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Targeting hexokinase II to mitochondria to modulate energy metabolism and reduce ischaemia‐reperfusion injury in heart

British journal of pharmacology, 2014-04, Vol.171 (8), p.2067-2079

2014

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Deletion of the innate immune NLRP3 receptor abolishes cardiac ischemic preconditioning and is associated with decreased Il-6/STAT3 signaling

PloS one, 2012-07, Vol.7 (7), p.e40643

2012

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Reduction in Hexokinase II Levels Results in Decreased Cardiac Function and Altered Remodeling After Ischemia/Reperfusion Injury

Circulation research, 2011-01, Vol.108 (1), p.60-69

2011

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Disruption of Hexokinase II–Mitochondrial Binding Blocks Ischemic Preconditioning and Causes Rapid Cardiac Necrosis

Circulation research, 2011-05, Vol.108 (10), p.1165-1169

2011

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Ischemic preconditioning, insulin, and morphine all cause hexokinase redistribution

American journal of physiology. Heart and circulatory physiology, 2005-07, Vol.289 (1), p.H496-H499

2005

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Ischemic preconditioning affects hexokinase activity and HKII in different subcellular compartments throughout cardiac ischemia-reperfusion

Journal of applied physiology (1985), 2009-06, Vol.106 (6), p.1909-1916

2009

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Delayed ischaemic contracture onset by empagliflozin associates with NHE1 inhibition and is dependent on insulin in isolated mouse hearts

Cardiovascular research, 2019-08, Vol.115 (10), p.1533-1545

2019

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Empagliflozin effects on ischemic contracture and I/R injury in isolated mouse hearts perfused with or without insulin

The FASEB journal, 2018-04, Vol.32 (S1), p.lb292-lb292

2018

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Cyclophilin D ablation is associated with increased end-ischemic mitochondrial hexokinase activity

Scientific reports, 2017-10, Vol.7 (1), p.12749-13, Article 12749

2017

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Increased in vivo mitochondrial oxygenation with right ventricular failure induced by pulmonary arterial hypertension: mitochondrial inhibition as driver of cardiac failure?

Respiratory research, 2015-02, Vol.16 (1), p.6-6, Article 6

2015

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Hexokinase cellular trafficking in ischemia–reperfusion and ischemic preconditioning is altered in type I diabetic heart

Molecular biology reports, 2013-07, Vol.40 (7), p.4153-4160

2013

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Mitochondrial oxygen tension within the heart

Journal of molecular and cellular cardiology, 2009-06, Vol.46 (6), p.943-951

2009

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Reducing mitochondrial bound hexokinase II mediates transition from non-injurious into injurious ischemia/reperfusion of the intact heart

Journal of physiology and biochemistry, 2016-08, Vol.73 (3), p.323-333

2016

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Abstract 107: The Innate Immune NALP3 Receptor Mediates Cardiac Ischemic Preconditioning in the Isolated Heart

Circulation research, 2012-08, Vol.111 (suppl_1)

2012

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Pathophysiological Consequences of TAT-HKII Peptide Administration Are Independent of Impaired Vascular Function and Ensuing Ischemia

Circulation research, 2013-01, Vol.112 (2), p.e8-e13

2013

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Reduced hexokinase II impairs muscle function 2 wk after ischemia-reperfusion through increased cell necrosis and fibrosis

Journal of applied physiology (1985), 2012-08, Vol.113 (4), p.608-618

2012

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Reduced hexokinase II impairs muscle function 2 wk after ischemia-reperfusion through increased cell necrosis and fibrosis

Journal of applied physiology (1985), 2012, Vol.113 (2), p.608-618

2012

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Partial hexokinase II knockout results in acute ischemia–reperfusion damage in skeletal muscle of male, but not female, mice

Pflügers Archiv, 2010-04, Vol.459 (5), p.705-712

2010

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Ischemic preconditioning, insulin, and morphine all cause hexokinase redistribution

American journal of physiology. Heart and circulatory physiology, 2005-07, Vol.58 (1), p.H496-H499

2005

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ISCHEMIC PRECONDITIONING (IPC) IS ASSOCIATED WITH ALTERED TEMPORAL AND SUBCELLULAR DISTRIBUTION OF HEXOKINASE ACTIVITY

The FASEB journal, 2007, Vol.21 (6), p.A1225-A1225

2007

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Ischemic preconditioning (IPC) is associated with altered temporal and subcellular distribution of hexokinase activity

Journal of molecular and cellular cardiology, 2007-06, Vol.42 (6), p.S175-S175

2007

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Ratiometric intracellular calcium imaging in the isolated beating rat heart using indo-1 fluorescence

Journal of applied physiology (1985), 2004-12, Vol.97 (6), p.2042-2050

2004

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Commonly Used Numbers of Microspheres Affect Cardiac Vascular Resistance

Journal of cardiovascular pharmacology, 2003-02, Vol.41 (2), p.223-232

2003

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Letters to the Editor

Muscle & nerve, 1988-01, Vol.11 (1), p.89-90

1988

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Letters to the Editor

Muscle & nerve, 1988-01, Vol.11 (1), p.89-90

1988

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Male
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Hexokinase - Metabolism
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Mitochondria
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Biological And Medical Sciences
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Ischemia
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Cardiac & Cardiovascular Systems
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Cardiovascular System & Cardiology
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Fundamental And Applied Biological Sciences. Psychology
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Targeting hexokinase II to mitochondria to modulate energy metabolism and reduce ischaemia‐reperfusion injury in heart

Deletion of the innate immune NLRP3 receptor abolishes cardiac ischemic preconditioning and is associated with decreased Il-6/STAT3 signaling

Reduction in Hexokinase II Levels Results in Decreased Cardiac Function and Altered Remodeling After Ischemia/Reperfusion Injury

Disruption of Hexokinase II–Mitochondrial Binding Blocks Ischemic Preconditioning and Causes Rapid Cardiac Necrosis

Ischemic preconditioning, insulin, and morphine all cause hexokinase redistribution

Ischemic preconditioning affects hexokinase activity and HKII in different subcellular compartments throughout cardiac ischemia-reperfusion

Delayed ischaemic contracture onset by empagliflozin associates with NHE1 inhibition and is dependent on insulin in isolated mouse hearts

Empagliflozin effects on ischemic contracture and I/R injury in isolated mouse hearts perfused with or without insulin

Cyclophilin D ablation is associated with increased end-ischemic mitochondrial hexokinase activity

Increased in vivo mitochondrial oxygenation with right ventricular failure induced by pulmonary arterial hypertension: mitochondrial inhibition as driver of cardiac failure?

Hexokinase cellular trafficking in ischemia–reperfusion and ischemic preconditioning is altered in type I diabetic heart

Mitochondrial oxygen tension within the heart

Reducing mitochondrial bound hexokinase II mediates transition from non-injurious into injurious ischemia/reperfusion of the intact heart

Abstract 107: The Innate Immune NALP3 Receptor Mediates Cardiac Ischemic Preconditioning in the Isolated Heart

Pathophysiological Consequences of TAT-HKII Peptide Administration Are Independent of Impaired Vascular Function and Ensuing Ischemia

Reduced hexokinase II impairs muscle function 2 wk after ischemia-reperfusion through increased cell necrosis and fibrosis

Reduced hexokinase II impairs muscle function 2 wk after ischemia-reperfusion through increased cell necrosis and fibrosis

Partial hexokinase II knockout results in acute ischemia–reperfusion damage in skeletal muscle of male, but not female, mice

Ischemic preconditioning, insulin, and morphine all cause hexokinase redistribution

ISCHEMIC PRECONDITIONING (IPC) IS ASSOCIATED WITH ALTERED TEMPORAL AND SUBCELLULAR DISTRIBUTION OF HEXOKINASE ACTIVITY

Ischemic preconditioning (IPC) is associated with altered temporal and subcellular distribution of hexokinase activity

Ratiometric intracellular calcium imaging in the isolated beating rat heart using indo-1 fluorescence

Commonly Used Numbers of Microspheres Affect Cardiac Vascular Resistance

Letters to the Editor

Letters to the Editor

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