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A Portrait of Alzheimer Secretases: New Features and Familiar Faces

Science (American Association for the Advancement of Science), 2001-08, Vol.293 (5534), p.1449-1454

2001

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Point Substitution in the Central Hydrophobic Cluster of a Human β-Amyloid Congener Disrupts Peptide Folding and Abolishes Plaque Competence

Biochemistry (Easton), 1996-11, Vol.35 (44), p.13914-13921

1996

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Activity-Dependent Isolation of the Presenilin- γ-Secretase Complex Reveals Nicastrin and a γ Substrate

Proceedings of the National Academy of Sciences - PNAS, 2002-03, Vol.99 (5), p.2720-2725

2002

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Alzheimer's Disease Amyloid Propagation by a Template-Dependent Dock-Lock Mechanism

Biochemistry (Easton), 2000-05, Vol.39 (21), p.6288-6295

2000

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Zinc‐Induced Aggregation of Human and Rat β‐Amyloid Peptides In Vitro

Journal of neurochemistry, 1996-02, Vol.66 (2), p.723-732

1996

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Fibrillar beta -Amyloid Induces Microglial Phagocytosis, Expression of Inducible Nitric Oxide Synthase, and Loss of a Select Population of Neurons in the Rat CNS In Vivo

The Journal of neuroscience, 1998-03, Vol.18 (6), p.2161-2173

1998

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Aβ deposition inhibitor screen using synthetic amyloid

Nature biotechnology, 1997-03, Vol.15 (3), p.258-263

1997

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Notch and the Amyloid Precursor Protein Are Cleaved by Similar γ-Secretase(s)

Biochemistry (Easton), 2003-01, Vol.42 (1), p.137-144

2003

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Deposition of Monomeric, Not Oligomeric, Aβ Mediates Growth of Alzheimer's Disease Amyloid Plaques in Human Brain Preparations

Biochemistry (Easton), 1999-08, Vol.38 (32), p.10424-10431

1999

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In Vitro Growth of Alzheimer's Disease β-Amyloid Plaques Displays First-Order Kinetics

Biochemistry (Easton), 1996-01, Vol.35 (3), p.749-757

1996

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Activation Barriers to Structural Transition Determine Deposition Rates of Alzheimer's Disease Aβ Amyloid

Journal of structural biology, 2000-06, Vol.130 (2-3), p.174-183

2000

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Continuing strategies for inhibiting Alzheimer's gamma-secretase

Journal of molecular neuroscience, 2002-08, Vol.19 (1-2), p.83-87

2002

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[23] Deposition of soluble amyloid-β onto amyloid templates: With application for the identification of amyloid fibril extension inhibitors

Methods in Enzymology, 1999, Vol.309, p.350-374

1999

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γ‐Secretase/presenilin inhibitors for Alzheimer's disease phenocopy Notch mutations in Drosophila

The FASEB journal, 2003-01, Vol.17 (1), p.79-81

2003

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Stereochemical specificity of Alzheimer's disease β-peptide assembly

BIOPOLYM, 1999-05, Vol.49 (6), p.505-514

1999

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Amyloid-lowering isocoumarins are not direct inhibitors of γ-secretase

Nature cell biology, 2002-05, Vol.4 (5), p.E110-E111

2002

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Zinc and Alzheimer's Disease

Science (American Association for the Advancement of Science), 1995-06, Vol.268 (5219), p.1920-1923

1995

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Deposition of monomeric, not oligomeric, Abeta mediates growth of Alzheimer's disease amyloid plaques in human brain preparations

Biochemistry (Easton), 1999-08, Vol.38 (32), p.10424-10431

1999

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Apolipoprotein E affects amyloid formation but not amyloid growth in vitro: Mechanistic implications for ApoE4 enhanced amyloid burden and risk for Alzheimer's disease

Amyloid, 2002, Vol.9 (1), p.1-12

2002

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Zinc and Alzheimer's disease

Science (American Association for the Advancement of Science), 1995-06, Vol.268 (5219), p.1920-1920

1995

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Zinc and Alzheimer's disease. Authors' reply

Science (American Association for the Advancement of Science), 1995, Vol.268 (5219), p.1920-1923

1995

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Mechanisms of Aβ Production and Aβ Degradation: Routes to the Treatment of Alzheimer's Disease

Alzheimer's Disease, 2001, p.421-432

2001

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Predictors of blood pressure response in the SYMPLICITY HTN-3 trial

European heart journal, 2015-01, Vol.36 (4), p.219-227

2015

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Renal Denervation in Moderate to Severe CKD

Journal of the American Society of Nephrology, 2012-07, Vol.23 (7), p.1250-1257

2012

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Sympathetic Activation in Chronic Renal Failure

Journal of the American Society of Nephrology, 2009-05, Vol.20 (5), p.933-939

2009

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A Portrait of Alzheimer Secretases: New Features and Familiar Faces

Point Substitution in the Central Hydrophobic Cluster of a Human β-Amyloid Congener Disrupts Peptide Folding and Abolishes Plaque Competence

Activity-Dependent Isolation of the Presenilin- γ-Secretase Complex Reveals Nicastrin and a γ Substrate

Alzheimer's Disease Amyloid Propagation by a Template-Dependent Dock-Lock Mechanism

Zinc‐Induced Aggregation of Human and Rat β‐Amyloid Peptides In Vitro

Fibrillar beta -Amyloid Induces Microglial Phagocytosis, Expression of Inducible Nitric Oxide Synthase, and Loss of a Select Population of Neurons in the Rat CNS In Vivo

Aβ deposition inhibitor screen using synthetic amyloid

Notch and the Amyloid Precursor Protein Are Cleaved by Similar γ-Secretase(s)

Deposition of Monomeric, Not Oligomeric, Aβ Mediates Growth of Alzheimer's Disease Amyloid Plaques in Human Brain Preparations

In Vitro Growth of Alzheimer's Disease β-Amyloid Plaques Displays First-Order Kinetics

Activation Barriers to Structural Transition Determine Deposition Rates of Alzheimer's Disease Aβ Amyloid

Continuing strategies for inhibiting Alzheimer's gamma-secretase

[23] Deposition of soluble amyloid-β onto amyloid templates: With application for the identification of amyloid fibril extension inhibitors

γ‐Secretase/presenilin inhibitors for Alzheimer's disease phenocopy Notch mutations in Drosophila

Stereochemical specificity of Alzheimer's disease β-peptide assembly

Amyloid-lowering isocoumarins are not direct inhibitors of γ-secretase

Zinc and Alzheimer's Disease

Deposition of monomeric, not oligomeric, Abeta mediates growth of Alzheimer's disease amyloid plaques in human brain preparations

Apolipoprotein E affects amyloid formation but not amyloid growth in vitro: Mechanistic implications for ApoE4 enhanced amyloid burden and risk for Alzheimer's disease

Zinc and Alzheimer's disease

Zinc and Alzheimer's disease. Authors' reply

Mechanisms of Aβ Production and Aβ Degradation: Routes to the Treatment of Alzheimer's Disease

Predictors of blood pressure response in the SYMPLICITY HTN-3 trial

Renal Denervation in Moderate to Severe CKD

Sympathetic Activation in Chronic Renal Failure

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