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Ca2+ toxicity due to reverse Na+/Ca2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation

Journal of neurophysiology, 2015-09, Vol.114 (3), p.1554-1564

2015

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NaV1.7 Gain-of-Function Mutations as a Continuum: A1632E Displays Physiological Changes Associated with Erythromelalgia and Paroxysmal Extreme Pain Disorder Mutations and Produces Symptoms of Both Disorders

The Journal of neuroscience, 2008-10, Vol.28 (43), p.11079-11088

2008

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Can robots patch‐clamp as well as humans? Characterization of a novel sodium channel mutation

The Journal of physiology, 2010-06, Vol.588 (11), p.1915-1927

2010

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Safety and efficacy of a Nav1.7 selective sodium channel blocker in patients with trigeminal neuralgia: a double-blind, placebo-controlled, randomised withdrawal phase 2a trial

Lancet neurology, 2017-04, Vol.16 (4), p.291-300

2017

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Human Na(v)1.8: enhanced persistent and ramp currents contribute to distinct firing properties of human DRG neurons

Journal of neurophysiology, 2015-05, Vol.113 (9), p.3172-3185

2015

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Nav1.7-A1632G Mutation from a Family with Inherited Erythromelalgia: Enhanced Firing of Dorsal Root Ganglia Neurons Evoked by Thermal Stimuli

The Journal of neuroscience, 2016-07, Vol.36 (28), p.7511-7522

2016

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A novel Nav1.7 mutation producing carbamazepine-responsive erythromelalgia

Annals of neurology, 2009-06, Vol.65 (6), p.733-741

2009

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A sodium channel mutation linked to epilepsy increases ramp and persistent current of Nav1.3 and induces hyperexcitability in hippocampal neurons

Experimental neurology, 2010-08, Vol.224 (2), p.362-368

2010

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Characterization of ion channels seen in subconfluent human dermal fibroblasts

The Journal of physiology, 1991-05, Vol.436 (1), p.579-601

Estacion, M

1991

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A sodium channel gene SCN9A polymorphism that increases nociceptor excitability

Annals of neurology, 2009-12, Vol.66 (6), p.862-866

2009

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Identification and localization of TRPC channels in the rat kidney

American journal of physiology. Renal physiology, 2006-05, Vol.290 (5), p.F1241-F1252

2006

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P2X7 receptor-dependent blebbing and the activation of Rho-effector kinases, caspases, and IL-1 beta release

The Journal of immunology (1950), 2003-06, Vol.170 (11), p.5728-5738

2003

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Human TRPC6 expressed in HEK 293 cells forms non-selective cation channels with limited Ca2+ permeability

The Journal of physiology, 2006-04, Vol.572 (2), p.359-377

2006

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Association of Immunophilins with Mammalian TRPC Channels

The Journal of biological chemistry, 2004-08, Vol.279 (33), p.34521-34529

2004

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Differential effect of D623N variant and wild-type Na(v)1.7 sodium channels on resting potential and interspike membrane potential of dorsal root ganglion neurons

Brain research, 2013-09, Vol.1529, p.165-177

2013

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Ca2+ influx via T-type channels modulates PDGF-induced replication of mouse fibroblasts

American Journal of Physiology: Cell Physiology, 1993-11, Vol.265 (5), p.C1239-C1246

1993

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Ca 2+ toxicity due to reverse Na + /Ca 2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation

Journal of neurophysiology, 2015-09, Vol.114 (3), p.1554-1564

2015

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Acute electrophysiological responses of bradykinin-stimulated human fibroblasts

The Journal of physiology, 1991-05, Vol.436 (1), p.603-620

Estacion, M

1991

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Effects of Ranolazine on Wild-Type and Mutant hNav1.7 Channels and on DRG Neuron Excitability

Molecular pain, 2010-06, Vol.6 (1), p.35-35

2010

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Activation of Human TRPC6 Channels by Receptor Stimulation

The Journal of biological chemistry, 2004-05, Vol.279 (21), p.22047-22056

2004

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Regulation of Drosophila transient receptor potential-like (TrpL) channels by phospholipase C-dependent mechanisms

The Journal of physiology, 2001-01, Vol.530 (1), p.1-19

2001

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Competence induction by PDGF requires sustained calcium influx by a mechanism distinct from storage-dependent calcium influx

Cell calcium (Edinburgh), 1993-06, Vol.14 (6), p.439-454

1993

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Calcium is permeable through a maitotoxin-activated nonselective cation channel in mouse L cells

American Journal of Physiology: Cell Physiology, 1996-04, Vol.270 (4), p.C1145-C1152

1996

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Mutations Causing Achondroplasia and Thanatophoric Dysplasia Alter bFGF-Induced Calcium Signals in Human Diploid Fibroblasts

Human molecular genetics, 1997-05, Vol.6 (5), p.681-688

1997

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Maitotoxin Induces Biphasic Interleukin-1β Secretion and Membrane Blebbing in Murine Macrophages

Molecular pharmacology, 2004-10, Vol.66 (4), p.909-920

2004

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Ca2+ toxicity due to reverse Na+/Ca2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation

NaV1.7 Gain-of-Function Mutations as a Continuum: A1632E Displays Physiological Changes Associated with Erythromelalgia and Paroxysmal Extreme Pain Disorder Mutations and Produces Symptoms of Both Disorders

Can robots patch‐clamp as well as humans? Characterization of a novel sodium channel mutation

Safety and efficacy of a Nav1.7 selective sodium channel blocker in patients with trigeminal neuralgia: a double-blind, placebo-controlled, randomised withdrawal phase 2a trial

Human Na(v)1.8: enhanced persistent and ramp currents contribute to distinct firing properties of human DRG neurons

Nav1.7-A1632G Mutation from a Family with Inherited Erythromelalgia: Enhanced Firing of Dorsal Root Ganglia Neurons Evoked by Thermal Stimuli

A novel Nav1.7 mutation producing carbamazepine-responsive erythromelalgia

A sodium channel mutation linked to epilepsy increases ramp and persistent current of Nav1.3 and induces hyperexcitability in hippocampal neurons

Characterization of ion channels seen in subconfluent human dermal fibroblasts

A sodium channel gene SCN9A polymorphism that increases nociceptor excitability

Identification and localization of TRPC channels in the rat kidney

P2X7 receptor-dependent blebbing and the activation of Rho-effector kinases, caspases, and IL-1 beta release

Human TRPC6 expressed in HEK 293 cells forms non-selective cation channels with limited Ca2+ permeability

Association of Immunophilins with Mammalian TRPC Channels

Differential effect of D623N variant and wild-type Na(v)1.7 sodium channels on resting potential and interspike membrane potential of dorsal root ganglion neurons

Ca2+ influx via T-type channels modulates PDGF-induced replication of mouse fibroblasts

Ca 2+ toxicity due to reverse Na + /Ca 2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation

Acute electrophysiological responses of bradykinin-stimulated human fibroblasts

Effects of Ranolazine on Wild-Type and Mutant hNav1.7 Channels and on DRG Neuron Excitability

Activation of Human TRPC6 Channels by Receptor Stimulation

Regulation of Drosophila transient receptor potential-like (TrpL) channels by phospholipase C-dependent mechanisms

Competence induction by PDGF requires sustained calcium influx by a mechanism distinct from storage-dependent calcium influx

Calcium is permeable through a maitotoxin-activated nonselective cation channel in mouse L cells

Mutations Causing Achondroplasia and Thanatophoric Dysplasia Alter bFGF-Induced Calcium Signals in Human Diploid Fibroblasts

Maitotoxin Induces Biphasic Interleukin-1β Secretion and Membrane Blebbing in Murine Macrophages

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