UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

The actin gene family: Function follows isoform

Cytoskeleton (Hoboken, N.J.), 2010-10, Vol.67 (10), p.630-634

2010

Volltextzugriff (PDF)

Actin Is Crucial for All Kinetically Distinguishable Forms of Endocytosis at Synapses

Neuron (Cambridge, Mass.), 2016-12, Vol.92 (5), p.1020-1035

2016

Volltextzugriff (PDF)

Isometric resistance training increases strength and alters histopathology of dystrophin-deficient mouse skeletal muscle

Journal of applied physiology (1985), 2019-02, Vol.126 (2), p.363-375

2019

Volltextzugriff (PDF)

Microtubule binding distinguishes dystrophin from utrophin

Proceedings of the National Academy of Sciences - PNAS, 2014-04, Vol.111 (15), p.5723-5728

2014

Volltextzugriff (PDF)

Xanthine oxidase is hyper-active in Duchenne muscular dystrophy

Free radical biology & medicine, 2018-12, Vol.129, p.364-371

2018

Volltextzugriff (PDF)

Length regulation of mechanosensitive stereocilia depends on very slow actin dynamics and filament-severing proteins

Nature communications, 2015-04, Vol.6 (1), p.6855-6855, Article 6855

2015

Volltextzugriff (PDF)

Multi-isotope imaging mass spectrometry reveals slow protein turnover in hair-cell stereocilia

Nature (London), 2012-01, Vol.481 (7382), p.520-524

2012

Volltextzugriff (PDF)

Dystrophin Is a Microtubule-Associated Protein

The Journal of cell biology, 2009-08, Vol.186 (3), p.363-369

2009

Volltextzugriff (PDF)

An ex vivo gene therapy approach to treat muscular dystrophy using inducible pluripotent stem cells

Nature communications, 2013, Vol.4 (1), p.1549-1549, Article 1549

2013

Volltextzugriff (PDF)

Deletion of exons 2 and 3 from Actb and cell immortalization lead to widespread, β-actin independent alterations in gene expression associated with cell cycle control

European journal of cell biology, 2024-06, Vol.103 (2), p.151397, Article 151397

2024

Volltextzugriff (PDF)

β-actin and γ-actin are each dispensable for auditory hair cell development but required for Stereocilia maintenance

PLoS genetics, 2010-10, Vol.6 (10), p.e1001158-e1001158

2010

Volltextzugriff (PDF)

High Frequency Hearing Loss and Hyperactivity in DUX4 Transgenic Mice

PloS one, 2016-03, Vol.11 (3), p.e0151467

2016

Volltextzugriff (PDF)

Disease-proportional proteasomal degradation of missense dystrophins

Proceedings of the National Academy of Sciences - PNAS, 2015-10, Vol.112 (40), p.12414-12419

2015

Volltextzugriff (PDF)

Disease-causing missense mutations in actin binding domain 1 of dystrophin induce thermodynamic instability and protein aggregation

Proceedings of the National Academy of Sciences - PNAS, 2010-05, Vol.107 (21), p.9632-9637

2010

Volltextzugriff (PDF)

Loss of peroxiredoxin-2 exacerbates eccentric contraction-induced force loss in dystrophin-deficient muscle

Nature communications, 2018-11, Vol.9 (1), p.5104-14, Article 5104

2018

Volltextzugriff (PDF)

Delayed embryonic development and impaired cell growth and survival in Actg1 null mice

Cytoskeleton (Hoboken, N.J.), 2010-09, Vol.67 (9), p.564-572

2010

Volltextzugriff (PDF)

Actin Isoforms in Neuronal Development and Function

International Review of Cell and Molecular Biology, 2013, Vol.301, p.157-213

2013

Volltextzugriff (PDF)

Restricted morphological and behavioral abnormalities following ablation of β-actin in the brain

PloS one, 2012-03, Vol.7 (3), p.e32970-e32970

2012

Volltextzugriff (PDF)

Functional substitution by TAT-utrophin in dystrophin-deficient mice

PLoS medicine, 2009-05, Vol.6 (5), p.e1000083-e1000083

2009

Volltextzugriff (PDF)

Dystrophin, its interactions with other proteins, and implications for muscular dystrophy

Biochimica et biophysica acta, 2007-02, Vol.1772 (2), p.108-117

Ervasti, James M.

2007

Volltextzugriff (PDF)

γ-Actin is required for cytoskeletal maintenance but not development

Proceedings of the National Academy of Sciences - PNAS, 2009-06, Vol.106 (24), p.9703-9708

2009

Volltextzugriff (PDF)

Mechanical factors tune the sensitivity of mdx muscle to eccentric strength loss and its protection by antioxidant and calcium modulators

Skeletal muscle, 2020-02, Vol.10 (1), p.3-3, Article 3

2020

Volltextzugriff (PDF)

Loss of β-Cytoplasmic Actin in the Intestinal Epithelium Increases Gut Barrier Permeability in vivo and Exaggerates the Severity of Experimental Colitis

Frontiers in cell and developmental biology, 2020-10, Vol.8, p.588836-588836

2020

Volltextzugriff (PDF)

Contraction-Induced Loss of Plasmalemmal Electrophysiological Function Is Dependent on the Dystrophin Glycoprotein Complex

Frontiers in physiology, 2021-10, Vol.12, p.757121-757121

2021

Volltextzugriff (PDF)

Rapid, redox-mediated mechanical susceptibility of the cortical microtubule lattice in skeletal muscle

Redox biology, 2020-10, Vol.37, p.101730-101730, Article 101730

2020

Volltextzugriff (PDF)

1 – 25 von 173

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(165)
Mit Volltextzugriff
(168)

Publikationsform

Artikel
(171)
Buchkapitel
(2)

Sprache

Englisch
(171)
Japanisch
(2)

Erscheinungsjahr

n.n

Thema

Science & Technology
(129)
Animals
(107)
Life Sciences & Biomedicine
(84)
Mice
(78)
Dystrophin
(47)
Muscular Dystrophy
(46)
Multidisciplinary Sciences
(45)
Science & Technology - Other Topics
(45)
Proteins
(43)
Biochemistry & Molecular Biology
(42)
Humans
(40)
Skeletal Muscle
(39)
Rodents
(38)
Actin
(35)
Actins - Metabolism
(35)
Dystrophin - Metabolism
(34)
Muscle, Skeletal - Metabolism
(34)
Cell Biology
(32)
Muscles
(32)
Musculoskeletal System
(26)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

The actin gene family: Function follows isoform

Actin Is Crucial for All Kinetically Distinguishable Forms of Endocytosis at Synapses

Isometric resistance training increases strength and alters histopathology of dystrophin-deficient mouse skeletal muscle

Microtubule binding distinguishes dystrophin from utrophin

Xanthine oxidase is hyper-active in Duchenne muscular dystrophy

Length regulation of mechanosensitive stereocilia depends on very slow actin dynamics and filament-severing proteins

Multi-isotope imaging mass spectrometry reveals slow protein turnover in hair-cell stereocilia

Dystrophin Is a Microtubule-Associated Protein

An ex vivo gene therapy approach to treat muscular dystrophy using inducible pluripotent stem cells

Deletion of exons 2 and 3 from Actb and cell immortalization lead to widespread, β-actin independent alterations in gene expression associated with cell cycle control

β-actin and γ-actin are each dispensable for auditory hair cell development but required for Stereocilia maintenance

High Frequency Hearing Loss and Hyperactivity in DUX4 Transgenic Mice

Disease-proportional proteasomal degradation of missense dystrophins

Disease-causing missense mutations in actin binding domain 1 of dystrophin induce thermodynamic instability and protein aggregation

Loss of peroxiredoxin-2 exacerbates eccentric contraction-induced force loss in dystrophin-deficient muscle

Delayed embryonic development and impaired cell growth and survival in Actg1 null mice

Actin Isoforms in Neuronal Development and Function

Restricted morphological and behavioral abnormalities following ablation of β-actin in the brain

Functional substitution by TAT-utrophin in dystrophin-deficient mice

Dystrophin, its interactions with other proteins, and implications for muscular dystrophy

γ-Actin is required for cytoskeletal maintenance but not development

Mechanical factors tune the sensitivity of mdx muscle to eccentric strength loss and its protection by antioxidant and calcium modulators

Loss of β-Cytoplasmic Actin in the Intestinal Epithelium Increases Gut Barrier Permeability in vivo and Exaggerates the Severity of Experimental Colitis

Contraction-Induced Loss of Plasmalemmal Electrophysiological Function Is Dependent on the Dystrophin Glycoprotein Complex

Rapid, redox-mediated mechanical susceptibility of the cortical microtubule lattice in skeletal muscle

Suchergebnisse filtern