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Proinflammatory cytokines upregulate sympathoexcitatory mechanisms in the subfornical organ of the rat

Hypertension (Dallas, Tex. 1979), 2015-05, Vol.65 (5), p.1126-1133

2015

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Leptin Mediates High-Fat Diet Sensitization of Angiotensin II–Elicited Hypertension by Upregulating the Brain Renin–Angiotensin System and Inflammation

Hypertension (Dallas, Tex. 1979), 2016-05, Vol.67 (5), p.970-976

2016

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TNF-α-induced sympathetic excitation requires EGFR and ERK1/2 signaling in cardiovascular regulatory regions of the forebrain

American journal of physiology. Heart and circulatory physiology, 2021-02, Vol.320 (2), p.H772-H786

2021

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Angiotensin II Type 1a Receptors in the Subfornical Organ Modulate Neuroinflammation in the Hypothalamic Paraventricular Nucleus in Heart Failure Rats

Neuroscience, 2018-06, Vol.381, p.46-58

2018

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Endoplasmic reticulum stress increases brain MAPK signaling, inflammation and renin-angiotensin system activity and sympathetic nerve activity in heart failure

American journal of physiology. Heart and circulatory physiology, 2016-10, Vol.311 (4), p.H871-H880

2016

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Blood-borne interleukin-1β acts on the subfornical organ to upregulate the sympathoexcitatory milieu of the hypothalamic paraventricular nucleus

American journal of physiology. Regulatory, integrative and comparative physiology, 2018-03, Vol.314 (3), p.R447-R458

2018

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Central Renin-Angiotensin System Activation and Inflammation Induced by High-Fat Diet Sensitize Angiotensin II-Elicited Hypertension

Hypertension (Dallas, Tex. 1979), 2016-01, Vol.67 (1), p.163-170

2016

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TNF-α receptor 1 knockdown in the subfornical organ ameliorates sympathetic excitation and cardiac hemodynamics in heart failure rats

American journal of physiology. Heart and circulatory physiology, 2017-10, Vol.313 (4), p.H744-H756

2017

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Mineralocorticoid receptors, inflammation and sympathetic drive in a rat model of systolic heart failure

Experimental physiology, 2010-01, Vol.95 (1), p.19-25

Felder, Robert B.

2010

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Sex differences in the central and peripheral manifestations of ischemia-induced heart failure in rats

American journal of physiology. Heart and circulatory physiology, 2019-01, Vol.316 (1), p.H70-H79

2019

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Silencing Epidermal Growth Factor Receptor in Hypothalamic Paraventricular Nucleus Reduces Extracellular Signal-regulated Kinase 1 and 2 Signaling and Sympathetic Excitation in Heart Failure Rats

Neuroscience, 2021-05, Vol.463, p.227-237

2021

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Brain TACE (Tumor Necrosis Factor-α–Converting Enzyme) Contributes to Sympathetic Excitation in Heart Failure Rats

Hypertension (Dallas, Tex. 1979), 2019-07, Vol.74 (1), p.63-72

2019

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Transforming Growth Factor-α Acts in Hypothalamic Paraventricular Nucleus to Upregulate ERK1/2 Signaling and Expression of Sympathoexcitatory Mediators in Heart Failure Rats

Neuroscience, 2022-02, Vol.483, p.13-23

2022

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Angiotensin II inhibits the A-type K + current of hypothalamic paraventricular nucleus neurons in rats with heart failure: role of MAPK-ERK1/2 signaling

American journal of physiology. Regulatory, integrative and comparative physiology, 2022-06, Vol.322 (6), p.R526-R534

2022

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Inhibition of Brain Mitogen-Activated Protein Kinase Signaling Reduces Central Endoplasmic Reticulum Stress and Inflammation and Sympathetic Nerve Activity in Heart Failure Rats

Hypertension (Dallas, Tex. 1979), 2016-01, Vol.67 (1), p.229-236

2016

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Subfornical Organ Mediates Sympathetic and Hemodynamic Responses to Blood-Borne Proinflammatory Cytokines

Hypertension (Dallas, Tex. 1979), 2013-07, Vol.62 (1), p.118-125

2013

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ERK1/2 MAPK signaling in hypothalamic paraventricular nucleus contributes to sympathetic excitation in rats with heart failure after myocardial infarction

American journal of physiology. Heart and circulatory physiology, 2016-03, Vol.310 (6), p.H732-H739

2016

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Stress-Induced Sensitization of Angiotensin II Hypertension Is Reversed by Blockade of Angiotensin-Converting Enzyme or Tumor Necrosis Factor-α

American journal of hypertension, 2019-08, Vol.32 (9), p.909-917

2019

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Reducing brain TACE activity improves neuroinflammation and cardiac function in heart failure rats

Frontiers in physiology, 2022-11, Vol.13, p.1052304

2022

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Brain Perivascular Macrophages and the Sympathetic Response to Inflammation in Rats After Myocardial Infarction

Hypertension (Dallas, Tex. 1979), 2010-03, Vol.55 (3), p.652-659

2010

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Predator Scent-Induced Sensitization of Hypertension and Anxiety-like Behaviors

Cellular and molecular neurobiology, 2022-05, Vol.42 (4), p.1141-1152

2022

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Angiotensin II upregulates hypothalamic AT1 receptor expression in rats via the mitogen-activated protein kinase pathway

American journal of physiology. Heart and circulatory physiology, 2009-05, Vol.296 (5), p.H1425-H1433

2009

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Aldosterone acts centrally to increase brain renin-angiotensin system activity and oxidative stress in normal rats

American journal of physiology. Heart and circulatory physiology, 2008-02, Vol.294 (2), p.H1067-H1074

2008

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Maternal Angiotensin II-Induced Hypertension Sensitizes Postweaning High-Fat Diet-Elicited Hypertensive Response Through Increased Brain Reactivity in Rat Offspring

Journal of the American Heart Association, 2021-09, Vol.10 (18), p.e022170-e022170

2021

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Activation of Central PPAR-γ Attenuates Angiotensin II–Induced Hypertension

Hypertension (Dallas, Tex. 1979), 2015-08, Vol.66 (2), p.403-411

2015

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Proinflammatory cytokines upregulate sympathoexcitatory mechanisms in the subfornical organ of the rat

Leptin Mediates High-Fat Diet Sensitization of Angiotensin II–Elicited Hypertension by Upregulating the Brain Renin–Angiotensin System and Inflammation

TNF-α-induced sympathetic excitation requires EGFR and ERK1/2 signaling in cardiovascular regulatory regions of the forebrain

Angiotensin II Type 1a Receptors in the Subfornical Organ Modulate Neuroinflammation in the Hypothalamic Paraventricular Nucleus in Heart Failure Rats

Endoplasmic reticulum stress increases brain MAPK signaling, inflammation and renin-angiotensin system activity and sympathetic nerve activity in heart failure

Blood-borne interleukin-1β acts on the subfornical organ to upregulate the sympathoexcitatory milieu of the hypothalamic paraventricular nucleus

Central Renin-Angiotensin System Activation and Inflammation Induced by High-Fat Diet Sensitize Angiotensin II-Elicited Hypertension

TNF-α receptor 1 knockdown in the subfornical organ ameliorates sympathetic excitation and cardiac hemodynamics in heart failure rats

Mineralocorticoid receptors, inflammation and sympathetic drive in a rat model of systolic heart failure

Sex differences in the central and peripheral manifestations of ischemia-induced heart failure in rats

Silencing Epidermal Growth Factor Receptor in Hypothalamic Paraventricular Nucleus Reduces Extracellular Signal-regulated Kinase 1 and 2 Signaling and Sympathetic Excitation in Heart Failure Rats

Brain TACE (Tumor Necrosis Factor-α–Converting Enzyme) Contributes to Sympathetic Excitation in Heart Failure Rats

Transforming Growth Factor-α Acts in Hypothalamic Paraventricular Nucleus to Upregulate ERK1/2 Signaling and Expression of Sympathoexcitatory Mediators in Heart Failure Rats

Angiotensin II inhibits the A-type K + current of hypothalamic paraventricular nucleus neurons in rats with heart failure: role of MAPK-ERK1/2 signaling

Inhibition of Brain Mitogen-Activated Protein Kinase Signaling Reduces Central Endoplasmic Reticulum Stress and Inflammation and Sympathetic Nerve Activity in Heart Failure Rats

Subfornical Organ Mediates Sympathetic and Hemodynamic Responses to Blood-Borne Proinflammatory Cytokines

ERK1/2 MAPK signaling in hypothalamic paraventricular nucleus contributes to sympathetic excitation in rats with heart failure after myocardial infarction

Stress-Induced Sensitization of Angiotensin II Hypertension Is Reversed by Blockade of Angiotensin-Converting Enzyme or Tumor Necrosis Factor-α

Reducing brain TACE activity improves neuroinflammation and cardiac function in heart failure rats

Brain Perivascular Macrophages and the Sympathetic Response to Inflammation in Rats After Myocardial Infarction

Predator Scent-Induced Sensitization of Hypertension and Anxiety-like Behaviors

Angiotensin II upregulates hypothalamic AT1 receptor expression in rats via the mitogen-activated protein kinase pathway

Aldosterone acts centrally to increase brain renin-angiotensin system activity and oxidative stress in normal rats

Maternal Angiotensin II-Induced Hypertension Sensitizes Postweaning High-Fat Diet-Elicited Hypertensive Response Through Increased Brain Reactivity in Rat Offspring

Activation of Central PPAR-γ Attenuates Angiotensin II–Induced Hypertension

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