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Effects of dietary protein restriction on the progression of advanced renal disease in the modification of diet in renal disease study

American journal of kidney diseases, 1996-05, Vol.27 (5), p.652-663

1996

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Metabolic acidosis stimulates muscle protein degradation by activating the adenosine triphosphate-dependent pathway involving ubiquitin and proteasomes

The Journal of clinical investigation, 1994-05, Vol.93 (5), p.2127-2133

1994

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The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway

The Journal of clinical investigation, 1996-03, Vol.97 (6), p.1447-1453

1996

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Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription

The Journal of clinical investigation, 1996-10, Vol.98 (8), p.1703-1708

1996

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Acidosis and glucocorticoids concomitantly increase ubiquitin and proteasome subunit mRNAs in rat muscle

American Journal of Physiology: Cell Physiology, 1994-10, Vol.267 (4), p.C955-C960

1994

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Glucocorticoids and acidosis stimulate protein and amino acid catabolism in vivo

Kidney international, 1996-03, Vol.49 (3), p.679-683

1996

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Predictors of the progression of renal disease in the Modification of Diet in Renal Disease Study

Kidney international, 1997-06, Vol.51 (6), p.1908-1919

1997

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Mechanisms contributing to muscle wasting in acute uremia : Activation of amino acid catabolism

Journal of the American Society of Nephrology, 1998-03, Vol.9 (3), p.439-443

1998

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Abnormalities in protein synthesis and degradation induced by extracellular pH in BC3H1 myocytes

American Journal of Physiology: Cell Physiology, 1991-02, Vol.260 (2), p.C277-C282

1991

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Rat muscle branched-chain ketoacid dehydrogenase activity and mRNAs increase with extracellular acidemia

American Journal of Physiology: Cell Physiology, 1995-06, Vol.268 (6), p.C1395-C1400

1995

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Experimental acidemia and muscle cell pH in chronic acidosis and renal failure

American Journal of Physiology: Cell Physiology, 1995-09, Vol.269 (3), p.C706-C712

1995

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Mechanisms causing muscle loss in chronic renal failure

American journal of kidney diseases, 1995-07, Vol.26 (1), p.242-247

1995

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Metabolic consequences of uremia : extending the concept of adaptive responses to protein metabolism

American journal of kidney diseases, 1994-02, Vol.23 (2), p.224-228

1994

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Mechanisms That Cause Protein and Amino Acid Catabolism in Uremia

American journal of kidney diseases, 1993-01, Vol.21 (1), p.91-95

1993

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Influence of ammonia and pH on protein and amino acid metabolism in LLC-PK1 cells

Kidney international, 1992-09, Vol.42 (3), p.595-601

1992

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Protein Degradation and Increased mRNAs Encoding Proteins of the Ubiquitin-Proteasome Proteolytic Pathway in BC3H1 Myocytes Require an Interaction between Glucocorticoids and Acidification

Proceedings of the National Academy of Sciences - PNAS, 1996-03, Vol.93 (5), p.1967-1971

1996

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Effects of acute and chronic uremia on active cation transport in rat myocardium

Kidney international, 1990-12, Vol.38 (6), p.1061-1067

1990

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Acidosis and glucocorticoids interact to provoke muscle protein and amino acid catabolism

Blood purification, 1995, Vol.13 (3-4), p.147-152

1995

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Signals regulating accelerated muscle protein catabolism in uremia

Mineral and electrolyte metabolism, 1997, Vol.23 (3-6), p.198

1997

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Influence of acid loading, extracellular pH and uremia on intracellular pH in muscle

Mineral and electrolyte metabolism, 1996, Vol.22 (1-3), p.66

1996

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Metabolic acidosis accelerates whole body protein degradation and leucine oxidation by a glucocorticoid-dependent mechanism

Mineral and electrolyte metabolism, 1992, Vol.18 (2-5), p.245

1992

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Effect of glucocorticoids and extracellular pH on protein metabolism in cultured cells

Mineral and electrolyte metabolism, 1992, Vol.18 (2-5), p.316

1992

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Na pump defects in chronic uremia cannot be attributed to changes in Na-K-ATPase mRNA or protein

American journal of physiology. Renal, fluid and electrolyte physiology, 1994-04, Vol.266 (4), p.536

1994

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Toxins versus abnormal cell metabolism in the pathophysiology of chronic renal failure

Contributions to nephrology, 1993, Vol.102, p.48

1993

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Necessary but not sufficient: the role of glucocorticoids in the acidosis-induced increase in levels of mRNAs encoding proteins of the ATP-dependent proteolytic pathway in rat muscle

Mineral and electrolyte metabolism, 1996, Vol.22 (1-3), p.72

1996

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Effects of dietary protein restriction on the progression of advanced renal disease in the modification of diet in renal disease study

Metabolic acidosis stimulates muscle protein degradation by activating the adenosine triphosphate-dependent pathway involving ubiquitin and proteasomes

The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway

Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription

Acidosis and glucocorticoids concomitantly increase ubiquitin and proteasome subunit mRNAs in rat muscle

Glucocorticoids and acidosis stimulate protein and amino acid catabolism in vivo

Predictors of the progression of renal disease in the Modification of Diet in Renal Disease Study

Mechanisms contributing to muscle wasting in acute uremia : Activation of amino acid catabolism

Abnormalities in protein synthesis and degradation induced by extracellular pH in BC3H1 myocytes

Rat muscle branched-chain ketoacid dehydrogenase activity and mRNAs increase with extracellular acidemia

Experimental acidemia and muscle cell pH in chronic acidosis and renal failure

Mechanisms causing muscle loss in chronic renal failure

Metabolic consequences of uremia : extending the concept of adaptive responses to protein metabolism

Mechanisms That Cause Protein and Amino Acid Catabolism in Uremia

Influence of ammonia and pH on protein and amino acid metabolism in LLC-PK1 cells

Protein Degradation and Increased mRNAs Encoding Proteins of the Ubiquitin-Proteasome Proteolytic Pathway in BC3H1 Myocytes Require an Interaction between Glucocorticoids and Acidification

Effects of acute and chronic uremia on active cation transport in rat myocardium

Acidosis and glucocorticoids interact to provoke muscle protein and amino acid catabolism

Signals regulating accelerated muscle protein catabolism in uremia

Influence of acid loading, extracellular pH and uremia on intracellular pH in muscle

Metabolic acidosis accelerates whole body protein degradation and leucine oxidation by a glucocorticoid-dependent mechanism

Effect of glucocorticoids and extracellular pH on protein metabolism in cultured cells

Na pump defects in chronic uremia cannot be attributed to changes in Na-K-ATPase mRNA or protein

Toxins versus abnormal cell metabolism in the pathophysiology of chronic renal failure

Necessary but not sufficient: the role of glucocorticoids in the acidosis-induced increase in levels of mRNAs encoding proteins of the ATP-dependent proteolytic pathway in rat muscle

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