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Periostin Promotes Cell Proliferation and Macrophage Polarization to Drive Repair after AKI

Journal of the American Society of Nephrology, 2020-01, Vol.31 (1), p.85-100

2020

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Inhibition of Periostin Expression Protects against the Development of Renal Inflammation and Fibrosis

Journal of the American Society of Nephrology, 2014-08, Vol.25 (8), p.1724-1736

2014

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Discoidin Domain Receptor 1 Is a Major Mediator of Inflammation and Fibrosis in Obstructive Nephropathy

The American journal of pathology, 2011-07, Vol.179 (1), p.83-91

2011

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Notch-3 receptor activation drives inflammation and fibrosis following tubulointerstitial kidney injury

The Journal of pathology, 2012-11, Vol.228 (3), p.286-299

2012

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Genetic inhibition of discoidin domain receptor 1 protects mice against crescentic glomerulonephritis

The FASEB journal, 2012-10, Vol.26 (10), p.4079-4091

2012

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Decreased Expression of Connexin 43 Blunts the Progression of Experimental GN

Journal of the American Society of Nephrology, 2017-10, Vol.28 (10), p.2915-2930

2017

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Asymmetric dimethylarginine (ADMA) induces chronic kidney disease through a mechanism involving collagen and TGF-β1 synthesis

The Journal of pathology, 2011-01, Vol.223 (1), p.37-45

2011

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Controversial Roles of the Renin Angiotensin System and Its Modulators During the COVID-19 Pandemic

Frontiers in physiology, 2021-02, Vol.12, p.624052-624052

2021

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Targeting connexin 43 protects against the progression of experimental chronic kidney disease in mice

Kidney international, 2014-10, Vol.86 (4), p.768-779

2014

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Proinflammatory Role of Angiotensin II in the Aorta of Normotensive Mice

BioMed research international, 2019-01, Vol.2019 (2019), p.1-11

2019

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Thrombospondin-1 plays a profibrotic and pro-inflammatory role during ureteric obstruction

Kidney international, 2012-06, Vol.81 (12), p.1226-1238

2012

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Role of Matrix Metalloproteinases in Early Hypertensive Vascular Remodeling

Hypertension (Dallas, Tex. 1979), 2007-07, Vol.50 (1), p.212-218

2007

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Identification of periostin as a critical marker of progression/reversal of hypertensive nephropathy

PloS one, 2012-03, Vol.7 (3), p.e31974

2012

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Discoidin domain receptor 1 null mice are protected against hypertension-induced renal disease

Journal of the American Society of Nephrology, 2006-12, Vol.17 (12), p.3374-3381

2006

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Endothelin receptor antagonism prevents hypoxia-induced mortality and morbidity in a mouse model of sickle-cell disease

The Journal of clinical investigation, 2008-05, Vol.118 (5), p.1924-1933

2008

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Notch3 Is Essential for Regulation of the Renal Vascular Tone

Hypertension (Dallas, Tex. 1979), 2011-06, Vol.57 (6), p.1176-1182

2011

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The Role of Discoidin Domain Receptor 1 in Inflammation, Fibrosis and Renal Disease

Nephron (2015), 2017-01, Vol.137 (3), p.212-220

2017

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Renin Inhibition Reverses Renal Disease in Transgenic Mice by Shifting the Balance Between Profibrotic and Antifibrotic Agents

Hypertension (Dallas, Tex. 1979), 2013-04, Vol.61 (4), p.901-907

2013

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Restoration of podocyte structure and improvement of chronic renal disease in transgenic mice overexpressing renin

PloS one, 2009-08, Vol.4 (8), p.e6721-e6721

2009

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The RenTg mice: a powerful tool to study renin-dependent chronic kidney disease

PloS one, 2012-12, Vol.7 (12), p.e52362-e52362

2012

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Activation of Notch3 in Glomeruli Promotes the Development of Rapidly Progressive Renal Disease

Journal of the American Society of Nephrology, 2015-07, Vol.26 (7), p.1561-1575

2015

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The role of cell plasticity in progression and reversal of renal fibrosis

International journal of experimental pathology, 2011-06, Vol.92 (3), p.151-157

2011

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Notch3 orchestrates epithelial and inflammatory responses to promote acute kidney injury

Kidney international, 2018-07, Vol.94 (1), p.126-138

2018

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Progression of renal fibrosis: the underestimated role of endothelial alterations

Fibrogenesis & tissue repair, 2012-06, Vol.5 (Suppl 1), p.S15-S15, Article S15

2012

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Periostin Promotes Cell Proliferation and Macrophage Polarization to Drive Repair after AKI

Inhibition of Periostin Expression Protects against the Development of Renal Inflammation and Fibrosis

Discoidin Domain Receptor 1 Is a Major Mediator of Inflammation and Fibrosis in Obstructive Nephropathy

Notch-3 receptor activation drives inflammation and fibrosis following tubulointerstitial kidney injury

Genetic inhibition of discoidin domain receptor 1 protects mice against crescentic glomerulonephritis

Epidermal growth factor receptor promotes glomerular injury and renal failure in rapidly progressive crescentic glomerulonephritis

Decreased Expression of Connexin 43 Blunts the Progression of Experimental GN

Asymmetric dimethylarginine (ADMA) induces chronic kidney disease through a mechanism involving collagen and TGF-β1 synthesis

Controversial Roles of the Renin Angiotensin System and Its Modulators During the COVID-19 Pandemic

Targeting connexin 43 protects against the progression of experimental chronic kidney disease in mice

Proinflammatory Role of Angiotensin II in the Aorta of Normotensive Mice

Thrombospondin-1 plays a profibrotic and pro-inflammatory role during ureteric obstruction

Role of Matrix Metalloproteinases in Early Hypertensive Vascular Remodeling

Identification of periostin as a critical marker of progression/reversal of hypertensive nephropathy

Discoidin domain receptor 1 null mice are protected against hypertension-induced renal disease

Endothelin receptor antagonism prevents hypoxia-induced mortality and morbidity in a mouse model of sickle-cell disease

Notch3 Is Essential for Regulation of the Renal Vascular Tone

The Role of Discoidin Domain Receptor 1 in Inflammation, Fibrosis and Renal Disease

Renin Inhibition Reverses Renal Disease in Transgenic Mice by Shifting the Balance Between Profibrotic and Antifibrotic Agents

Restoration of podocyte structure and improvement of chronic renal disease in transgenic mice overexpressing renin

The RenTg mice: a powerful tool to study renin-dependent chronic kidney disease

Activation of Notch3 in Glomeruli Promotes the Development of Rapidly Progressive Renal Disease

The role of cell plasticity in progression and reversal of renal fibrosis

Notch3 orchestrates epithelial and inflammatory responses to promote acute kidney injury

Progression of renal fibrosis: the underestimated role of endothelial alterations

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