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Endothelial-Specific Deletion of Connexin40 Promotes Atherosclerosis by Increasing CD73-Dependent Leukocyte Adhesion

Circulation (New York, N.Y.), 2010-01, Vol.121 (1), p.123-131

2010

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Shear stress-induced atherosclerotic plaque composition in ApoE−/− mice is modulated by connexin37

Atherosclerosis, 2015-11, Vol.243 (1), p.1-10

2015

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Hypoxia-inducible factor 2alpha drives the progression of experimental non-alcoholic fatty liver disease by stimulating hepatocyte production of histidine rich glycoprotein

Journal of hepatology, 2017, Vol.66 (1), p.S169-S170

2017

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Oncostatin M induces increased invasiveness and angiogenesis in hepatic cancer cells through HIF1alpha-related release of VEGF-A

Journal of hepatology, 2017, Vol.66 (1), p.S634-S634

2017

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Hypoxia-inducible factor 2α drives the progression of experimental non-alcoholic fatty liver disease by stimulating hepatocyte production of histidine rich glycoprotein

Digestive and liver disease, 2017-02, Vol.49 (1), p.e27-e27

2017

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Serpinb3 Up-Regulates Hypoxia Inducible Factors-1alpha and -2alpha in Liver Cancer Cells through Different Mechanisms

Journal of hepatology, 2016, Vol.64 (2), p.S579-S580

2016

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Oncostatin M induces increased invasiveness and angiogenesis in hepatic cancer cells through HIF1α-related release of VEGF-A

Digestive and liver disease, 2017-02, Vol.49 (1), p.e5-e5

2017

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SerpinB3 up-regulates hypoxia inducible factors-1α and -2α in liver cancer cells through different mechanisms

Digestive and liver disease, 2016-02, Vol.48, p.e19-e19

2016

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Hypoxia-inducible factor-2α affects fibrogenic progression of experimental non-alcoholic fatty liver disease

Digestive and liver disease, 2016-02, Vol.48, p.e10-e10

2016

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PO-1814 Evaluation of strategies for dose reconstruction from prompt-gamma radiation in proton therapy

Radiotherapy and oncology, 2023-05, Vol.182, p.S1540-S1542

2023

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Oscillatory shear stress augments endothelial pannexin1 by inhibiting macro-autophagy

Cardiovascular research, 2022-06, Vol.118 (Supplement_1)

2022

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The OSM/OSMRβ signalling axis in the development of MASLD-related hepatocellular carcinoma

Digestive and liver disease, 2024-02, Vol.56, p.S95

2024

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Oncostatin M is overexpressed in NASH-related hepatocellular carcinoma and promotes a pro-tumorigenic inflammatory response

Digestive and liver disease, 2023-03, Vol.55, p.S34-S34

2023

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Regulation of endothelial Pannexin1 by oscillatory shear stress

Archives of Cardiovascular Diseases Supplements, 2020-10, Vol.12 (2-4), p.200-201

2020

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The role of histidine-rich glycoprotein in non alcoholic steatohepatitis-related experimental liver carcinogenesis

Digestive and liver disease, 2022-03, Vol.54, p.S38

2022

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Oncostatin M is overexpressed in NASH-related hepatocellular carcinoma and promotes cancer cell invasiveness and angiogenesis

Digestive and liver disease, 2022-03, Vol.54, p.S41

2022

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Oncostatin M, a novel profibrogenic mediator, is involved in the progression non-alcoholic fatty liver disease and stimulates migration of myofibroblasts

Digestive and liver disease, 2020-02, Vol.52, p.e14-e15

2020

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Targeting Cx43 protects against lung inflammation

Journal of cystic fibrosis, 2008-06, Vol.7, p.S52-S52

2008

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THU-388 - Hypoxia-inducible factor 2alpha drives the progression of experimental non-alcoholic fatty liver disease by stimulating hepatocyte production of histidine rich glycoprotein

Journal of hepatology, 2017, Vol.66 (1), p.S169-S170

2017

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Gap junctional communication in tissue inflammation and repair

Biochimica et biophysica acta, 2005-06, Vol.1711 (2), p.197-207

2005

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NASH-related liver carcinogenesis is critically affected by hypoxia-inducible factor 2α

Digestive and liver disease, 2019-02, Vol.51, p.e8-e8

2019

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SAT-111 - Oncostatin M induces increased invasiveness and angiogenesis in hepatic cancer cells through HIF1alpha-related release of VEGF-A

Journal of hepatology, 2017, Vol.66 (1), p.S634-S634

2017

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SAT-326 - Hypoxia-Inducible Factor-2A Affects Fibrogenic Progression of Experimental Non-Alcoholic Fatty Liver Disease

Journal of hepatology, 2016, Vol.64 (2), p.S682-S683

2016

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FRI-091 - Serpinb3 Up-Regulates Hypoxia Inducible Factors-1alpha and -2alpha in Liver Cancer Cells through Different Mechanisms

Journal of hepatology, 2016, Vol.64 (2), p.S579-S580

2016

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Hypoxia-Inducible Factor-2A Affects Fibrogenic Progression of Experimental Non-Alcoholic Fatty Liver Disease

Journal of hepatology, 2016, Vol.64 (2), p.S682-S683

2016

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Endothelial-Specific Deletion of Connexin40 Promotes Atherosclerosis by Increasing CD73-Dependent Leukocyte Adhesion

Shear stress-induced atherosclerotic plaque composition in ApoE−/− mice is modulated by connexin37

Hypoxia-inducible factor 2alpha drives the progression of experimental non-alcoholic fatty liver disease by stimulating hepatocyte production of histidine rich glycoprotein

Oncostatin M induces increased invasiveness and angiogenesis in hepatic cancer cells through HIF1alpha-related release of VEGF-A

Hypoxia-inducible factor 2α drives the progression of experimental non-alcoholic fatty liver disease by stimulating hepatocyte production of histidine rich glycoprotein

Serpinb3 Up-Regulates Hypoxia Inducible Factors-1alpha and -2alpha in Liver Cancer Cells through Different Mechanisms

Oncostatin M induces increased invasiveness and angiogenesis in hepatic cancer cells through HIF1α-related release of VEGF-A

SerpinB3 up-regulates hypoxia inducible factors-1α and -2α in liver cancer cells through different mechanisms

Hypoxia-inducible factor-2α affects fibrogenic progression of experimental non-alcoholic fatty liver disease

PO-1814 Evaluation of strategies for dose reconstruction from prompt-gamma radiation in proton therapy

Oscillatory shear stress augments endothelial pannexin1 by inhibiting macro-autophagy

The OSM/OSMRβ signalling axis in the development of MASLD-related hepatocellular carcinoma

Oncostatin M is overexpressed in NASH-related hepatocellular carcinoma and promotes a pro-tumorigenic inflammatory response

Regulation of endothelial Pannexin1 by oscillatory shear stress

The role of histidine-rich glycoprotein in non alcoholic steatohepatitis-related experimental liver carcinogenesis

Oncostatin M is overexpressed in NASH-related hepatocellular carcinoma and promotes cancer cell invasiveness and angiogenesis

Oncostatin M, a novel profibrogenic mediator, is involved in the progression non-alcoholic fatty liver disease and stimulates migration of myofibroblasts

Targeting Cx43 protects against lung inflammation

THU-388 - Hypoxia-inducible factor 2alpha drives the progression of experimental non-alcoholic fatty liver disease by stimulating hepatocyte production of histidine rich glycoprotein

Gap junctional communication in tissue inflammation and repair

NASH-related liver carcinogenesis is critically affected by hypoxia-inducible factor 2α

SAT-111 - Oncostatin M induces increased invasiveness and angiogenesis in hepatic cancer cells through HIF1alpha-related release of VEGF-A

SAT-326 - Hypoxia-Inducible Factor-2A Affects Fibrogenic Progression of Experimental Non-Alcoholic Fatty Liver Disease

FRI-091 - Serpinb3 Up-Regulates Hypoxia Inducible Factors-1alpha and -2alpha in Liver Cancer Cells through Different Mechanisms

Hypoxia-Inducible Factor-2A Affects Fibrogenic Progression of Experimental Non-Alcoholic Fatty Liver Disease

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