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Elevated serum homocysteine levels are associated with the development of chronic venous ulcers

Vascular medicine (London, England), 2022-08, Vol.27 (4), p.358-364

2022

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Negative pressure wound therapy promoted wound healing by suppressing inflammation via down-regulating MAPK-JNK signaling pathway in diabetic foot patients

Diabetes research and clinical practice, 2019-04, Vol.150, p.81-89

2019

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Circadian misalignment promotes vascular smooth muscle cell apoptosis via defective autophagy

Journal of molecular histology, 2021-08, Vol.52 (4), p.799-808

2021

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High expression levels of haem oxygenase-1 promote ferroptosis in macrophage-derived foam cells and exacerbate plaque instability

Redox biology, 2024-10, Vol.76, p.103345, Article 103345

2024

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Excimer laser ablation combined with drug-coated balloon versus drug-coated balloon in the treatment of de novo atherosclerotic lesions in lower extremities (ELABORATE): study protocol for a real-world clinical trial

BMC cardiovascular disorders, 2022-07, Vol.22 (1), p.1-317, Article 317

2022

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KTN1 Variants Underlying Putamen Gray Matter Volumes and Parkinson's Disease

Frontiers in neuroscience, 2020-06, Vol.14, p.651

2020

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CLOCK Promotes Endothelial Damage by Inducing Autophagy through Reactive Oxygen Species

Oxidative medicine and cellular longevity, 2016-01, Vol.2016 (2016), p.1-10

2016

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METTL14/YTHDF1 axis-modified UCHL5 aggravates atherosclerosis by activating the NLRP3 inflammasome

Experimental cell research, 2023-06, Vol.427 (2), p.113587-113587, Article 113587

2023

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Circadian gene CLOCK accelerates atherosclerosis by promoting endothelial autophagy

Biotechnology & genetic engineering reviews, 2024-07, Vol.40 (2), p.1230-1245

2024

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Clinical assessment of endovenous thermal ablation combined with concomitant phlebectomy for the treatment of lower limb varicose veins with or without poor glycemic control

Surgery, 2022-05, Vol.171 (5), p.1427-1433

2022

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Mzb1 Attenuates Atherosclerotic Plaque Vulnerability in ApoE-/- Mice by Alleviating Apoptosis and Modulating Mitochondrial Function

Journal of cardiovascular translational research, 2024-08, Vol.17 (4), p.782-794

2024

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Biomechanical stretch-induced CLOCK upregulation in venous smooth muscle cells promotes phenotypic and functional transformation

Vascular pharmacology, 2022-10, Vol.146, p.107097-107097, Article 107097

2022

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A Positive Feedback Loop of Profilin-1 and RhoA/ROCK1 Promotes Endothelial Dysfunction and Oxidative Stress

Oxidative medicine and cellular longevity, 2018-01, Vol.2018 (2018), p.1-9

2018

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Suppression of endoplasmic reticulum stress by 4-PBA enhanced atherosclerotic plaque stability via up-regulating CLOCK expression

Pathology, research and practice, 2024-01, Vol.253, p.154969-154969, Article 154969

2024

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Negative Pressure Wound Therapy Promotes Wound Healing by Inhibiting Inflammation in Diabetic Foot Wounds: A Role for NOD1 Receptor

International journal of lower extremity wounds, 2022-10, p.15347346221131844-15347346221131844

2022

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Pivotal Evaluation of Novel Dedicated Venous Stent for Iliofemoral Venous Obstruction: A Prospective Cohort Study

Journal of endovascular therapy, 2024-04, p.15266028241245325-15266028241245325

2024

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Effectiveness of iliac vein stenting combined with high ligation/endovenous laser treatment of the great saphenous veins in patients with Clinical, Etiology, Anatomy, Pathophysiology class 4 to 6 chronic venous disease

Journal of vascular surgery. Venous and lymphatic disorders (New York, NY), 2020-01, Vol.8 (1), p.74-83

2020

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A Positive Feedback Loop of Profilin-1 and RhoA/ROCKl Promotes Endothelial Dysfunction and Oxidative Stress

Oxidative medicine and cellular longevity, 2018-01, Vol.2018

2018

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Journal of vascular surgery. Venous and lymphatic disorders (New York, NY), 2020-09, Vol.8 (5), p.905-906

2020

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Journal of vascular surgery. Venous and lymphatic disorders (New York, NY), 2020-09, Vol.8 (5), p.905

2020

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Journal of vascular surgery. Venous and lymphatic disorders (New York, NY), 2020-09, Vol.8 (5), p.905-906

2020

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hCLOCK Causes Rho-Kinase-Mediated Endothelial Dysfunction and NF-κB-Mediated Inflammatory Responses

Oxidative medicine and cellular longevity, 2015-01, Vol.2015 (2015), p.1-9

2015

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hCLOCK induction by hypoxia promotes inflammatory responses by activating the NF‑κB pathway

Molecular medicine reports, 2017-03, Vol.15 (3), p.1401-1406

2017

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hCLOCK induction by hypoxia promotes inflammatory responses by activating the NF-[kappa]B pathway

Molecular medicine reports, 2017-03, Vol.15 (3), p.1401

2017

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Upregulation of the gene expression of CLOCK is correlated with hypoxia-inducible factor 1α in advanced varicose lesions

Molecular medicine reports, 2015-10, Vol.12 (4), p.6164-6170

2015

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Elevated serum homocysteine levels are associated with the development of chronic venous ulcers

Negative pressure wound therapy promoted wound healing by suppressing inflammation via down-regulating MAPK-JNK signaling pathway in diabetic foot patients

Circadian misalignment promotes vascular smooth muscle cell apoptosis via defective autophagy

High expression levels of haem oxygenase-1 promote ferroptosis in macrophage-derived foam cells and exacerbate plaque instability

Excimer laser ablation combined with drug-coated balloon versus drug-coated balloon in the treatment of de novo atherosclerotic lesions in lower extremities (ELABORATE): study protocol for a real-world clinical trial

KTN1 Variants Underlying Putamen Gray Matter Volumes and Parkinson's Disease

CLOCK Promotes Endothelial Damage by Inducing Autophagy through Reactive Oxygen Species

METTL14/YTHDF1 axis-modified UCHL5 aggravates atherosclerosis by activating the NLRP3 inflammasome

Circadian gene CLOCK accelerates atherosclerosis by promoting endothelial autophagy

Clinical assessment of endovenous thermal ablation combined with concomitant phlebectomy for the treatment of lower limb varicose veins with or without poor glycemic control

Mzb1 Attenuates Atherosclerotic Plaque Vulnerability in ApoE-/- Mice by Alleviating Apoptosis and Modulating Mitochondrial Function

Biomechanical stretch-induced CLOCK upregulation in venous smooth muscle cells promotes phenotypic and functional transformation

A Positive Feedback Loop of Profilin-1 and RhoA/ROCK1 Promotes Endothelial Dysfunction and Oxidative Stress

Suppression of endoplasmic reticulum stress by 4-PBA enhanced atherosclerotic plaque stability via up-regulating CLOCK expression

Negative Pressure Wound Therapy Promotes Wound Healing by Inhibiting Inflammation in Diabetic Foot Wounds: A Role for NOD1 Receptor

Pivotal Evaluation of Novel Dedicated Venous Stent for Iliofemoral Venous Obstruction: A Prospective Cohort Study

Effectiveness of iliac vein stenting combined with high ligation/endovenous laser treatment of the great saphenous veins in patients with Clinical, Etiology, Anatomy, Pathophysiology class 4 to 6 chronic venous disease

A Positive Feedback Loop of Profilin-1 and RhoA/ROCKl Promotes Endothelial Dysfunction and Oxidative Stress

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hCLOCK Causes Rho-Kinase-Mediated Endothelial Dysfunction and NF-κB-Mediated Inflammatory Responses

hCLOCK induction by hypoxia promotes inflammatory responses by activating the NF‑κB pathway

hCLOCK induction by hypoxia promotes inflammatory responses by activating the NF-[kappa]B pathway

Upregulation of the gene expression of CLOCK is correlated with hypoxia-inducible factor 1α in advanced varicose lesions

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