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ORY-1001, a Potent and Selective Covalent KDM1A Inhibitor, for the Treatment of Acute Leukemia

Cancer cell, 2018-03, Vol.33 (3), p.495-511.e12

2018

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Histone demethylase LSD1 is required for germinal center formation and BCL6-driven lymphomagenesis

Nature immunology, 2019-01, Vol.20 (1), p.86-96

2019

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Rational Targeting of Cooperating Layers of the Epigenome Yields Enhanced Therapeutic Efficacy against AML

Cancer discovery, 2019-07, Vol.9 (7), p.872-889

2019

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The B Cell Mutator AID Promotes B Lymphoid Blast Crisis and Drug Resistance in Chronic Myeloid Leukemia

Cancer cell, 2009-09, Vol.16 (3), p.232-245

2009

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Pre-B cell receptor-mediated cell cycle arrest in Philadelphia chromosome-positive acute lymphoblastic leukemia requires IKAROS function

The Journal of experimental medicine, 2009-08, Vol.206 (8), p.1739-1753

2009

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Pre-B cell receptor–mediated activation of BCL6 induces pre-B cell quiescence through transcriptional repression of MYC

Blood, 2011-10, Vol.118 (15), p.4174-4178

2011

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BCL6 is critical for the development of a diverse primary B cell repertoire

The Journal of experimental medicine, 2010-06, Vol.207 (6), p.1209-1221

2010

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Activation-induced cytidine deaminase acts as a mutator in BCR-ABL1-transformed acute lymphoblastic leukemia cells

The Journal of experimental medicine, 2007-05, Vol.204 (5), p.1157-1166

2007

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Untangling the Role of Polycomb Complexes in Chemotherapy Resistance

Cancer discovery, 2018-11, Vol.8 (11), p.1348-1351

2018

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Combined epigenetic and metabolic treatments overcome differentiation blockade in acute myeloid leukemia

iScience, 2021-06, Vol.24 (6), p.102651, Article 102651

2021

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Epigenetic Mechanisms in Leukemias and Lymphomas

Cold Spring Harbor perspectives in biology, 2020-12, Vol.10 (12), p.a034959

2020

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Chemotherapy Induces Senescence-Like Resilient Cells Capable of Initiating AML Recurrence

Cancer discovery, 2021-06, Vol.11 (6), p.1542-1561

2021

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BACH2–BCL6 balance regulates selection at the pre-B cell receptor checkpoint

Trends in immunology, 2014-03, Vol.35 (3), p.131-137

2014

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Non-oncogene Addiction to SIRT3 Plays a Critical Role in Lymphomagenesis

Cancer cell, 2019-06, Vol.35 (6), p.916-931.e9

2019

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Abstract 651: Chemotherapy induces a transient senescence-like state capable of initiating AML recurrence

Cancer research (Chicago, Ill.), 2020-08, Vol.80 (16_Supplement), p.651-651

2020

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Abstract LB014: Translational activation of ATF4 through mitochondrial anaplerotic metabolic pathways is required for DLBCL growth and survival

Cancer research (Chicago, Ill.), 2021-07, Vol.81 (13_Supplement), p.LB014-LB014

2021

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Thermostability of Irreversible Unfolding α-Amylases Analyzed by Unfolding Kinetics

The Journal of biological chemistry, 2005-11, Vol.280 (45), p.37360-37365

2005

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BACH2-BCL6 balance regulates selection at the pre-BCR checkpoint

Trends in immunology, 2013-12, Vol.35 (3), p.131-137

2013

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Epigenetic balance ensures mechanistic control of MLL amplification and rearrangement

Cell, 2023-10, Vol.186 (21), p.4528-4545.e18

2023

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Hematopoietic stem cell origin of BRAFV600E mutations in hairy cell leukemia

Science translational medicine, 2014-05, Vol.6 (238), p.238ra71

2014

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BCL6 enables Ph+ acute lymphoblastic leukaemia cells to survive BCR–ABL1 kinase inhibition

Nature (London), 2011-05, Vol.473 (7347), p.384-388

2011

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Acute Myeloid Leukemia Cells Resist Chemotherapy through a Reversible Senescence-like State Maintaining Repopulation Potential

Blood, 2016-12, Vol.128 (22), p.582-582

2016

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(R)-2-Hydroxyglutarate Inhibits KDM5 Histone Lysine Demethylases to Drive Transformation in IDH-Mutant Cancers

Cancer discovery, 2023-06, Vol.13 (6), p.1478-1497

2023

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Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival

Blood cancer discovery, 2022-01, Vol.3 (1), p.50-65

2022

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ORY-1001, a Potent and Selective Covalent KDM1A Inhibitor, for the Treatment of Acute Leukemia

Histone demethylase LSD1 is required for germinal center formation and BCL6-driven lymphomagenesis

Rational Targeting of Cooperating Layers of the Epigenome Yields Enhanced Therapeutic Efficacy against AML

The B Cell Mutator AID Promotes B Lymphoid Blast Crisis and Drug Resistance in Chronic Myeloid Leukemia

Pre-B cell receptor-mediated cell cycle arrest in Philadelphia chromosome-positive acute lymphoblastic leukemia requires IKAROS function

Pre-B cell receptor–mediated activation of BCL6 induces pre-B cell quiescence through transcriptional repression of MYC

BCL6 is critical for the development of a diverse primary B cell repertoire

Activation-induced cytidine deaminase acts as a mutator in BCR-ABL1-transformed acute lymphoblastic leukemia cells

Untangling the Role of Polycomb Complexes in Chemotherapy Resistance

Combined epigenetic and metabolic treatments overcome differentiation blockade in acute myeloid leukemia

Epigenetic Mechanisms in Leukemias and Lymphomas

An Embryonic Diapause-like Adaptation with Suppressed Myc Activity Enables Tumor Treatment Persistence

Chemotherapy Induces Senescence-Like Resilient Cells Capable of Initiating AML Recurrence

BACH2–BCL6 balance regulates selection at the pre-B cell receptor checkpoint

Non-oncogene Addiction to SIRT3 Plays a Critical Role in Lymphomagenesis

Abstract 651: Chemotherapy induces a transient senescence-like state capable of initiating AML recurrence

Abstract LB014: Translational activation of ATF4 through mitochondrial anaplerotic metabolic pathways is required for DLBCL growth and survival

Thermostability of Irreversible Unfolding α-Amylases Analyzed by Unfolding Kinetics

BACH2-BCL6 balance regulates selection at the pre-BCR checkpoint

Epigenetic balance ensures mechanistic control of MLL amplification and rearrangement

Hematopoietic stem cell origin of BRAFV600E mutations in hairy cell leukemia

BCL6 enables Ph+ acute lymphoblastic leukaemia cells to survive BCR–ABL1 kinase inhibition

Acute Myeloid Leukemia Cells Resist Chemotherapy through a Reversible Senescence-like State Maintaining Repopulation Potential

(R)-2-Hydroxyglutarate Inhibits KDM5 Histone Lysine Demethylases to Drive Transformation in IDH-Mutant Cancers

Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival

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