UB Paderborn / Katalog / Suche / in CDI

The valproic acid-induced rodent model of autism

Experimental neurology, 2018-01, Vol.299 (Pt A), p.217-227

2018

Volltextzugriff (PDF)

ProNGF and Neurodegeneration in Alzheimer's Disease

Frontiers in neuroscience, 2019-02, Vol.13, p.129-129

2019

Volltextzugriff (PDF)

Nurr1 Is Not an Essential Regulator of BDNF in Mouse Cortical Neurons

International journal of molecular sciences, 2022-06, Vol.23 (12), p.6853

2022

Volltextzugriff (PDF)

The Microglial Innate Immune Receptor TREM2 Is Required for Synapse Elimination and Normal Brain Connectivity

Immunity (Cambridge, Mass.), 2018-05, Vol.48 (5), p.979-991.e8

2018

Volltextzugriff (PDF)

ProNGF, but Not NGF, Switches from Neurotrophic to Apoptotic Activity in Response to Reductions in TrkA Receptor Levels

International journal of molecular sciences, 2017-03, Vol.18 (3), p.599-599

2017

Volltextzugriff (PDF)

Mechanisms of the Beneficial Effects of Exercise on Brain-Derived Neurotrophic Factor Expression in Alzheimer's Disease

Biomolecules (Basel, Switzerland), 2023-10, Vol.13 (11), p.1577

2023

Volltextzugriff (PDF)

Effects of Reactive Oxygen and Nitrogen Species on TrkA Expression and Signalling: Implications for proNGF in Aging and Alzheimer’s Disease

Cells (Basel, Switzerland), 2021-08, Vol.10 (8), p.1983

2021

Volltextzugriff (PDF)

Oligomeric Amyloid Decreases Basal Levels of Brain-Derived Neurotrophic factor (BDNF) mRNA via Specific Downregulation of BDNF Transcripts IV and V in Differentiated Human Neuroblastoma Cells

The Journal of neuroscience, 2007-03, Vol.27 (10), p.2628-2635

2007

Volltextzugriff (PDF)

Precursor form of brain‐derived neurotrophic factor and mature brain‐derived neurotrophic factor are decreased in the pre‐clinical stages of Alzheimer's disease

Journal of neurochemistry, 2005-06, Vol.93 (6), p.1412-1421

2005

Volltextzugriff (PDF)

A Single Bout of High-intensity Interval Exercise Increases Corticospinal Excitability, Brain-derived Neurotrophic Factor, and Uncarboxylated Osteolcalcin in Sedentary, Healthy Males

Neuroscience, 2020-06, Vol.437, p.242-255

2020

Volltextzugriff (PDF)

BDNF, proBDNF and IGF-1 serum levels in naïve and medicated subjects with autism

Scientific reports, 2022-08, Vol.12 (1), p.13768-13768, Article 13768

2022

Volltextzugriff (PDF)

Differential deregulation of NGF and BDNF neurotrophins in a transgenic rat model of Alzheimer's disease

Neurobiology of disease, 2017-12, Vol.108, p.307-323

2017

Volltextzugriff (PDF)

Induction of Oxidative Stress in SH-SY5Y Cells by Overexpression of hTau40 and Its Mitigation by Redox-Active Nanoparticles

International journal of molecular sciences, 2022-12, Vol.24 (1), p.359

2022

Volltextzugriff (PDF)

The Effects of Biological Sex and Ovarian Hormones on Exercise-Induced Neuroplasticity

Neuroscience, 2019-07, Vol.410, p.29-40

2019

Volltextzugriff (PDF)

Age-induced nitrative stress decreases retrograde transport of proNGF via TrkA and increases proNGF retrograde transport and neurodegeneration via p75NTR

Frontiers in molecular neuroscience, 2023-11, Vol.16, p.1241420-1241420

2023

Volltextzugriff (PDF)

CREB expression mediates amyloid β-induced basal BDNF downregulation

Neurobiology of aging, 2015-08, Vol.36 (8), p.2406-2413

2015

Volltextzugriff (PDF)

Decreased Brain-Derived Neurotrophic Factor Depends on Amyloid Aggregation State in Transgenic Mouse Models of Alzheimer's Disease

The Journal of neuroscience, 2009-07, Vol.29 (29), p.9321-9329

2009

Volltextzugriff (PDF)

Neuroimmunologic and Neurotrophic Interactions in Autism Spectrum Disorders: Relationship to Neuroinflammation

Neuromolecular medicine, 2018-06, Vol.20 (2), p.161-173

2018

Volltextzugriff (PDF)

Decreased mTOR signaling pathway in human idiopathic autism and in rats exposed to valproic acid

Acta neuropathologica communications, 2015-01, Vol.3 (1), p.3-3, Article 3

2015

Volltextzugriff (PDF)

Aberrant AZIN2 and polyamine metabolism precipitates tau neuropathology

The Journal of clinical investigation, 2021-02, Vol.131 (4), p.1-22

2021

Volltextzugriff (PDF)

Biological Activity of Nerve Growth Factor Precursor Is Dependent upon Relative Levels of Its Receptors

The Journal of biological chemistry, 2009-07, Vol.284 (27), p.18424-18433

2009

Volltextzugriff (PDF)

Object recognition memory and BDNF expression are reduced in young TgCRND8 mice

Neurobiology of aging, 2012-03, Vol.33 (3), p.555-563

2012

Volltextzugriff (PDF)

The Precursor Pro-Nerve Growth Factor Is the Predominant Form of Nerve Growth Factor in Brain and Is Increased in Alzheimer's Disease

Molecular and cellular neuroscience, 2001-08, Vol.18 (2), p.210-220

2001

Volltextzugriff (PDF)

Cerebrolysin modulates pronerve growth factor/nerve growth factor ratio and ameliorates the cholinergic deficit in a transgenic model of Alzheimer's disease

Journal of neuroscience research, 2013-02, Vol.91 (2), p.167-177

2013

Volltextzugriff (PDF)

Retrograde axonal transport of BDNF and proNGF diminishes with age in basal forebrain cholinergic neurons

Neurobiology of aging, 2019-12, Vol.84, p.131-140

2019

Volltextzugriff (PDF)

Menü

Weitere Dienste

Einstellungen

The valproic acid-induced rodent model of autism

ProNGF and Neurodegeneration in Alzheimer's Disease

Nurr1 Is Not an Essential Regulator of BDNF in Mouse Cortical Neurons

The Microglial Innate Immune Receptor TREM2 Is Required for Synapse Elimination and Normal Brain Connectivity

ProNGF, but Not NGF, Switches from Neurotrophic to Apoptotic Activity in Response to Reductions in TrkA Receptor Levels

Mechanisms of the Beneficial Effects of Exercise on Brain-Derived Neurotrophic Factor Expression in Alzheimer's Disease

Effects of Reactive Oxygen and Nitrogen Species on TrkA Expression and Signalling: Implications for proNGF in Aging and Alzheimer’s Disease

Oligomeric Amyloid Decreases Basal Levels of Brain-Derived Neurotrophic factor (BDNF) mRNA via Specific Downregulation of BDNF Transcripts IV and V in Differentiated Human Neuroblastoma Cells

Precursor form of brain‐derived neurotrophic factor and mature brain‐derived neurotrophic factor are decreased in the pre‐clinical stages of Alzheimer's disease

A Single Bout of High-intensity Interval Exercise Increases Corticospinal Excitability, Brain-derived Neurotrophic Factor, and Uncarboxylated Osteolcalcin in Sedentary, Healthy Males

BDNF, proBDNF and IGF-1 serum levels in naïve and medicated subjects with autism

Differential deregulation of NGF and BDNF neurotrophins in a transgenic rat model of Alzheimer's disease

Induction of Oxidative Stress in SH-SY5Y Cells by Overexpression of hTau40 and Its Mitigation by Redox-Active Nanoparticles

The Effects of Biological Sex and Ovarian Hormones on Exercise-Induced Neuroplasticity

Age-induced nitrative stress decreases retrograde transport of proNGF via TrkA and increases proNGF retrograde transport and neurodegeneration via p75NTR

CREB expression mediates amyloid β-induced basal BDNF downregulation

Decreased Brain-Derived Neurotrophic Factor Depends on Amyloid Aggregation State in Transgenic Mouse Models of Alzheimer's Disease

Neuroimmunologic and Neurotrophic Interactions in Autism Spectrum Disorders: Relationship to Neuroinflammation

Decreased mTOR signaling pathway in human idiopathic autism and in rats exposed to valproic acid

Aberrant AZIN2 and polyamine metabolism precipitates tau neuropathology

Biological Activity of Nerve Growth Factor Precursor Is Dependent upon Relative Levels of Its Receptors

Object recognition memory and BDNF expression are reduced in young TgCRND8 mice

The Precursor Pro-Nerve Growth Factor Is the Predominant Form of Nerve Growth Factor in Brain and Is Increased in Alzheimer's Disease

Cerebrolysin modulates pronerve growth factor/nerve growth factor ratio and ameliorates the cholinergic deficit in a transgenic model of Alzheimer's disease

Retrograde axonal transport of BDNF and proNGF diminishes with age in basal forebrain cholinergic neurons

Suchergebnisse filtern