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Simulation Modeling of Reduced Glycosylation Effects on Potassium Channels of Mouse Cardiomyocytes

Frontiers in physiology, 2022-03, Vol.13, p.816651-816651

2022

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Aberrant sialylation causes dilated cardiomyopathy and stress-induced heart failure

Basic research in cardiology, 2016-09, Vol.111 (5), p.57-57, Article 57

2016

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Isoform-specific Effects of the β2 Subunit on Voltage-gated Sodium Channel Gating

The Journal of biological chemistry, 2006-09, Vol.281 (36), p.25875-25881

2006

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Ethanol administration exacerbates the abnormalities in hepatic lipid oxidation in genetically obese mice

American journal of physiology: Gastrointestinal and liver physiology, 2013-01, Vol.304 (1), p.G38-G47

2013

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Sialic Acids Attached to O-Glycans Modulate Voltage-gated Potassium Channel Gating

The Journal of biological chemistry, 2011-02, Vol.286 (6), p.4123-4132

2011

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Regulated and aberrant glycosylation modulate cardiac electrical signaling

Proceedings of the National Academy of Sciences - PNAS, 2009-09, Vol.106 (38), p.16517-16522

2009

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Isoform-specific effects of sialic acid on voltage-dependent Na+ channel gating: functional sialic acids are localized to the S5-S6 loop of domain I

The Journal of physiology, 2002-02, Vol.538 (3), p.675-690

Bennett, Eric S.

2002

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Intracellular O-linked glycosylation directly regulates cardiomyocyte L-type Ca2+ channel activity and excitation–contraction coupling

Basic research in cardiology, 2020-12, Vol.115 (6), p.59-59, Article 59

2020

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Reduced O-GlcNAcylation diminishes cardiomyocyte Ca2+ dependent facilitation and frequency dependent acceleration of relaxation

Journal of molecular and cellular cardiology, 2023-07, Vol.180, p.10-21

2023

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Voltage-gated Na+ channels confer invasive properties on human prostate cancer cells

Pflügers Archiv, 2004-03, Vol.447 (6), p.908-914

2004

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Reductions in external divalent cations evoke novel voltage-gated currents in sensory neurons

PloS one, 2012-02, Vol.7 (2), p.e31585-e31585

2012

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Reduced myocyte complex N‐glycosylation causes dilated cardiomyopathy

The FASEB journal, 2019-01, Vol.33 (1), p.1248-1261

2019

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Reduced hybrid/complex N-glycosylation disrupts cardiac electrical signaling and calcium handling in a model of dilated cardiomyopathy

Journal of molecular and cellular cardiology, 2019-07, Vol.132, p.13-23

2019

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Model-guided concurrent data assimilation for calibrating cardiac ion-channel kinetics

IISE transactions on healthcare systems engineering, 2024-04, Vol.14 (2), p.1-14

2024

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In-Silico Modeling of the Functional Role of Reduced Sialylation in Sodium and Potassium Channel Gating of Mouse Ventricular Myocytes

IEEE journal of biomedical and health informatics, 2018-03, Vol.22 (2), p.631-639

2018

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Reduced Sialylation Impacts Ventricular Repolarization by Modulating Specific K+ Channel Isoforms Distinctly

The Journal of biological chemistry, 2015-01, Vol.290 (5), p.2769-2783

2015

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Statistical Metamodeling and Sequential Design of Computer Experiments to Model Glyco-Altered Gating of Sodium Channels in Cardiac Myocytes

IEEE journal of biomedical and health informatics, 2016-09, Vol.20 (5), p.1439-1452

2016

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High level of oxygen treatment causes cardiotoxicity with arrhythmias and redox modulation

Toxicology and applied pharmacology, 2015-01, Vol.282 (1), p.100-107

2015

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Tacrolimus induces fibroblast-to-myofibroblast transition via a TGF-β-dependent mechanism to contribute to renal fibrosis

American journal of physiology. Renal physiology, 2023-05, Vol.324 (5), p.F433-F445

2023

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Expression of the sialyltransferase, ST3Gal4, impacts cardiac voltage-gated sodium channel activity, refractory period and ventricular conduction

Journal of molecular and cellular cardiology, 2013-06, Vol.59, p.117-127

2013

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Sialic acids attached to N- and O-glycans within the Nav1.4 D1S5-S6 linker contribute to channel gating

Biochimica et biophysica acta, 2015-02, Vol.1850 (2), p.307-317

2015

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Elevated potassium outward currents in hyperoxia treated atrial cardiomyocytes

Journal of cellular physiology, 2018-05, Vol.233 (5), p.4317-4326

2018

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In-Silico Modeling of Glycosylation Modulation Dynamics in hERG Ion Channels and Cardiac Electrical Signals

IEEE journal of biomedical and health informatics, 2014-01, Vol.18 (1), p.205-214

2014

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Modulation of Voltage‐Gated Ion Channels by Sialylation

Comprehensive Physiology, 2012-04, Vol.2 (2), p.1269-1301

2012

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Gating of the shaker potassium channel is modulated differentially by N-glycosylation and sialic acids

Pflügers Archiv, 2008-05, Vol.456 (2), p.393-405

2008

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Simulation Modeling of Reduced Glycosylation Effects on Potassium Channels of Mouse Cardiomyocytes

Aberrant sialylation causes dilated cardiomyopathy and stress-induced heart failure

Isoform-specific Effects of the β2 Subunit on Voltage-gated Sodium Channel Gating

Ethanol administration exacerbates the abnormalities in hepatic lipid oxidation in genetically obese mice

Sialic Acids Attached to O-Glycans Modulate Voltage-gated Potassium Channel Gating

Regulated and aberrant glycosylation modulate cardiac electrical signaling

Isoform-specific effects of sialic acid on voltage-dependent Na+ channel gating: functional sialic acids are localized to the S5-S6 loop of domain I

Intracellular O-linked glycosylation directly regulates cardiomyocyte L-type Ca2+ channel activity and excitation–contraction coupling

Reduced O-GlcNAcylation diminishes cardiomyocyte Ca2+ dependent facilitation and frequency dependent acceleration of relaxation

Voltage-gated Na+ channels confer invasive properties on human prostate cancer cells

Reductions in external divalent cations evoke novel voltage-gated currents in sensory neurons

Reduced myocyte complex N‐glycosylation causes dilated cardiomyopathy

Reduced hybrid/complex N-glycosylation disrupts cardiac electrical signaling and calcium handling in a model of dilated cardiomyopathy

Model-guided concurrent data assimilation for calibrating cardiac ion-channel kinetics

In-Silico Modeling of the Functional Role of Reduced Sialylation in Sodium and Potassium Channel Gating of Mouse Ventricular Myocytes

Reduced Sialylation Impacts Ventricular Repolarization by Modulating Specific K+ Channel Isoforms Distinctly

Statistical Metamodeling and Sequential Design of Computer Experiments to Model Glyco-Altered Gating of Sodium Channels in Cardiac Myocytes

High level of oxygen treatment causes cardiotoxicity with arrhythmias and redox modulation

Tacrolimus induces fibroblast-to-myofibroblast transition via a TGF-β-dependent mechanism to contribute to renal fibrosis

Expression of the sialyltransferase, ST3Gal4, impacts cardiac voltage-gated sodium channel activity, refractory period and ventricular conduction

Sialic acids attached to N- and O-glycans within the Nav1.4 D1S5-S6 linker contribute to channel gating

Elevated potassium outward currents in hyperoxia treated atrial cardiomyocytes

In-Silico Modeling of Glycosylation Modulation Dynamics in hERG Ion Channels and Cardiac Electrical Signals

Modulation of Voltage‐Gated Ion Channels by Sialylation

Gating of the shaker potassium channel is modulated differentially by N-glycosylation and sialic acids

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