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Use of Curcumin, a Natural Polyphenol for Targeting Molecular Pathways in Treating Age-Related Neurodegenerative Diseases

International journal of molecular sciences, 2018-05, Vol.19 (6), p.1637

2018

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Current understanding of the molecular mechanisms in Parkinson's disease: Targets for potential treatments

Translational neurodegeneration, 2017-10, Vol.6 (1), p.28-28, Article 28

2017

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Combination treatment of berberine and solid lipid curcumin particles increased cell death and inhibited PI3K/Akt/mTOR pathway of human cultured glioblastoma cells more effectively than did individual treatments

PloS one, 2019-12, Vol.14 (12), p.e0225660-e0225660

2019

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Curcumin and Solid Lipid Curcumin Particles Induce Autophagy, but Inhibit Mitophagy and the PI3K-Akt/mTOR Pathway in Cultured Glioblastoma Cells

International journal of molecular sciences, 2019-01, Vol.20 (2), p.399

2019

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Use of Polyamidoamine Dendrimers in Brain Diseases

Molecules (Basel, Switzerland), 2018-09, Vol.23 (9), p.2238

2018

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Retraction Note: Solid lipid curcumin particles provide greater anti-amyloid, anti-inflammatory and neuroprotective effects than curcumin in the 5xFAD mouse model of Alzheimer's disease

BMC neuroscience, 2023-02, Vol.24 (1), p.16-16, Article 16

2023

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Strategies to Maximize the Involvement of Undergraduates in Publishable Research at an R2 University

Frontiers in psychology, 2019-02, Vol.10, p.214-214

Dunbar, Gary L

2019

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Liraglutide Has Anti-Inflammatory and Anti-Amyloid Properties in Streptozotocin-Induced and 5xFAD Mouse Models of Alzheimer's Disease

International journal of molecular sciences, 2021-01, Vol.22 (2), p.860

2021

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A comparative study of dietary curcumin, nanocurcumin, and other classical amyloid-binding dyes for labeling and imaging of amyloid plaques in brain tissue of 5×-familial Alzheimer’s disease mice

Histochemistry and cell biology, 2016-11, Vol.146 (5), p.609-625

2016

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Delivery of PAMAM dendrimers and dendriplexes across natural barriers (blood–brain barrier and placental barrier) in healthy pregnant mice

Discover nano, 2024-09, Vol.19 (1), p.148-8, Article 148

2024

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Use of genetically modified mesenchymal stem cells to treat neurodegenerative diseases

International Journal of Molecular Sciences, 2014-01, Vol.15 (2), p.1719-1745

2014

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Role of Epigenetics in Stem Cell Proliferation and Differentiation: Implications for Treating Neurodegenerative Diseases

International journal of molecular sciences, 2016-02, Vol.17 (2), p.199-199

2016

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Retraction Note: Preservation of dendritic spine morphology and postsynaptic signaling markers after treatment with solid lipid curcumin particles in the 5xFAD mouse model of Alzheimer’s amyloidosis

Alzheimer's research & therapy, 2023-09, Vol.15 (1), p.1-162, Article 162

2023

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Ameliorative Properties of Boronic Compounds in In Vitro and In Vivo Models of Alzheimer's Disease

International journal of molecular sciences, 2020-09, Vol.21 (18), p.6664

2020

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Recent, Innovative Approaches to Neural Therapy and Repair: Introduction to the ASNTR Special issue from the 2015 Meeting

Cell transplantation, 2016-04, Vol.25 (4), p.619-620

Dunbar, Gary L.

2016

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Solid Lipid Curcumin Particles Protect Medium Spiny Neuronal Morphology, and Reduce Learning and Memory Deficits in the YAC128 Mouse Model of Huntington's Disease

International journal of molecular sciences, 2020-12, Vol.21 (24), p.9542

2020

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Modeling Neurological Disorders in 3D Organoids Using Human-Derived Pluripotent Stem Cells

Frontiers in cell and developmental biology, 2021-05, Vol.9, p.640212

2021

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Curcugreen Treatment Prevented Splenomegaly and Other Peripheral Organ Abnormalities in 3xTg and 5xFAD Mouse Models of Alzheimer’s Disease

Antioxidants, 2021-06, Vol.10 (6), p.899

2021

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Prion‐like mechanisms in neurodegenerative disease: Implications for Huntington's disease therapy

Stem cells translational medicine, 2020-05, Vol.9 (5), p.559-566

2020

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Correction to: Preservation of dendritic spine morphology and postsynaptic signaling markers after treatment with solid lipid curcumin particles in the 5xFAD mouse model of Alzheimer's amyloidosis

Alzheimer's research & therapy, 2022-03, Vol.14 (1), p.42-42, Article 42

2022

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Solid lipid curcumin particles provide greater anti-amyloid, anti-inflammatory and neuroprotective effects than curcumin in the 5xFAD mouse model of Alzheimer's disease

BMC neuroscience, 2018-02, Vol.19 (1), p.7-7

2018

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Silencing of the Mutant Huntingtin Gene through CRISPR-Cas9 Improves the Mitochondrial Biomarkers in an In Vitro Model of Huntington’s Disease

Cell Transplantation, 2019-04, Vol.28 (4), p.460-463

2019

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Tetrahydrocurcumin Has Similar Anti-Amyloid Properties as Curcumin: In Vitro Comparative Structure-Activity Studies

Antioxidants, 2021-10, Vol.10 (10), p.1592

2021

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Induced Pluripotent Stem Cell-Derived Neural Stem Cell Transplantations Reduced Behavioral Deficits and Ameliorated Neuropathological Changes in YAC128 Mouse Model of Huntington's Disease

Frontiers in neuroscience, 2017-11, Vol.11, p.628-628

2017

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Mesenchymal Stem Cells as Treatment for Behavioral Deficits and Neuropathology in the 5xFAD Mouse Model of Alzheimer's Disease

Cell transplantation, 2016-04, Vol.25 (4), p.687-703

2016

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Use of Curcumin, a Natural Polyphenol for Targeting Molecular Pathways in Treating Age-Related Neurodegenerative Diseases

Current understanding of the molecular mechanisms in Parkinson's disease: Targets for potential treatments

Combination treatment of berberine and solid lipid curcumin particles increased cell death and inhibited PI3K/Akt/mTOR pathway of human cultured glioblastoma cells more effectively than did individual treatments

Curcumin and Solid Lipid Curcumin Particles Induce Autophagy, but Inhibit Mitophagy and the PI3K-Akt/mTOR Pathway in Cultured Glioblastoma Cells

Use of Polyamidoamine Dendrimers in Brain Diseases

Retraction Note: Solid lipid curcumin particles provide greater anti-amyloid, anti-inflammatory and neuroprotective effects than curcumin in the 5xFAD mouse model of Alzheimer's disease

Strategies to Maximize the Involvement of Undergraduates in Publishable Research at an R2 University

Liraglutide Has Anti-Inflammatory and Anti-Amyloid Properties in Streptozotocin-Induced and 5xFAD Mouse Models of Alzheimer's Disease

A comparative study of dietary curcumin, nanocurcumin, and other classical amyloid-binding dyes for labeling and imaging of amyloid plaques in brain tissue of 5×-familial Alzheimer’s disease mice

Delivery of PAMAM dendrimers and dendriplexes across natural barriers (blood–brain barrier and placental barrier) in healthy pregnant mice

Use of genetically modified mesenchymal stem cells to treat neurodegenerative diseases

Role of Epigenetics in Stem Cell Proliferation and Differentiation: Implications for Treating Neurodegenerative Diseases

Retraction Note: Preservation of dendritic spine morphology and postsynaptic signaling markers after treatment with solid lipid curcumin particles in the 5xFAD mouse model of Alzheimer’s amyloidosis

Ameliorative Properties of Boronic Compounds in In Vitro and In Vivo Models of Alzheimer's Disease

Recent, Innovative Approaches to Neural Therapy and Repair: Introduction to the ASNTR Special issue from the 2015 Meeting

Solid Lipid Curcumin Particles Protect Medium Spiny Neuronal Morphology, and Reduce Learning and Memory Deficits in the YAC128 Mouse Model of Huntington's Disease

Modeling Neurological Disorders in 3D Organoids Using Human-Derived Pluripotent Stem Cells

Curcugreen Treatment Prevented Splenomegaly and Other Peripheral Organ Abnormalities in 3xTg and 5xFAD Mouse Models of Alzheimer’s Disease

Prion‐like mechanisms in neurodegenerative disease: Implications for Huntington's disease therapy

Correction to: Preservation of dendritic spine morphology and postsynaptic signaling markers after treatment with solid lipid curcumin particles in the 5xFAD mouse model of Alzheimer's amyloidosis

Solid lipid curcumin particles provide greater anti-amyloid, anti-inflammatory and neuroprotective effects than curcumin in the 5xFAD mouse model of Alzheimer's disease

Silencing of the Mutant Huntingtin Gene through CRISPR-Cas9 Improves the Mitochondrial Biomarkers in an In Vitro Model of Huntington’s Disease

Tetrahydrocurcumin Has Similar Anti-Amyloid Properties as Curcumin: In Vitro Comparative Structure-Activity Studies

Induced Pluripotent Stem Cell-Derived Neural Stem Cell Transplantations Reduced Behavioral Deficits and Ameliorated Neuropathological Changes in YAC128 Mouse Model of Huntington's Disease

Mesenchymal Stem Cells as Treatment for Behavioral Deficits and Neuropathology in the 5xFAD Mouse Model of Alzheimer's Disease

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