde dehydrogenase 2 activation in heart failure restores mitochondrial function and improves ventricular function and remodelling

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