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Targeting the TWEAK-Fn14 pathway prevents dysfunction in cardiac calcium handling after acute kidney injury

The Journal of pathology, 2023-12, Vol.261 (4), p.427-441

2023

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Resolution-Based Therapies: The Potential of Lipoxins to Treat Human Diseases

Frontiers in immunology, 2021-04, Vol.12, p.658840-658840

2021

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PTH, vitamin D, and the FGF‐23–klotho axis and heart: Going beyond the confines of nephrology

European journal of clinical investigation, 2018-04, Vol.48 (4), p.n/a

2018

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Beneficial effects of paricalcitol on cardiac dysfunction and remodelling in a model of established heart failure

British journal of pharmacology, 2020-07, Vol.177 (14), p.3273-3290

2020

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BML‐111 treatment prevents cardiac apoptosis and oxidative stress in a mouse model of autoimmune myocarditis

The FASEB journal, 2020-08, Vol.34 (8), p.10531-10546

2020

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Lipoxin-mediated signaling: ALX/FPR2 interaction and beyond

Pharmacological research, 2023-11, Vol.197, p.106982-106982, Article 106982

2023

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Experience with the ACURATE neo and neo2 transcatheter aortic valves in Spain. The PRECISA (PRospective Evaluation Complementing Investigation with ACURATE devices) registry

Catheterization and cardiovascular interventions, 2024-05, Vol.103 (6), p.1015-1022

2024

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The Aryl Hydrocarbon Receptor Ligand FICZ Improves Left Ventricular Remodeling and Cardiac Function at the Onset of Pressure Overload-Induced Heart Failure in Mice

International journal of molecular sciences, 2022-05, Vol.23 (10), p.5403

2022

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Activation of autophagy in macrophages by pro-resolving lipid mediators

Autophagy, 2015-10, Vol.11 (10), p.1729-1744

2015

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Partial Genetic Deletion of Klotho Aggravates Cardiac Calcium Mishandling in Acute Kidney Injury

International journal of molecular sciences, 2023-01, Vol.24 (2), p.1322

2023

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Editorial: Evolving Picture of Calcium Handling in Cardiac Disease

Frontiers in physiology, 2020-09, Vol.11, p.1013-1013

2020

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Role of NOD1 in Heart Failure Progression via Regulation of Ca2+ Handling

Journal of the American College of Cardiology, 2017-01, Vol.69 (4), p.423-433

2017

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Fibroblast Growth Factor-23-Klotho Axis in Cardiorenal Syndrome: Mediators and Potential Therapeutic Targets

Frontiers in physiology, 2021-11, Vol.12, p.775029-775029

2021

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Abnormal Ca2+ spark/STOC coupling in cerebral artery smooth muscle cells of obese type 2 diabetic mice

PloS one, 2013-01, Vol.8 (1), p.e53321-e53321

2013

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The anti-aging factor Klotho protects against acquired long QT syndrome induced by uremia and promoted by fibroblast growth factor 23

BMC medicine, 2022-01, Vol.20 (1), p.14-14, Article 14

2022

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New play for an old actress: A Cinderella in cardiovascular disease

International journal of cardiology, 2023-01, Vol.371, p.76-77

2023

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Enhanced Klotho availability protects against cardiac dysfunction induced by uraemic cardiomyopathy by regulating Ca2+ handling

British journal of pharmacology, 2020-10, Vol.177 (20), p.4701-4719

2020

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The cAMP binding protein Epac modulates Ca2+ sparks by a Ca2+/calmodulin kinase signalling pathway in rat cardiac myocytes

The Journal of physiology, 2007-09, Vol.583 (2), p.685-694

2007

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Cardiac protection induced by urocortin-2 enables the regulation of apoptosis and fibrosis after ischemia and reperfusion involving miR-29a modulation

Molecular therapy. Nucleic acids, 2022-03, Vol.27, p.838-853

2022

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Genetic Deletion of NOD1 Prevents Cardiac Ca2+ Mishandling Induced by Experimental Chronic Kidney Disease

International journal of molecular sciences, 2020-11, Vol.21 (22), p.8868

2020

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Calcitriol, the Bioactive Metabolite of Vitamin D, Increases Ventricular K + Currents in Isolated Mouse Cardiomyocytes

Frontiers in physiology, 2018-08, Vol.9, p.1186-1186

2018

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LA419, a Novel Nitric Oxide Donor, Prevents Pathological Cardiac Remodeling in Pressure-Overloaded Rats Via Endothelial Nitric Oxide Synthase Pathway Regulation

Hypertension (Dallas, Tex. 1979), 2007-12, Vol.50 (6), p.1049-1056

2007

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TRPC Channels: Dysregulation and Ca2+ Mishandling in Ischemic Heart Disease

Cells (Basel, Switzerland), 2020-01, Vol.9 (1), p.173

2020

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NOD1 activation induces cardiac dysfunction and modulates cardiac fibrosis and cardiomyocyte apoptosis

PloS one, 2012-09, Vol.7 (9), p.e45260

2012

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Sustained Epac activation induces calmodulin dependent positive inotropic effect in adult cardiomyocytes

Journal of molecular and cellular cardiology, 2012-11, Vol.53 (5), p.617-625

2012

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Targeting the TWEAK-Fn14 pathway prevents dysfunction in cardiac calcium handling after acute kidney injury

Resolution-Based Therapies: The Potential of Lipoxins to Treat Human Diseases

PTH, vitamin D, and the FGF‐23–klotho axis and heart: Going beyond the confines of nephrology

Beneficial effects of paricalcitol on cardiac dysfunction and remodelling in a model of established heart failure

BML‐111 treatment prevents cardiac apoptosis and oxidative stress in a mouse model of autoimmune myocarditis

Lipoxin-mediated signaling: ALX/FPR2 interaction and beyond

Experience with the ACURATE neo and neo2 transcatheter aortic valves in Spain. The PRECISA (PRospective Evaluation Complementing Investigation with ACURATE devices) registry

The Aryl Hydrocarbon Receptor Ligand FICZ Improves Left Ventricular Remodeling and Cardiac Function at the Onset of Pressure Overload-Induced Heart Failure in Mice

Activation of autophagy in macrophages by pro-resolving lipid mediators

Partial Genetic Deletion of Klotho Aggravates Cardiac Calcium Mishandling in Acute Kidney Injury

Editorial: Evolving Picture of Calcium Handling in Cardiac Disease

Role of NOD1 in Heart Failure Progression via Regulation of Ca2+ Handling

Fibroblast Growth Factor-23-Klotho Axis in Cardiorenal Syndrome: Mediators and Potential Therapeutic Targets

Abnormal Ca2+ spark/STOC coupling in cerebral artery smooth muscle cells of obese type 2 diabetic mice

The anti-aging factor Klotho protects against acquired long QT syndrome induced by uremia and promoted by fibroblast growth factor 23

New play for an old actress: A Cinderella in cardiovascular disease

Enhanced Klotho availability protects against cardiac dysfunction induced by uraemic cardiomyopathy by regulating Ca2+ handling

The cAMP binding protein Epac modulates Ca2+ sparks by a Ca2+/calmodulin kinase signalling pathway in rat cardiac myocytes

Cardiac protection induced by urocortin-2 enables the regulation of apoptosis and fibrosis after ischemia and reperfusion involving miR-29a modulation

Genetic Deletion of NOD1 Prevents Cardiac Ca2+ Mishandling Induced by Experimental Chronic Kidney Disease

Calcitriol, the Bioactive Metabolite of Vitamin D, Increases Ventricular K + Currents in Isolated Mouse Cardiomyocytes

LA419, a Novel Nitric Oxide Donor, Prevents Pathological Cardiac Remodeling in Pressure-Overloaded Rats Via Endothelial Nitric Oxide Synthase Pathway Regulation

TRPC Channels: Dysregulation and Ca2+ Mishandling in Ischemic Heart Disease

NOD1 activation induces cardiac dysfunction and modulates cardiac fibrosis and cardiomyocyte apoptosis

Sustained Epac activation induces calmodulin dependent positive inotropic effect in adult cardiomyocytes

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