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The M-band: The underestimated part of the sarcomere

Biochimica et biophysica acta. Molecular cell research, 2020-03, Vol.1867 (3), p.118440-118440, Article 118440

2020

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Isolation and culture of neonatal mouse cardiomyocytes

Journal of visualized experiments, 2013-09 (79)

2013

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Cardiomyocytes Sense Matrix Rigidity through a Combination of Muscle and Non-muscle Myosin Contractions

Developmental cell, 2018-02, Vol.44 (3), p.326-336.e3

2018

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Genetics of and pathogenic mechanisms in arrhythmogenic right ventricular cardiomyopathy

Biophysical reviews, 2018-08, Vol.10 (4), p.973-982

2018

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α5β1 integrin recycling promotes Arp2/3-independent cancer cell invasion via the formin FHOD3

The Journal of cell biology, 2015-09, Vol.210 (6), p.1013-1031

2015

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OBSCN Mutations Associated with Dilated Cardiomyopathy and Haploinsufficiency

PloS one, 2015-09, Vol.10 (9), p.e0138568-e0138568

2015

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FHOD1 Is Needed for Directed Forces and Adhesion Maturation during Cell Spreading and Migration

Developmental cell, 2013-12, Vol.27 (5), p.545-559

2013

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Cardiac cytoarchitecture — why the “hardware” is important for heart function

Biochimica et biophysica acta, 2016-07, Vol.1863 (7), p.1857-1863

Ehler, Elisabeth

2016

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From A to Z and back? Multicompartment proteins in the sarcomere

Trends in cell biology, 2006, Vol.16 (1), p.11-18

2006

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Formin follows function: a muscle-specific isoform of FHOD3 is regulated by CK2 phosphorylation and promotes myofibril maintenance

The Journal of cell biology, 2010-12, Vol.191 (6), p.1159-1172

2010

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MLP and CARP are linked to chronic PKCα signalling in dilated cardiomyopathy

Nature communications, 2016-06, Vol.7 (1), p.12120-12120, Article 12120

2016

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Thymosin β4 facilitates epicardial neovascularization of the injured adult heart

Annals of the New York Academy of Sciences, 2010-05, Vol.1194 (1), p.97-104

2010

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Kinase Domain of Titin Controls Muscle Gene Expression and Protein Turnover

Science (American Association for the Advancement of Science), 2005-06, Vol.308 (5728), p.1599-1603

2005

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Establishment of cardiac cytoarchitecture in the developing mouse heart

Developmental biology, 2006-01, Vol.289 (2), p.430-441

2006

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Obscurin, a Giant Sarcomeric Rho Guanine Nucleotide Exchange Factor Protein Involved in Sarcomere Assembly

The Journal of cell biology, 2001-07, Vol.154 (1), p.123-136

2001

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Sulforaphane induced NRF2 activation in obese pregnancy attenuates developmental redox imbalance and improves early-life cardiovascular function in offspring

Redox biology, 2023-11, Vol.67, p.102883-102883, Article 102883

2023

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Mutant Muscle LIM Protein C58G causes cardiomyopathy through protein depletion

Journal of molecular and cellular cardiology, 2018-08, Vol.121, p.287-296

2018

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Two distinct phosphorylation events govern the function of muscle FHOD3

Cellular and molecular life sciences : CMLS, 2013-03, Vol.70 (5), p.893-908

2013

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Proteomics Analysis of the Cardiac Myofilament Subproteome Reveals Dynamic Alterations in Phosphatase Subunit Distribution

Molecular & cellular proteomics, 2010-03, Vol.9 (3), p.497-509

2010

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Biophysical Reviews “Meet the Editor Series”—Elisabeth Ehler

Biophysical reviews, 2021-10, Vol.13 (5), p.579-581

Ehler, Elisabeth

2021

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Actin-associated proteins and cardiomyopathy—the ‘unknown’ beyond troponin and tropomyosin

Biophysical reviews, 2018-08, Vol.10 (4), p.1121-1128

Ehler, Elisabeth

2018

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Non-muscle myosin IIB (Myh10) is required for epicardial function and coronary vessel formation during mammalian development

PLoS genetics, 2017-10, Vol.13 (10), p.e1007068

2017

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Ryanodine receptors are part of the myospryn complex in cardiac muscle

Scientific reports, 2017-07, Vol.7 (1), p.6312-12, Article 6312

2017

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The Cardiac Stress Response Factor Ms1 Can Bind to DNA and Has a Function in the Nucleus

PloS one, 2015-12, Vol.10 (12), p.e0144614-e0144614

2015

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Beyond the sarcomere: CSRP3 mutations cause hypertrophic cardiomyopathy

Human molecular genetics, 2008-09, Vol.17 (18), p.2753-2765

2008

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The M-band: The underestimated part of the sarcomere

Isolation and culture of neonatal mouse cardiomyocytes

Cardiomyocytes Sense Matrix Rigidity through a Combination of Muscle and Non-muscle Myosin Contractions

Genetics of and pathogenic mechanisms in arrhythmogenic right ventricular cardiomyopathy

α5β1 integrin recycling promotes Arp2/3-independent cancer cell invasion via the formin FHOD3

OBSCN Mutations Associated with Dilated Cardiomyopathy and Haploinsufficiency

FHOD1 Is Needed for Directed Forces and Adhesion Maturation during Cell Spreading and Migration

Cardiac cytoarchitecture — why the “hardware” is important for heart function

From A to Z and back? Multicompartment proteins in the sarcomere

Formin follows function: a muscle-specific isoform of FHOD3 is regulated by CK2 phosphorylation and promotes myofibril maintenance

MLP and CARP are linked to chronic PKCα signalling in dilated cardiomyopathy

Thymosin β4 facilitates epicardial neovascularization of the injured adult heart

Kinase Domain of Titin Controls Muscle Gene Expression and Protein Turnover

Establishment of cardiac cytoarchitecture in the developing mouse heart

Obscurin, a Giant Sarcomeric Rho Guanine Nucleotide Exchange Factor Protein Involved in Sarcomere Assembly

Sulforaphane induced NRF2 activation in obese pregnancy attenuates developmental redox imbalance and improves early-life cardiovascular function in offspring

Mutant Muscle LIM Protein C58G causes cardiomyopathy through protein depletion

Two distinct phosphorylation events govern the function of muscle FHOD3

Proteomics Analysis of the Cardiac Myofilament Subproteome Reveals Dynamic Alterations in Phosphatase Subunit Distribution

Biophysical Reviews “Meet the Editor Series”—Elisabeth Ehler

Actin-associated proteins and cardiomyopathy—the ‘unknown’ beyond troponin and tropomyosin

Non-muscle myosin IIB (Myh10) is required for epicardial function and coronary vessel formation during mammalian development

Ryanodine receptors are part of the myospryn complex in cardiac muscle

The Cardiac Stress Response Factor Ms1 Can Bind to DNA and Has a Function in the Nucleus

Beyond the sarcomere: CSRP3 mutations cause hypertrophic cardiomyopathy

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