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Smad3 as a mediator of the fibrotic response

International journal of experimental pathology, 2004-04, Vol.85 (2), p.47-64

Flanders, Kathleen C.

2004

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A flexible reporter system for direct observation and isolation of cancer stem cells

Stem cell reports, 2015-01, Vol.4 (1), p.155-169

2015

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Impaired healing of cornea incision injury in a TRPV1-deficient mouse

Cell and tissue research, 2018-11, Vol.374 (2), p.329-338

2018

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Pharmacodynamic characterization of chemopreventive triterpenoids as exceptionally potent inducers of Nrf2-regulated genes

Molecular cancer therapeutics, 2007-01, Vol.6 (1), p.154-162

2007

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Targeted disruption of Smad3 confers resistance to the development of dimethylnitrosamine-induced hepatic fibrosis in mice

Liver international, 2009-08, Vol.29 (7), p.997-1009

2009

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Smad3 Signaling Is Required for Epithelial-Mesenchymal Transition of Lens Epithelium after Injury

The American journal of pathology, 2004-02, Vol.164 (2), p.651-663

2004

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Transient tumor-fibroblast interactions increase tumor cell malignancy by a TGF-Beta mediated mechanism in a mouse xenograft model of breast cancer

PloS one, 2010-03, Vol.5 (3), p.e9832-e9832

2010

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TGF-β-SMAD3 signaling mediates hepatic bile acid and phospholipid metabolism following lithocholic acid-induced liver injury[S]

Journal of lipid research, 2012-12, Vol.53 (12), p.2698-2707

2012

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Impaired cornea wound healing in a tenascin C-deficient mouse model

Laboratory investigation, 2013-02, Vol.93 (2), p.207-217

2013

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Conditional overexpression of TGF-β1 disrupts mouse salivary gland development and function

Laboratory investigation, 2010-04, Vol.90 (4), p.543-555

2010

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Erythropoietin receptor signalling is required for normal brain development

Development (Cambridge), 2002-01, Vol.129 (2), p.505-516

2002

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Mice Lacking Smad3 Are Protected Against Cutaneous Injury Induced by Ionizing Radiation

The American journal of pathology, 2002-03, Vol.160 (3), p.1057-1068

2002

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Smad3 is required for dedifferentiation of retinal pigment epithelium following retinal detachment in mice

Laboratory investigation, 2004-10, Vol.84 (10), p.1245-1258

2004

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Expression of Smad7 in Mouse Eyes Accelerates Healing of Corneal Tissue after Exposure to Alkali

The American journal of pathology, 2005-05, Vol.166 (5), p.1405-1418

2005

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Adenoviral gene transfer of BMP-7, Id2, or Id3 suppresses injury-induced epithelial-to-mesenchymal transition of lens epithelium in mice

American Journal of Physiology: Cell Physiology, 2006-01, Vol.290 (1), p.C282-C289

2006

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Transforming growth factor-β stimulates Smad1/5 signaling in pulmonary artery smooth muscle cells and fibroblasts of the newborn mouse through ALK1

American journal of physiology. Lung cellular and molecular physiology, 2017-09, Vol.313 (3), p.L615-L627

2017

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Smad3 Deficiency Alters Key Structural Elements of the Extracellular Matrix and Mechanotransduction of Wound Closure

Proceedings of the National Academy of Sciences - PNAS, 2006-06, Vol.103 (24), p.9250-9255

2006

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Inhibition of development of laser-induced choroidal neovascularization with suppression of infiltration of macrophages in Smad3-null mice

Laboratory investigation, 2016-06, Vol.96 (6), p.641-651

2016

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Inhibition of p38MAP kinase suppresses fibrotic reaction of retinal pigment epithelial cells

Laboratory investigation, 2005-07, Vol.85 (7), p.838-850

2005

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Transforming Growth Factor-β3 (TGF-β3) Knock-in Ameliorates Inflammation Due to TGF-β1 Deficiency While Promoting Glucose Tolerance

The Journal of biological chemistry, 2013-11, Vol.288 (44), p.32074-32092

2013

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Antagonizing Deactivating Cytokines To Enhance Host Defense and Chemotherapy in Experimental Visceral Leishmaniasis

Infection and Immunity, 2005-07, Vol.73 (7), p.3903-3911

2005

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TRPA1 is required for TGF-β signaling and its loss blocks inflammatory fibrosis in mouse corneal stroma

Laboratory investigation, 2014-09, Vol.94 (9), p.1030-1041

2014

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Smad3 is key to TGF-β-mediated epithelial-to-mesenchymal transition, fibrosis, tumor suppression and metastasis

Cytokine & growth factor reviews, 2006-02, Vol.17 (1), p.19-27

2006

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Suppression of In Vivo Neovascularization by the Loss of TRPV1 in Mouse Cornea

Journal of Ophthalmology, 2015-01, Vol.2015 (2015), p.1-9-181

2015

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Therapeutic effects of adenoviral gene transfer of bone morphogenic protein-7 on a corneal alkali injury model in mice

Laboratory investigation, 2005-04, Vol.85 (4), p.474-486

2005

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Smad3 as a mediator of the fibrotic response

A flexible reporter system for direct observation and isolation of cancer stem cells

Impaired healing of cornea incision injury in a TRPV1-deficient mouse

Pharmacodynamic characterization of chemopreventive triterpenoids as exceptionally potent inducers of Nrf2-regulated genes

Targeted disruption of Smad3 confers resistance to the development of dimethylnitrosamine-induced hepatic fibrosis in mice

Smad3 Signaling Is Required for Epithelial-Mesenchymal Transition of Lens Epithelium after Injury

Transient tumor-fibroblast interactions increase tumor cell malignancy by a TGF-Beta mediated mechanism in a mouse xenograft model of breast cancer

TGF-β-SMAD3 signaling mediates hepatic bile acid and phospholipid metabolism following lithocholic acid-induced liver injury[S]

Impaired cornea wound healing in a tenascin C-deficient mouse model

Conditional overexpression of TGF-β1 disrupts mouse salivary gland development and function

Erythropoietin receptor signalling is required for normal brain development

Mice Lacking Smad3 Are Protected Against Cutaneous Injury Induced by Ionizing Radiation

Smad3 is required for dedifferentiation of retinal pigment epithelium following retinal detachment in mice

Expression of Smad7 in Mouse Eyes Accelerates Healing of Corneal Tissue after Exposure to Alkali

Adenoviral gene transfer of BMP-7, Id2, or Id3 suppresses injury-induced epithelial-to-mesenchymal transition of lens epithelium in mice

Transforming growth factor-β stimulates Smad1/5 signaling in pulmonary artery smooth muscle cells and fibroblasts of the newborn mouse through ALK1

Smad3 Deficiency Alters Key Structural Elements of the Extracellular Matrix and Mechanotransduction of Wound Closure

Inhibition of development of laser-induced choroidal neovascularization with suppression of infiltration of macrophages in Smad3-null mice

Inhibition of p38MAP kinase suppresses fibrotic reaction of retinal pigment epithelial cells

Transforming Growth Factor-β3 (TGF-β3) Knock-in Ameliorates Inflammation Due to TGF-β1 Deficiency While Promoting Glucose Tolerance

Antagonizing Deactivating Cytokines To Enhance Host Defense and Chemotherapy in Experimental Visceral Leishmaniasis

TRPA1 is required for TGF-β signaling and its loss blocks inflammatory fibrosis in mouse corneal stroma

Smad3 is key to TGF-β-mediated epithelial-to-mesenchymal transition, fibrosis, tumor suppression and metastasis

Suppression of In Vivo Neovascularization by the Loss of TRPV1 in Mouse Cornea

Therapeutic effects of adenoviral gene transfer of bone morphogenic protein-7 on a corneal alkali injury model in mice

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