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A PGC-1α Isoform Induced by Resistance Training Regulates Skeletal Muscle Hypertrophy

Cell, 2012-12, Vol.151 (6), p.1319-1331

2012

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Diet-Induced Models of Non-Alcoholic Fatty Liver Disease: Food for Thought on Sugar, Fat, and Cholesterol

Cells (Basel, Switzerland), 2021-07, Vol.10 (7), p.1805

2021

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Mitochondrial Dysfunction in the Transition from NASH to HCC

Metabolites, 2019-10, Vol.9 (10), p.233

2019

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Of Mice and Men, Redux: Modern Challenges in β Cell Gene Targeting

Endocrinology (Philadelphia), 2020-08, Vol.161 (8)

2020

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The pancreas: Bandmaster of glucose homeostasis

Experimental cell research, 2017-11, Vol.360 (1), p.19-23

2017

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Development of insulin resistance in mice lacking PGC-1α in adipose tissues

Proceedings of the National Academy of Sciences - PNAS, 2012-06, Vol.109 (24), p.9635-9640

2012

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Integrated Regulation of Hepatic Metabolism by Fibroblast Growth Factor 21 (FGF21) in Vivo

Endocrinology (Philadelphia), 2011-08, Vol.152 (8), p.2996-3004

2011

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The Protein Level of PGC-1α, a Key Metabolic Regulator, Is Controlled by NADH-NQO1

Molecular and Cellular Biology, 2013-07, Vol.33 (13), p.2603-2613

2013

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An Intimate Relationship between ROS and Insulin Signalling: Implications for Antioxidant Treatment of Fatty Liver Disease

International Journal of Cell Biology, 2014, Vol.2014 (2014), p.1-9

2014

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The lncRNA H19-Derived MicroRNA-675 Promotes Liver Necroptosis by Targeting FADD

Cancers, 2021-01, Vol.13 (3), p.411

2021

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Is MASLD lost in translation in mice?

Trends in endocrinology and metabolism, 2024-06, Vol.35 (6), p.459-461

2024

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PGC-1α negatively regulates hepatic FGF21 expression by modulating the heme/Rev-Erbα axis

Proceedings of the National Academy of Sciences - PNAS, 2009-12, Vol.106 (52), p.22510-22515

2009

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Loss of hepatic Flcn protects against fibrosis and inflammation by activating autophagy pathways

Scientific reports, 2021-10, Vol.11 (1), p.21268-21268, Article 21268

2021

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Glucagon-like peptide-2

Annual review of nutrition, 2006-01, Vol.26 (1), p.391-411

2006

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Sensitivity of Lipid Metabolism and Insulin Signaling to Genetic Alterations in Hepatic Peroxisome Proliferator–Activated Receptor-γ Coactivator-1α Expression

Diabetes (New York, N.Y.), 2009-07, Vol.58 (7), p.1499-1508

2009

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PGC-1α isoforms coordinate to balance hepatic metabolism and apoptosis in inflammatory environments

Molecular metabolism (Germany), 2020-04, Vol.34, p.72-84

2020

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Estrogen Signals Through Peroxisome Proliferator-Activated Receptor−γ Coactivator 1α to Reduce Oxidative Damage Associated With Diet-induced Fatty Liver Disease

Gastroenterology (New York, N.Y. 1943), 2017-01, Vol.152 (1), p.243-256

2017

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ErbB Signaling Is Required for the Proliferative Actions of GLP-2 in the Murine Gut

Gastroenterology (New York, N.Y. 1943), 2009-09, Vol.137 (3), p.986-996

2009

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PGC1A regulates the IRS1: IRS2 ratio during fasting to influence hepatic metabolism downstream of insulin

Proceedings of the National Academy of Sciences - PNAS, 2019-03, Vol.116 (10), p.4285-4290

2019

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PGC-1 coactivators in β-cells regulate lipid metabolism and are essential for insulin secretion coupled to fatty acids

Molecular metabolism (Germany), 2015-11, Vol.4 (11), p.811-822

2015

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Lower plasma PCSK9 in normocholesterolemic subjects is associated with upregulated adipose tissue surface‐expression of LDLR and CD36 and NLRP3 inflammasome

Physiological reports, 2021-02, Vol.9 (3), p.e14721-n/a

2021

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PGC-1α4 Interacts with REST to Upregulate Neuronal Genes and Augment Energy Consumption in Developing Cardiomyocytes

Cells (Basel, Switzerland), 2022-09, Vol.11 (19), p.2944

2022

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Linking Metabolic Disease With the PGC-1α Gly482Ser Polymorphism

Endocrinology (Philadelphia), 2018-02, Vol.159 (2), p.853-865

2018

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Mucosal Adaptation to Enteral Nutrients is Dependent on the Physiologic Actions of Glucagon-Like Peptide-2 in Mice

Gastroenterology (New York, N.Y. 1943), 2005-05, Vol.128 (5), p.1340-1353

2005

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Anti-diabetic drugs inhibit obesity-linked phosphorylation of PPARγ by Cdk5

Nature (London), 2010-07, Vol.466 (7305), p.451-456

2010

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A PGC-1α Isoform Induced by Resistance Training Regulates Skeletal Muscle Hypertrophy

Diet-Induced Models of Non-Alcoholic Fatty Liver Disease: Food for Thought on Sugar, Fat, and Cholesterol

Mitochondrial Dysfunction in the Transition from NASH to HCC

Of Mice and Men, Redux: Modern Challenges in β Cell Gene Targeting

The pancreas: Bandmaster of glucose homeostasis

Development of insulin resistance in mice lacking PGC-1α in adipose tissues

Integrated Regulation of Hepatic Metabolism by Fibroblast Growth Factor 21 (FGF21) in Vivo

The Protein Level of PGC-1α, a Key Metabolic Regulator, Is Controlled by NADH-NQO1

An Intimate Relationship between ROS and Insulin Signalling: Implications for Antioxidant Treatment of Fatty Liver Disease

The lncRNA H19-Derived MicroRNA-675 Promotes Liver Necroptosis by Targeting FADD

Is MASLD lost in translation in mice?

PGC-1α negatively regulates hepatic FGF21 expression by modulating the heme/Rev-Erbα axis

Loss of hepatic Flcn protects against fibrosis and inflammation by activating autophagy pathways

Glucagon-like peptide-2

Sensitivity of Lipid Metabolism and Insulin Signaling to Genetic Alterations in Hepatic Peroxisome Proliferator–Activated Receptor-γ Coactivator-1α Expression

PGC-1α isoforms coordinate to balance hepatic metabolism and apoptosis in inflammatory environments

Estrogen Signals Through Peroxisome Proliferator-Activated Receptor−γ Coactivator 1α to Reduce Oxidative Damage Associated With Diet-induced Fatty Liver Disease

ErbB Signaling Is Required for the Proliferative Actions of GLP-2 in the Murine Gut

PGC1A regulates the IRS1: IRS2 ratio during fasting to influence hepatic metabolism downstream of insulin

PGC-1 coactivators in β-cells regulate lipid metabolism and are essential for insulin secretion coupled to fatty acids

Lower plasma PCSK9 in normocholesterolemic subjects is associated with upregulated adipose tissue surface‐expression of LDLR and CD36 and NLRP3 inflammasome

PGC-1α4 Interacts with REST to Upregulate Neuronal Genes and Augment Energy Consumption in Developing Cardiomyocytes

Linking Metabolic Disease With the PGC-1α Gly482Ser Polymorphism

Mucosal Adaptation to Enteral Nutrients is Dependent on the Physiologic Actions of Glucagon-Like Peptide-2 in Mice

Anti-diabetic drugs inhibit obesity-linked phosphorylation of PPARγ by Cdk5

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