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Impaired TRPV4-eNOS signaling in trabecular meshwork elevates intraocular pressure in glaucoma

Proceedings of the National Academy of Sciences - PNAS, 2021-04, Vol.118 (16), p.1

2021

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Modulation of Mitochondrial Metabolic Parameters and Antioxidant Enzymes in Healthy and Glaucomatous Trabecular Meshwork Cells with Hybrid Small Molecule SA-2

International journal of molecular sciences, 2023-07, Vol.24 (14), p.11557

2023

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Novel Thiol Containing Hybrid Antioxidant-Nitric Oxide Donor Small Molecules for Treatment of Glaucoma

Antioxidants, 2021-04, Vol.10 (4), p.575

2021

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Sigma-1 receptor stimulation protects retinal ganglion cells from ischemia-like insult through the activation of extracellular-signal-regulated kinases 1/2

Experimental eye research, 2014-11, Vol.128, p.156-169

2014

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Sigma-1R Protects Retinal Ganglion Cells in Optic Nerve Crush Model for Glaucoma

Investigative ophthalmology & visual science, 2021-08, Vol.62 (10), p.17-17

2021

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Sigma-1 Receptor Regulates Mitochondrial Function in Glucose- and Oxygen-Deprived Retinal Ganglion Cells

Investigative ophthalmology & visual science, 2017-05, Vol.58 (5), p.2755

2017

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Hybrid Compound SA-2 is Neuroprotective in Animal Models of Retinal Ganglion Cell Death

Investigative ophthalmology & visual science, 2019-07, Vol.60 (8), p.3064

2019

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Endogenous regulation of human Schlemm's canal cell volume by nitric oxide signaling

Investigative ophthalmology & visual science, 2010-11, Vol.51 (11), p.5817-5824

2010

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Guanylate Cyclase Activators, Cell Volume Changes and IOP Reduction

Cellular physiology and biochemistry, 2011-01, Vol.28 (6), p.1145-1154

Ellis, Dorette Z.

2011

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NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BKCa ion channel

American Journal of Physiology: Cell Physiology, 2008-06, Vol.294 (6), p.C1378-C1386

2008

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Nanoencapsulated hybrid compound SA-2 with long-lasting intraocular pressure-lowering activity in rodent eyes

Molecular vision, 2021, Vol.27, p.37-49

2021

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Neuroprotective effects of curcumin on endothelin-1 mediated cell death in hippocampal neurons

Nutritional neuroscience, 2017-05, Vol.20 (5), p.273-283

2017

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Characterization of Soluble Guanylate Cyclase in NO-Induced Increases in Aqueous Humor Outflow Facility and in the Trabecular Meshwork

Investigative ophthalmology & visual science, 2009-04, Vol.50 (4), p.1808-1813

2009

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Human Trabecular Meshwork Cell Volume Decrease by NO-Independent Soluble Guanylate Cyclase Activators YC-1 and BAY-58-2667 Involves the BKCa Ion Channel

Investigative ophthalmology & visual science, 2009-07, Vol.50 (7), p.3353-3359

2009

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Global loss of Na,K-ATPase and its nitric oxide-mediated regulation in a transgenic mouse model of amyotrophic lateral sclerosis

The Journal of neuroscience, 2003-01, Vol.23 (1), p.43-51

2003

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Carbachol inhibits Na+-K+-ATPase activity in choroid plexus via stimulation of the NO/cGMP pathway

American Journal of Physiology: Cell Physiology, 2000-12, Vol.279 (6), p.C1685-C1693

2000

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Mitochondrial Dihydrolipoamide Dehydrogenase is Upregulated in Response to Intermittent Hypoxic Preconditioning

International journal of medical sciences, 2015-01, Vol.12 (5), p.432-440

2015

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NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BK^sub Ca^ ion channel

American Journal of Physiology: Cell Physiology, 2008-06, Vol.294 (6), p.C1378

2008

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NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BK Ca ion channel

American Journal of Physiology: Cell Physiology, 2008-06, Vol.294 (6), p.C1378-C1386

2008

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NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BK@@dCa@ ion channel

American Journal of Physiology: Cell Physiology, 2008-06, Vol.294 (6), p.C1378-C1386

2008

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NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BKCa ion channel

American Journal of Physiology: Cell Physiology, 2008, Vol.63 (6)

2008

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Activation of the BK(Ca) channel increases outflow facility and decreases trabecular meshwork cell volume

Journal of ocular pharmacology and therapeutics, 2009-08, Vol.25 (4), p.309

2009

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NO Regulation of Na,K-ATPase

Annals of the New York Academy of Sciences, 2003-04, Vol.986 (1), p.534-535

2003

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Human Trabecular Meshwork Cell Volume Decrease by NO-Independent Soluble Guanylate Cyclase Activators YC-1 and BAY-58-2667 Involves the BK Ca Ion Channel

Investigative ophthalmology & visual science, 2009-07, Vol.50 (7), p.3353

2009

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Carbachol and Nitric Oxide Inhibition of Na,K-ATPase Activity in Bovine Ciliary Processes

Investigative ophthalmology & visual science, 2001-10, Vol.42 (11), p.2625-2631

2001

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Impaired TRPV4-eNOS signaling in trabecular meshwork elevates intraocular pressure in glaucoma

Modulation of Mitochondrial Metabolic Parameters and Antioxidant Enzymes in Healthy and Glaucomatous Trabecular Meshwork Cells with Hybrid Small Molecule SA-2

Novel Thiol Containing Hybrid Antioxidant-Nitric Oxide Donor Small Molecules for Treatment of Glaucoma

Sigma-1 receptor stimulation protects retinal ganglion cells from ischemia-like insult through the activation of extracellular-signal-regulated kinases 1/2

Sigma-1R Protects Retinal Ganglion Cells in Optic Nerve Crush Model for Glaucoma

Sigma-1 Receptor Regulates Mitochondrial Function in Glucose- and Oxygen-Deprived Retinal Ganglion Cells

Hybrid Compound SA-2 is Neuroprotective in Animal Models of Retinal Ganglion Cell Death

Endogenous regulation of human Schlemm's canal cell volume by nitric oxide signaling

Guanylate Cyclase Activators, Cell Volume Changes and IOP Reduction

NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BKCa ion channel

Nanoencapsulated hybrid compound SA-2 with long-lasting intraocular pressure-lowering activity in rodent eyes

Neuroprotective effects of curcumin on endothelin-1 mediated cell death in hippocampal neurons

Characterization of Soluble Guanylate Cyclase in NO-Induced Increases in Aqueous Humor Outflow Facility and in the Trabecular Meshwork

Human Trabecular Meshwork Cell Volume Decrease by NO-Independent Soluble Guanylate Cyclase Activators YC-1 and BAY-58-2667 Involves the BKCa Ion Channel

Global loss of Na,K-ATPase and its nitric oxide-mediated regulation in a transgenic mouse model of amyotrophic lateral sclerosis

Carbachol inhibits Na+-K+-ATPase activity in choroid plexus via stimulation of the NO/cGMP pathway

Mitochondrial Dihydrolipoamide Dehydrogenase is Upregulated in Response to Intermittent Hypoxic Preconditioning

NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BK^sub Ca^ ion channel

NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BK Ca ion channel

NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BK@@dCa@ ion channel

NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BKCa ion channel

Activation of the BK(Ca) channel increases outflow facility and decreases trabecular meshwork cell volume

NO Regulation of Na,K-ATPase

Human Trabecular Meshwork Cell Volume Decrease by NO-Independent Soluble Guanylate Cyclase Activators YC-1 and BAY-58-2667 Involves the BK Ca Ion Channel

Carbachol and Nitric Oxide Inhibition of Na,K-ATPase Activity in Bovine Ciliary Processes

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