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Clostridium difficile Toxins Disrupt Epithelial Barrier Function by Altering Membrane Microdomain Localization of Tight Junction Proteins

Infection and Immunity, 2001-03, Vol.69 (3), p.1329-1336

2001

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Glucosylation of Rho proteins by Clostridium difficile toxin B

Nature (London), 1995-06, Vol.375 (6531), p.500-503

1995

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P266Down regulation of the cardiac sodium channel Nav1.5 by the MAGUK protein CASK: role of its functional domains

Cardiovascular research, 2018-04, Vol.114 (suppl_1), p.S68-S68

2018

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Clostridium difficile toxin B acts on the GTP-binding protein Rho

The Journal of biological chemistry, 1994-04, Vol.269 (14), p.10706-10712

1994

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Rho proteins and the p38-MAPK pathway are important mediators for LPS-induced interleukin-8 expression in human endothelial cells

Blood, 2000-05, Vol.95 (10), p.3044-3051

2000

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Increased number of Clostridium difficile infections and prevalence of Clostridium difficile PCR ribotype 001 in southern Germany

Euro surveillance : bulletin européen sur les maladies transmissibles, 2008-12, Vol.13 (49)

2008

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Simulation system for puncture of the Vena jugularis sinistra in horses

Biomedizinische Technik, 2012-09, Vol.57 (SI-1 Track-O), p.815-815

2012

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Evidence for a modular structure of the homologous repetitive C-terminal carbohydrate-binding sites of Clostridium difficile toxins and Streptococcus mutans glucosyltransferases

Journal of Bacteriology, 1992-10, Vol.174 (20), p.6707-6710

1992

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Transient expression of RhoA, -B, and -C GTPases in HeLa cells potentiates resistance to Clostridium difficile toxins A and B but not to Clostridium sordellii lethal toxin

Infection and Immunity, 1995-10, Vol.63 (10), p.4063-4071

1995

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Clostridium difficile infection in Europe: a hospital-based survey

The Lancet (British edition), 2011, Vol.377 (9759), p.63-73

2011

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Emergence of clostridium difficile ribotype 027 in Germany: epidemiological and clinical characteristics

Zeitschrift fur Gastroenterologie, 2010-09, Vol.48 (9), p.1120

2010

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Purification of two high molecular weight toxins of Clostridium difficile which are antigenically related

Microbial pathogenesis, 1987-05, Vol.2 (5), p.307-318

1987

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A novel toxinotyping scheme and correlation of toxinotypes with serogroups of Clostridium difficile isolates

Journal of clinical microbiology, 1998-08, Vol.36 (8), p.2240-2247

1998

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Specific inhibition of phorbol ester-stimulated phospholipase D by Clostridium sordellii lethal toxin and Clostridium difficile toxin B-1470 in HEK-293 cells. Restoration by Ral GTPases

The Journal of biological chemistry, 1998-03, Vol.273 (13), p.7413-7422

1998

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A comparative biochemical, pharmacological and immunological study of Clostridium novyi α-toxin, C. difficile toxin B and C. sordellii lethal toxin

Toxicon (Oxford), 1991, Vol.29 (7), p.877-887

1991

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Demonstration that the Group II Intron from the Clostridial Conjugative Transposon Tn5397 Undergoes Splicing In Vivo

Journal of Bacteriology, 2001-02, Vol.183 (4), p.1296-1299

2001

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Rho Protein Inhibition Blocks Protein Kinase C Translocation and Activation

Biochemical and biophysical research communications, 1998-04, Vol.245 (3), p.830-834

1998

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Rho protein inhibition blocks cyclooxygenase-2 expression by proinflammatory mediators in endothelial cells

Inflammation, 2003-04, Vol.27 (2), p.89-95

2003

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O328 Community rather than hospital factors promote increase of Clostridium difficile associated diseases in Northern Bavaria

International journal of antimicrobial agents, 2007, Vol.29, p.S68-S69

2007

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Doping of InAs, GaSb and InPSb by low pressure MOVPE

Journal of crystal growth, 1997-01, Vol.170 (1-4), p.783-787

1997

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The water-soluble fraction of marine aerosol particles measured on the Island of Helgoland, North Sea

Journal of aerosol science, 1997-09, Vol.28 (suppl. 1), p.S229-S230

1997

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Rac1 and PAK1 are upstream of IKK-ε and TBK-1 in the viral activation of interferon regulatory factor-3

FEBS letters, 2004-06, Vol.567 (2), p.230-238

2004

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Characterization of the cleavage site and function of resulting cleavage fragments after limited proteolysis of Clostridium difficile toxin B (TcdB) by host cells

Microbiology (Society for General Microbiology), 2005-01, Vol.151 (Pt 1), p.199-208

2005

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Tyrosine-phosphorylation-dependent and rho-protein-mediated control of cellular phosphatidylinositol 4,5-bisphosphate levels

Biochemical journal, 1998-09, Vol.334 ( Pt 3) (3), p.625-631

1998

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Clostridium difficile Toxins Disrupt Epithelial Barrier Function by Altering Membrane Microdomain Localization of Tight Junction Proteins

Glucosylation of Rho proteins by Clostridium difficile toxin B

P266Down regulation of the cardiac sodium channel Nav1.5 by the MAGUK protein CASK: role of its functional domains

Claude de Seyssel

Clostridium difficile toxin B acts on the GTP-binding protein Rho

Rho proteins and the p38-MAPK pathway are important mediators for LPS-induced interleukin-8 expression in human endothelial cells

Increased number of Clostridium difficile infections and prevalence of Clostridium difficile PCR ribotype 001 in southern Germany

Simulation system for puncture of the Vena jugularis sinistra in horses

Evidence for a modular structure of the homologous repetitive C-terminal carbohydrate-binding sites of Clostridium difficile toxins and Streptococcus mutans glucosyltransferases

Transient expression of RhoA, -B, and -C GTPases in HeLa cells potentiates resistance to Clostridium difficile toxins A and B but not to Clostridium sordellii lethal toxin

Clostridium difficile infection in Europe: a hospital-based survey

Emergence of clostridium difficile ribotype 027 in Germany: epidemiological and clinical characteristics

Purification of two high molecular weight toxins of Clostridium difficile which are antigenically related

A novel toxinotyping scheme and correlation of toxinotypes with serogroups of Clostridium difficile isolates

Specific inhibition of phorbol ester-stimulated phospholipase D by Clostridium sordellii lethal toxin and Clostridium difficile toxin B-1470 in HEK-293 cells. Restoration by Ral GTPases

A comparative biochemical, pharmacological and immunological study of Clostridium novyi α-toxin, C. difficile toxin B and C. sordellii lethal toxin

Demonstration that the Group II Intron from the Clostridial Conjugative Transposon Tn5397 Undergoes Splicing In Vivo

Rho Protein Inhibition Blocks Protein Kinase C Translocation and Activation

Rho protein inhibition blocks cyclooxygenase-2 expression by proinflammatory mediators in endothelial cells

O328 Community rather than hospital factors promote increase of Clostridium difficile associated diseases in Northern Bavaria

Doping of InAs, GaSb and InPSb by low pressure MOVPE

The water-soluble fraction of marine aerosol particles measured on the Island of Helgoland, North Sea

Rac1 and PAK1 are upstream of IKK-ε and TBK-1 in the viral activation of interferon regulatory factor-3

Characterization of the cleavage site and function of resulting cleavage fragments after limited proteolysis of Clostridium difficile toxin B (TcdB) by host cells

Tyrosine-phosphorylation-dependent and rho-protein-mediated control of cellular phosphatidylinositol 4,5-bisphosphate levels

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