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Ultrafast and Slow Cholinergic Transmission. Different Involvement of Acetylcholinesterase Molecular Forms

Molecules (Basel, Switzerland), 2017-08, Vol.22 (8), p.1300

2017

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Synaptotagmin 1 is required for vesicular Ca2+/H+‐antiport activity

Journal of neurochemistry, 2013-07, Vol.126 (1), p.37-46

2013

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Synaptic vesicles control the time course of neurotransmitter secretion via a Ca2+/H+ antiport

The Journal of physiology, 2011-01, Vol.589 (1), p.149-167

2011

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Exocytosis, Mediatophore, and Vesicular Ca2+/H+ Antiport in Rapid Neurotransmission

Annals of the New York Academy of Sciences, 2009-01, Vol.1152 (1), p.100-112

2009

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Two SUR1-specific Histidine Residues Mandatory for Zinc-induced Activation of the Rat KATP Channel

The Journal of biological chemistry, 2005-03, Vol.280 (10), p.8793-8799

2005

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Presynaptic K super(+) Channels, Vesicular Ca super(2+)/H super(+) Antiport-Synaptotagmin, and Acetylcholinesterase, Three Mechanisms Cutting Short the Cholinergic Signal at Neuromuscular and Nerve-Electroplaque Junctions

Journal of molecular neuroscience, 2014-07, Vol.53 (3), p.377-386

2014

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Presynaptic K^sup +^ Channels, Vesicular Ca^sup 2+^/H^sup +^ Antiport--Synaptotagmin, and Acetylcholinesterase, Three Mechanisms Cutting Short the Cholinergic Signal at Neuromuscular and Nerve-Electroplaque Junctions

Journal of molecular neuroscience, 2014-07, Vol.53 (3), p.377

2014

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Zinc inhibits glutamate release via activation of pre‐synaptic KATP channels and reduces ischaemic damage in rat hippocampus

Journal of neurochemistry, 2004-09, Vol.90 (5), p.1243-1250

2004

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Zinc inhibits glutamate release via activation of pre‐synaptic K ATP channels and reduces ischaemic damage in rat hippocampus

Journal of neurochemistry, 2004-09, Vol.90 (5), p.1243-1250

2004

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Synaptotagmin 1 is required for vesicular Ca 2+ /H + ‐antiport activity

Journal of neurochemistry, 2013-07, Vol.126 (1), p.37-46

2013

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Presynaptic K(+) channels, vesicular Ca(2+)/H (+) antiport--synaptotagmin, and acetylcholinesterase, three mechanisms cutting short the cholinergic signal at neuromuscular and nerve-electroplaque junctions

Journal of molecular neuroscience, 2014-07, Vol.53 (3), p.377-386

2014

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Presynaptic K+ Channels, Vesicular Ca2+/H+ Antiport—Synaptotagmin, and Acetylcholinesterase, Three Mechanisms Cutting Short the Cholinergic Signal at Neuromuscular and Nerve–Electroplaque Junctions

Journal of molecular neuroscience, 2014-07, Vol.53 (3), p.377-386

2014

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Acetylcholine release in rapid synapses: two fast partners--mediatophore and vesicular Ca2+/H+ antiport

Journal of molecular neuroscience, 2006-01, Vol.30 (1-2), p.209-214

Dunant, Yves

2006

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Nicotine-induced and depolarisation-induced glutamate release from hippocampus mossy fibre synaptosomes: two distinct mechanisms

Journal of neurochemistry, 2009-07, Vol.110 (2), p.570-580

2009

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Acetylcholine release and the cholinergic genomic locus

Molecular neurobiology, 1998-02, Vol.16 (1), p.1-20

1998

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Acetylcholine synthesis and quantal release reconstituted by transfection of mediatophore and choline acetyltranferase cDNAs

The European journal of neuroscience, 1999-05, Vol.11 (5), p.1523-1534

1999

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Synaptic vesicles control the time course of neurotransmitter secretion via a Ca 2+ /H + antiport

The Journal of physiology, 2011-01, Vol.589 (1), p.149-167

2011

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Exocytosis, Mediatophore, and Vesicular Ca super(2+)/H super(+) Antiport in Rapid Neurotransmission

Annals of the New York Academy of Sciences, 2009-01, Vol.1152 (1), p.100-112

2009

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Ultra-Fast Versus Sustained Cholinergic Transmission: A Variety of Different Mechanisms

Journal of molecular neuroscience, 2010, Vol.40 (1-2), p.27-31

2010

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Exocytosis, Mediatophore, and Vesicular Ca 2+ /H + Antiport in Rapid Neurotransmission

Annals of the New York Academy of Sciences, 2009-01, Vol.1152 (1), p.100-112

2009

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An improved approach to freeze-fracture morphology of monolayer cell cultures

Journal of neuroscience methods, 1998-07, Vol.82 (1), p.97-103

1998

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Quantal acetylcholine release: Vesicle fusion or intramembrane particles?

Microscopy research and technique, 2000-04, Vol.49 (1), p.38-46

Dunant, Yves

2000

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Zinc‐induced changes in ionic currents of clonal rat pancreatic β‐cells: activation of ATP‐sensitive K+ channels

The Journal of physiology, 2000-12, Vol.529 (3), p.723-734

2000

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Tumor Necrosis Factor-{alpha} and Angiostatin Are Mediators of Endothelial Cytotoxicity in Bronchoalveolar Lavages of Patients with Acute Respiratory Distress Syndrome

American journal of respiratory and critical care medicine, 2002-09, Vol.166 (5), p.651

2002

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Tumor necrosis factor-α and angiostatin are mediators of endothelial cytotoxicity in bronchoalveolar lavages of patients with acute respiratory distress syndrome

American journal of respiratory and critical care medicine, 2002-09, Vol.166 (5), p.651-656

2002

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Ultrafast and Slow Cholinergic Transmission. Different Involvement of Acetylcholinesterase Molecular Forms

Synaptotagmin 1 is required for vesicular Ca2+/H+‐antiport activity

Synaptic vesicles control the time course of neurotransmitter secretion via a Ca2+/H+ antiport

Exocytosis, Mediatophore, and Vesicular Ca2+/H+ Antiport in Rapid Neurotransmission

Two SUR1-specific Histidine Residues Mandatory for Zinc-induced Activation of the Rat KATP Channel

Presynaptic K super(+) Channels, Vesicular Ca super(2+)/H super(+) Antiport-Synaptotagmin, and Acetylcholinesterase, Three Mechanisms Cutting Short the Cholinergic Signal at Neuromuscular and Nerve-Electroplaque Junctions

Presynaptic K^sup +^ Channels, Vesicular Ca^sup 2+^/H^sup +^ Antiport--Synaptotagmin, and Acetylcholinesterase, Three Mechanisms Cutting Short the Cholinergic Signal at Neuromuscular and Nerve-Electroplaque Junctions

Zinc inhibits glutamate release via activation of pre‐synaptic KATP channels and reduces ischaemic damage in rat hippocampus

Zinc inhibits glutamate release via activation of pre‐synaptic K ATP channels and reduces ischaemic damage in rat hippocampus

Synaptotagmin 1 is required for vesicular Ca 2+ /H + ‐antiport activity

Presynaptic K(+) channels, vesicular Ca(2+)/H (+) antiport--synaptotagmin, and acetylcholinesterase, three mechanisms cutting short the cholinergic signal at neuromuscular and nerve-electroplaque junctions

Presynaptic K+ Channels, Vesicular Ca2+/H+ Antiport—Synaptotagmin, and Acetylcholinesterase, Three Mechanisms Cutting Short the Cholinergic Signal at Neuromuscular and Nerve–Electroplaque Junctions

Acetylcholine release in rapid synapses: two fast partners--mediatophore and vesicular Ca2+/H+ antiport

Nicotine-induced and depolarisation-induced glutamate release from hippocampus mossy fibre synaptosomes: two distinct mechanisms

Acetylcholine release and the cholinergic genomic locus

Acetylcholine synthesis and quantal release reconstituted by transfection of mediatophore and choline acetyltranferase cDNAs

Synaptic vesicles control the time course of neurotransmitter secretion via a Ca 2+ /H + antiport

Exocytosis, Mediatophore, and Vesicular Ca super(2+)/H super(+) Antiport in Rapid Neurotransmission

Ultra-Fast Versus Sustained Cholinergic Transmission: A Variety of Different Mechanisms

Exocytosis, Mediatophore, and Vesicular Ca 2+ /H + Antiport in Rapid Neurotransmission

An improved approach to freeze-fracture morphology of monolayer cell cultures

Quantal acetylcholine release: Vesicle fusion or intramembrane particles?

Zinc‐induced changes in ionic currents of clonal rat pancreatic β‐cells: activation of ATP‐sensitive K+ channels

Tumor Necrosis Factor-{alpha} and Angiostatin Are Mediators of Endothelial Cytotoxicity in Bronchoalveolar Lavages of Patients with Acute Respiratory Distress Syndrome

Tumor necrosis factor-α and angiostatin are mediators of endothelial cytotoxicity in bronchoalveolar lavages of patients with acute respiratory distress syndrome

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