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The Multicenter Cancer of Pancreas Screening Study: Impact on Stage and Survival

Journal of clinical oncology, 2022-10, Vol.40 (28), p.3257-3266

2022

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Prospective study of germline genetic testing in incident cases of pancreatic adenocarcinoma

Cancer, 2018-09, Vol.124 (17), p.3520-3527

2018

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Clinicopathological comparison of colorectal and endometrial carcinomas in patients with Lynch-like syndrome versus patients with Lynch syndrome

Human pathology, 2015-11, Vol.46 (11), p.1616-1625

2015

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rxCOV is a quantitative metric for assessing immunoassay analyte fidelity

Scientific reports, 2023-01, Vol.13 (1), p.88-88, Article 88

2023

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DNA mismatch repair protein deficient non-neoplastic colonic crypts: a novel indicator of Lynch syndrome

Modern pathology, 2018-10, Vol.31 (10), p.1608-1618

2018

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Immune microenvironment profiling of normal appearing colorectal mucosa biopsied over repeat patient visits reproducibly separates lynch syndrome patients based on their history of colon cancer

Frontiers in oncology, 2023-08, Vol.13, p.1174831-1174831

2023

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The Effect of Pancreatic Juice Collection Time on the Detection of KRAS Mutations

Pancreas, 2018-01, Vol.47 (1), p.35-39

2018

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Neoadjuvant therapy in microsatellite-stable colorectal carcinoma induces concomitant loss of MSH6 and Ki-67 expression

Human pathology, 2017-05, Vol.63, p.33-39

2017

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Detection of DNA mismatch repair deficient crypts in random colonoscopic biopsies identifies Lynch syndrome patients

Familial cancer, 2020-04, Vol.19 (2), p.169-175

2020

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Lynch syndrome–associated colorectal carcinoma: frequent involvement of the left colon and rectum and late-onset presentation supports a universal screening approach

Human pathology, 2013-11, Vol.44 (11), p.2518-2528

2013

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Mo1153 OUTCOMES OF GASTRIC CANCER SURVEILLANCE IN LYNCH SYNDROME

Gastroenterology (New York, N.Y. 1943), 2023-05, Vol.164 (6), p.S-777-S-777

2023

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896: THE GUT MICROBIOME PREDICTS EARLY-ONSET COLORECTAL CANCER BASED ON KNOWN RISK FACTORS

Gastroenterology (New York, N.Y. 1943), 2022-05, Vol.162 (7), p.S-220-S-220

2022

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829 – The Identification of Dna Mismatch Repair Deficiency in Nonneoplastic Crypts by Colonoscopic Biopsy in Lynch Syndrome Patients

Gastroenterology (New York, N.Y. 1943), 2019-05, Vol.156 (6), p.S-179-S-179

2019

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Pancreatic Cancer Surveillance and Novel Strategies for Screening

Gastrointestinal endoscopy clinics of North America, 2022-01, Vol.32 (1), p.13-25

2022

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559: IMMUNE MICROENVIRONMENT PROFILING OF NORMAL APPEARING COLORECTAL TISSUES SEPARATES LYNCH SYNDROME PATIENTS ALONG THEIR COLON CANCER STATUS

Gastroenterology (New York, N.Y. 1943), 2022-05, Vol.162 (7), p.S-133-S-134

2022

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Mo1737 - Accuracy of Physician Elicited Family History in Predicting Genetic Susceptibility to Pancreatic Ductal Adenocarcinoma

Gastroenterology (New York, N.Y. 1943), 2018-05, Vol.154 (6), p.S-790-S-790

2018

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Tu1125 IMMUNE MICROENVIRONMENT PROFILING OF NORMAL APPEARING COLORECTAL TISSUES REPRODUCIABLY IDENTIFIES LYNCH SYNDROME PATIENTS BY THEIR COLON CANCER STATUS ACROSS SEQUENTIAL COLONOSCOPIES

Gastroenterology (New York, N.Y. 1943), 2023-05, Vol.164 (6), p.S-987-S-987

2023

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DNA mismatch repair‐deficient non‐neoplastic endometrial glands are common in Lynch syndrome patients and are present at a higher density than in the colon

Histopathology, 2021-10, Vol.79 (4), p.573-583

2021

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Germline MLH1 Mutations Are Frequently Identified in Lynch Syndrome Patients With Colorectal and Endometrial Carcinoma Demonstrating Isolated Loss of PMS2 Immunohistochemical Expression

The American journal of surgical pathology, 2015-08, Vol.39 (8), p.1114-1120

2015

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Su1174 CHARACTERIZATION OF IMMUNE MICROENVIRONMENT IN LYNCH SYNDROME PATIENTS

Gastroenterology (New York, N.Y. 1943), 2020-05, Vol.158 (6), p.S-532-S-532

2020

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MLH1-deficient Colorectal Carcinoma With Wild-type BRAF and MLH1 Promoter Hypermethylation Harbor KRAS Mutations and Arise From Conventional Adenomas

The American journal of surgical pathology, 2016-10, Vol.40 (10), p.1390-1399

2016

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Germline mutation prevalence in individuals with pancreatic cancer and a history of previous malignancy

Cancer, 2018-04, Vol.124 (8), p.1691-1700

2018

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Recommendations for a More Organized and Effective Approach to the Early Detection of Pancreatic Cancer From the PRECEDE (Pancreatic Cancer Early Detection) Consortium

Gastroenterology (New York, N.Y. 1943), 2021-12, Vol.161 (6), p.1751-1757

2021

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Family communication and patient distress after germline genetic testing in individuals with pancreatic ductal adenocarcinoma

Cancer, 2019-07, Vol.125 (14), p.2488-2496

2019

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The Multicenter Cancer of Pancreas Screening Study: Impact on Stage and Survival

Prospective study of germline genetic testing in incident cases of pancreatic adenocarcinoma

Clinicopathological comparison of colorectal and endometrial carcinomas in patients with Lynch-like syndrome versus patients with Lynch syndrome

rxCOV is a quantitative metric for assessing immunoassay analyte fidelity

DNA mismatch repair protein deficient non-neoplastic colonic crypts: a novel indicator of Lynch syndrome

Immune microenvironment profiling of normal appearing colorectal mucosa biopsied over repeat patient visits reproducibly separates lynch syndrome patients based on their history of colon cancer

The Effect of Pancreatic Juice Collection Time on the Detection of KRAS Mutations

Neoadjuvant therapy in microsatellite-stable colorectal carcinoma induces concomitant loss of MSH6 and Ki-67 expression

Detection of DNA mismatch repair deficient crypts in random colonoscopic biopsies identifies Lynch syndrome patients

Lynch syndrome–associated colorectal carcinoma: frequent involvement of the left colon and rectum and late-onset presentation supports a universal screening approach

Mo1153 OUTCOMES OF GASTRIC CANCER SURVEILLANCE IN LYNCH SYNDROME

896: THE GUT MICROBIOME PREDICTS EARLY-ONSET COLORECTAL CANCER BASED ON KNOWN RISK FACTORS

829 – The Identification of Dna Mismatch Repair Deficiency in Nonneoplastic Crypts by Colonoscopic Biopsy in Lynch Syndrome Patients

Pancreatic Cancer Surveillance and Novel Strategies for Screening

559: IMMUNE MICROENVIRONMENT PROFILING OF NORMAL APPEARING COLORECTAL TISSUES SEPARATES LYNCH SYNDROME PATIENTS ALONG THEIR COLON CANCER STATUS

Mo1737 - Accuracy of Physician Elicited Family History in Predicting Genetic Susceptibility to Pancreatic Ductal Adenocarcinoma

Tu1125 IMMUNE MICROENVIRONMENT PROFILING OF NORMAL APPEARING COLORECTAL TISSUES REPRODUCIABLY IDENTIFIES LYNCH SYNDROME PATIENTS BY THEIR COLON CANCER STATUS ACROSS SEQUENTIAL COLONOSCOPIES

DNA mismatch repair‐deficient non‐neoplastic endometrial glands are common in Lynch syndrome patients and are present at a higher density than in the colon

Germline MLH1 Mutations Are Frequently Identified in Lynch Syndrome Patients With Colorectal and Endometrial Carcinoma Demonstrating Isolated Loss of PMS2 Immunohistochemical Expression

Su1174 CHARACTERIZATION OF IMMUNE MICROENVIRONMENT IN LYNCH SYNDROME PATIENTS

MLH1-deficient Colorectal Carcinoma With Wild-type BRAF and MLH1 Promoter Hypermethylation Harbor KRAS Mutations and Arise From Conventional Adenomas

Germline mutation prevalence in individuals with pancreatic cancer and a history of previous malignancy

The role of family history in predicting germline pathogenic variant carriers who develop pancreatic cancer: Results of a multicenter collaboration

Recommendations for a More Organized and Effective Approach to the Early Detection of Pancreatic Cancer From the PRECEDE (Pancreatic Cancer Early Detection) Consortium

Family communication and patient distress after germline genetic testing in individuals with pancreatic ductal adenocarcinoma

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