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High plasma concentrations of autoantibodies against native peptide 210 of apoB-100 are related to less coronary atherosclerosis and lower risk of myocardial infarction

European heart journal, 2008-09, Vol.29 (18), p.2218-2226

2008

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Serum matrix metalloproteinase‐3 concentration is influenced by MMP‐3 −1612 5A/6A promoter genotype and associated with myocardial infarction

Journal of internal medicine, 2005-11, Vol.258 (5), p.411-419

2005

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Angiographic assessment of effects of bezafibrate on progression of coronary artery disease in young male postinfarction patients

The Lancet (British edition), 1996-03, Vol.347 (9005), p.849-853

1996

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Peritonectomy with high voltage electrocautery generates higher levels of ultrafine smoke particles

European journal of surgical oncology, 2009-07, Vol.35 (7), p.780-784

2009

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Lower serum concentration of matrix metalloproteinase‐3 in the acute stage of myocardial infarction

Journal of internal medicine, 2006-05, Vol.259 (5), p.530-536

2006

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Treatment effects on serum lipoprotein lipids, apolipoproteins and low density lipoprotein particle size and relationships of lipoprotein variables to progression of coronary artery disease in the Bezafibrate Coronary Atherosclerosis Intervention Trial (BECAIT)

Journal of the American College of Cardiology, 1998-11, Vol.32 (6), p.1648-1656

1998

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Evidence for a role of tumor necrosis factor α in disturbances of triglyceride and glucose metabolism predisposing to coronary heart disease

Metabolism, clinical and experimental, 1998, Vol.47 (1), p.113-118

1998

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Serum insulin-like growth factor-I level is independently associated with coronary artery disease progression in young male survivors of myocardial infarction: beneficial effects of bezafibrate treatment

Journal of the American College of Cardiology, 2000-03, Vol.35 (3), p.647-654

2000

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Prognostic value of plasma C‐reactive protein and fibrinogen determinations in patients with acute myocardial infarction treated with thrombolysis

Journal of internal medicine, 2003-09, Vol.254 (3), p.244-250

2003

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Effect of Bezafibrate Treatment Over Five Years on Coronary Plaques Causing 20% to 50% Diameter Narrowing (The Bezafibrate Coronary Atherosclerosis Intervention Trial [BECAIT])

The American journal of cardiology, 1997-11, Vol.80 (9), p.1125-1129

1997

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Accumulation of apolipoprotein C-I-rich and cholesterol-rich VLDL remnants during exaggerated postprandial triglyceridemia in normolipidemic patients with coronary artery disease

Circulation (New York, N.Y.), 2000-01, Vol.101 (3), p.227-230

2000

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Helicobacter pylori seropositivity is not associated with inflammatory parameters, lipid concentrations and degree of coronary artery disease

Journal of internal medicine, 1998-02, Vol.243 (2), p.109-113

1998

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Emotional distress before coronary bypass grafting limits the benefits of surgery

The American heart journal, 1998-09, Vol.136 (3), p.510-517

1998

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Gender specific associations between matrix metalloproteinases and inflammatory markers in post myocardial infarction patients

Atherosclerosis, 2009-02, Vol.202 (2), p.550-556

2009

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Results of the Bezafibrate Coronary atherosclerosis Intervention Trial (BECAIT) and an update on trials now in progress

European heart journal, 1998-07, Vol.19 (JUL), p.H37-H41

ERICSSON, C.-G

1998

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Genetic and environmental influences on the plasma interleukin-6 concentration in patients with a recent myocardial infarction: a case-control study

Journal of interferon & cytokine research, 2011-02, Vol.31 (2), p.259-264

2011

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Human evidence that the cystatin C gene is implicated in focal progression of coronary artery disease

Arteriosclerosis, thrombosis, and vascular biology, 2004-03, Vol.24 (3), p.551-557

2004

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Diabetes mellitus is a strong negative prognostic factor in patients with myocardial infarction treated with thrombolytic therapy

Journal of internal medicine, 2000-08, Vol.248 (2), p.119-125

2000

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Relationships of plasma factor VIIa-antithrombin complexes to manifest and future cardiovascular disease

Thrombosis research, 2012-08, Vol.130 (2), p.221-225

2012

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Secondary preventive potential of lipid-lowering drugs. The Bezafibrate Coronary Atherosclerosis Intervention Trial (BECAIT)

European heart journal, 1996, Vol.17 Suppl F (suppl F), p.37-42

1996

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Human genetic evidence that OX40 is implicated in myocardial infarction

Biochemical and biophysical research communications, 2006-01, Vol.339 (3), p.1001-1006

2006

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Retardation of coronary atherosclerosis: The bezafibrate coronary atherosclerosis Intervention Trial (BECAIT) and other angiographic trials

CARDIOVASCULAR DRUGS AND THERAPY, 1997, Vol.11, p.257-263

1997

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Genetic Predisposition of the Interleukin-6 Response to Inflammation: Implications for a Variety of Major Diseases?

Clinical chemistry (Baltimore, Md.), 2004-11, Vol.50 (11), p.2136-2140

2004

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Design features of a five-year Bezafibrate Coronary Atherosclerosis Intervention Trial (BECAIT)

Drugs under experimental and clinical research, 1995, Vol.21 (3), p.105

1995

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Two‐year survival after myocardial infarction

Journal of internal medicine, 1990-03, Vol.227 (3), p.195-199

1990

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High plasma concentrations of autoantibodies against native peptide 210 of apoB-100 are related to less coronary atherosclerosis and lower risk of myocardial infarction

Serum matrix metalloproteinase‐3 concentration is influenced by MMP‐3 −1612 5A/6A promoter genotype and associated with myocardial infarction

Angiographic assessment of effects of bezafibrate on progression of coronary artery disease in young male postinfarction patients

Peritonectomy with high voltage electrocautery generates higher levels of ultrafine smoke particles

Lower serum concentration of matrix metalloproteinase‐3 in the acute stage of myocardial infarction

Treatment effects on serum lipoprotein lipids, apolipoproteins and low density lipoprotein particle size and relationships of lipoprotein variables to progression of coronary artery disease in the Bezafibrate Coronary Atherosclerosis Intervention Trial (BECAIT)

Evidence for a role of tumor necrosis factor α in disturbances of triglyceride and glucose metabolism predisposing to coronary heart disease

Serum insulin-like growth factor-I level is independently associated with coronary artery disease progression in young male survivors of myocardial infarction: beneficial effects of bezafibrate treatment

Prognostic value of plasma C‐reactive protein and fibrinogen determinations in patients with acute myocardial infarction treated with thrombolysis

Effect of Bezafibrate Treatment Over Five Years on Coronary Plaques Causing 20% to 50% Diameter Narrowing (The Bezafibrate Coronary Atherosclerosis Intervention Trial [BECAIT])

Accumulation of apolipoprotein C-I-rich and cholesterol-rich VLDL remnants during exaggerated postprandial triglyceridemia in normolipidemic patients with coronary artery disease

Helicobacter pylori seropositivity is not associated with inflammatory parameters, lipid concentrations and degree of coronary artery disease

Emotional distress before coronary bypass grafting limits the benefits of surgery

Gender specific associations between matrix metalloproteinases and inflammatory markers in post myocardial infarction patients

Results of the Bezafibrate Coronary atherosclerosis Intervention Trial (BECAIT) and an update on trials now in progress

Genetic and environmental influences on the plasma interleukin-6 concentration in patients with a recent myocardial infarction: a case-control study

Human evidence that the cystatin C gene is implicated in focal progression of coronary artery disease

Diabetes mellitus is a strong negative prognostic factor in patients with myocardial infarction treated with thrombolytic therapy

Relationships of plasma factor VIIa-antithrombin complexes to manifest and future cardiovascular disease

Secondary preventive potential of lipid-lowering drugs. The Bezafibrate Coronary Atherosclerosis Intervention Trial (BECAIT)

Human genetic evidence that OX40 is implicated in myocardial infarction

Retardation of coronary atherosclerosis: The bezafibrate coronary atherosclerosis Intervention Trial (BECAIT) and other angiographic trials

Genetic Predisposition of the Interleukin-6 Response to Inflammation: Implications for a Variety of Major Diseases?

Design features of a five-year Bezafibrate Coronary Atherosclerosis Intervention Trial (BECAIT)

Two‐year survival after myocardial infarction

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