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Amrinone, Forerunner of Novel Cardiotonic Agents, Caused Paradigm Shift of Heart Failure Pharmacotherapy

Circulation research, 2013-08, Vol.113 (4), p.358-361

Endoh, Masao

2013

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Cardiac Ca2+ Signaling and Ca2+ Sensitizers

Circulation Journal, 2008, Vol.72(12), pp.1915-1925

Endoh, Masao

2008

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Force–frequency relationship in intact mammalian ventricular myocardium: physiological and pathophysiological relevance

European journal of pharmacology, 2004-10, Vol.500 (1), p.73-86

Endoh, Masao

2004

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Signal Transduction and Ca2+ Signaling in Intact Myocardium

Journal of Pharmacological Sciences, 2006, Vol.100(5), pp.525-537

Endoh, Masao

2006

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SERCA in heterogeneity of diastolic dysfunction in post-infarction heart failure with reduced ejection fraction

Cardiovascular research, 2019-03, Vol.115 (4), p.693-695

Endoh, Masao

2019

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Cardiac α 1 -Adrenoceptors and Inotropy: Myofilament Ca 2+ Sensitivity, Intracellular Ca2+ Mobilization, Signaling Pathway, and Pathophysiological Relevance

Circulation research, 2016-08, Vol.119 (5), p.587-590

Endoh, Masao

2016

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Cardiac α1-Adrenoceptors and Inotropy: Myofilament Ca2+ Sensitivity, Intracellular Ca2+ Mobilization, Signaling Pathway, and Pathophysiological Relevance

Circulation research, 2016-08, Vol.119 (5), p.587-590

Endoh, Masao

2016

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Does levosimendan act as a Ca2+ sensitizer or PDE3 inhibitor?: Commentary on Orstavik etal., Br J Pharmacol 171: 5169-5181

British journal of pharmacology, 2015-09, Vol.172 (18), p.4594

Endoh, Masao

2015

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Mechanisms of Action of Novel Cardiotonic Agents

Journal of cardiovascular pharmacology, 2002-09, Vol.40 (3), p.323-338

Endoh, Masao

2002

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Does levosimendan act as a Ca 2+ sensitizer or PDE 3 inhibitor?: Commentary on Orstavik et al ., Br J Pharmacol 171: 5169–5181

British journal of pharmacology, 2015-09, Vol.172 (18), p.4594-4596

Endoh, Masao

2015

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Does levosimendan act as a Ca2+ sensitizer or PDE3 inhibitor?: Commentary on Orstavik et al., Br J Pharmacol 171: 5169–5181

British journal of pharmacology, 2015-09, Vol.172 (18), p.4594-4596

Endoh, Masao

2015

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Basic and clinical characteristics of PDE 3 inhibitors as cardiotonic agents

Cardiovascular drugs and therapy, 2007-06, Vol.21 (3), p.135-139

Endoh, Masao

2007

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Meet Our Editorial Board Member

Current cardiology reviews, 2018-08, Vol.14 (3), p.141-141

Endoh, Masao

2018

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Meet Our Editorial Board Member

Current cardiology reviews, 2018-08, Vol.14 (3), p.141

Endoh, Masao

2018

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Meet Our Editorial Board Member

Current cardiology reviews, 2018-08, Vol.14 (3), p.141-141

Endoh, Masao

2018

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Current Cardiology Reviews: Meet Our Editorial Board Member

Current cardiology reviews, 2018, Vol.14 (3), p.141-141

Masao Endoh

2018

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Dual regulation of myofilament Ca2+ sensitivity by levosimendan in normal and acidotic conditions in aequorin‐loaded canine ventricular myocardium

British journal of pharmacology, 2005-08, Vol.145 (8), p.1143-1152

2005

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The large part German medicine has played in the development of experimental pharmacology in Japan

Naunyn-Schmiedeberg's archives of pharmacology, 2023, Vol.396 (1), p.35-42

2023

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Wortmannin Inhibits the Increase in Myofilament Ca2+ Sensitivity Induced by Cross-Talk of Endothelin-1 With Norepinephrine in Canine Ventricular Myocardium

Journal of Pharmacological Sciences, 2009, Vol.109(2), pp.193-202

2009

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The therapeutic potential of novel cardiotonic agents

Expert opinion on investigational drugs, 2003-05, Vol.12 (5), p.735-750

Endoh, Masao

2003

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Inotropic effects of OR-1896, an active metabolite of levosimendan, on canine ventricular myocardium

European journal of pharmacology, 2000-07, Vol.400 (1), p.103-112

2000

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Does levosimendan act as a Ca 2+ sensitizer or PDE3 inhibitor?: Commentary on Orstavik et al., Br J Pharmacol 171: 5169-5181

British journal of pharmacology, 2015-09, Vol.172 (18), p.4594

Endoh, Masao

2015

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Wortmannin inhibits the myofilament Ca2+ sensitization induced by endothelin-1

European journal of pharmacology, 2005-01, Vol.507 (1-3), p.135-143

2005

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Acidic pH-induced Contractile Dysfunction via Downstream Mechanism: Identification of pH-sensitive Domain in Troponin I

Journal of molecular and cellular cardiology, 2001-07, Vol.33 (7), p.1297-1300

Endoh, Masao

2001

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Novel signalling cascade for cardiac hypertrophy activation by uncoupling and internalization of β1-adrenoceptors

Cardiovascular research, 2008-04, Vol.78 (1), p.5-7

Endoh, Masao

2008

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Amrinone, Forerunner of Novel Cardiotonic Agents, Caused Paradigm Shift of Heart Failure Pharmacotherapy

Cardiac Ca2+ Signaling and Ca2+ Sensitizers

Force–frequency relationship in intact mammalian ventricular myocardium: physiological and pathophysiological relevance

Signal Transduction and Ca2+ Signaling in Intact Myocardium

SERCA in heterogeneity of diastolic dysfunction in post-infarction heart failure with reduced ejection fraction

Cardiac α 1 -Adrenoceptors and Inotropy: Myofilament Ca 2+ Sensitivity, Intracellular Ca2+ Mobilization, Signaling Pathway, and Pathophysiological Relevance

Cardiac α1-Adrenoceptors and Inotropy: Myofilament Ca2+ Sensitivity, Intracellular Ca2+ Mobilization, Signaling Pathway, and Pathophysiological Relevance

Does levosimendan act as a Ca2+ sensitizer or PDE3 inhibitor?: Commentary on Orstavik etal., Br J Pharmacol 171: 5169-5181

Mechanisms of Action of Novel Cardiotonic Agents

Does levosimendan act as a Ca 2+ sensitizer or PDE 3 inhibitor?: Commentary on Orstavik et al ., Br J Pharmacol 171: 5169–5181

Does levosimendan act as a Ca2+ sensitizer or PDE3 inhibitor?: Commentary on Orstavik et al., Br J Pharmacol 171: 5169–5181

Basic and clinical characteristics of PDE 3 inhibitors as cardiotonic agents

Meet Our Editorial Board Member

Meet Our Editorial Board Member

Meet Our Editorial Board Member

Current Cardiology Reviews: Meet Our Editorial Board Member

Dual regulation of myofilament Ca2+ sensitivity by levosimendan in normal and acidotic conditions in aequorin‐loaded canine ventricular myocardium

The large part German medicine has played in the development of experimental pharmacology in Japan

Wortmannin Inhibits the Increase in Myofilament Ca2+ Sensitivity Induced by Cross-Talk of Endothelin-1 With Norepinephrine in Canine Ventricular Myocardium

The therapeutic potential of novel cardiotonic agents

Inotropic effects of OR-1896, an active metabolite of levosimendan, on canine ventricular myocardium

Does levosimendan act as a Ca 2+ sensitizer or PDE3 inhibitor?: Commentary on Orstavik et al., Br J Pharmacol 171: 5169-5181

Wortmannin inhibits the myofilament Ca2+ sensitization induced by endothelin-1

Acidic pH-induced Contractile Dysfunction via Downstream Mechanism: Identification of pH-sensitive Domain in Troponin I

Novel signalling cascade for cardiac hypertrophy activation by uncoupling and internalization of β1-adrenoceptors

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