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High-mobility group box 1 potentiates antineutrophil cytoplasmic antibody-inducing neutrophil extracellular traps formation

Arthritis research & therapy, 2016-01, Vol.18 (2), p.2-2, Article 2

2016

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Predictors of Treatment Resistance and Relapse in Antineutrophil Cytoplasmic Antibody–Associated Vasculitis: A Study of 439 Cases in a Single Chinese Center

Arthritis & rheumatology (Hoboken, N.J.), 2014-07, Vol.66 (7), p.1920-1926

2014

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Glutathione Peroxidase 4 Is a Predictor of Diabetic Kidney Disease Progression in Type 2 Diabetes Mellitus

Oxidative medicine and cellular longevity, 2022-10, Vol.2022, p.1-10

2022

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Association between serum uric acid level and mortality in China

Chinese medical journal, 2021-09, Vol.134 (17), p.2073-2080

2021

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Dapagliflozin Ameliorates Diabetic Kidney Disease via Upregulating Crry and Alleviating Complement Over-activation in db/db Mice

Frontiers in pharmacology, 2021-10, Vol.12, p.729334-729334

2021

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Clinical and pathological characteristics of DKD patients with early-onset type 2 diabetes

Journal of diabetes and its complications, 2023-08, Vol.37 (8), p.108520-108520, Article 108520

2023

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High mobility group box-1 contributes to anti-myeloperoxidase antibody-induced glomerular endothelial cell injury through a moesin-dependent route

Arthritis research & therapy, 2017-06, Vol.19 (1), p.125-125, Article 125

2017

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HMGB1 contributes to glomerular endothelial cell injury in ANCA‐associated vasculitis through enhancing endothelium–neutrophil interactions

Journal of cellular and molecular medicine, 2017-07, Vol.21 (7), p.1351-1360

2017

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C3c deposition predicts worse renal outcomes in patients with biopsy‐proven diabetic kidney disease in type 2 diabetes mellitus

Journal of diabetes, 2022-04, Vol.14 (4), p.291-297

2022

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Unstably controlled systolic blood pressure trajectories are associated with markers for kidney damage in prediabetic population: results from the INDEED cohort study

Journal of translational medicine, 2020-05, Vol.18 (1), p.194-194, Article 194

2020

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Urinary levels of high mobility group box-1 are associated with disease activity in antineutrophil cytoplasmic autoantibody-associated vasculitis

PloS one, 2015-04, Vol.10 (4), p.e0123586-e0123586

2015

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Clinical and Pathological Characteristics of Patients With Nonproteinuric Diabetic Nephropathy

Frontiers in endocrinology (Lausanne), 2021-10, Vol.12, p.761386-761386

2021

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Deficiency of C3a receptor attenuates the development of diabetic nephropathy

BMJ open diabetes research & care, 2019-11, Vol.7 (1), p.e000817-e000817

2019

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Association of Circulating Level of High Mobility Group Box 1 With Disease Activity in Antineutrophil Cytoplasmic Autoantibody–Associated Vasculitis

Arthritis care & research (2010), 2013-11, Vol.65 (11), p.1828-1834

2013

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Persistent hematuria in patients with antineutrophil cytoplasmic antibody-associated vasculitis during clinical remission: chronic glomerular lesion or low-grade active renal vasculitis?

BMC nephrology, 2017-12, Vol.18 (1), p.354-354, Article 354

2017

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Association of HLA Genes with Clinical Outcomes of ANCA-Associated Vasculitis

Clinical journal of the American Society of Nephrology, 2012-08, Vol.7 (8), p.1293-1299

2012

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Dapagliflozin Alleviates Diabetic Kidney Disease via Hypoxia Inducible Factor 1α/Heme Oxygenase 1-Mediated Ferroptosis

Antioxidants & redox signaling, 2024-03, Vol.40 (7-9), p.492-509

2024

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Myeloperoxidase-ANCA-positive granulomatosis with polyangiitis is a distinct subset of ANCA-associated vasculitis: A retrospective analysis of 455 patients from a single center in China

Seminars in arthritis and rheumatism, 2019-02, Vol.48 (4), p.701-706

2019

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Annexin A1 alleviates kidney injury by promoting the resolution of inflammation in diabetic nephropathy

Kidney international, 2021-07, Vol.100 (1), p.107-121

2021

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Associations of urinary epidermal growth factor and monocyte chemotactic protein-1 with kidney involvement in patients with diabetic kidney disease

Nephrology, dialysis, transplantation, 2020-02, Vol.35 (2), p.291-297

2020

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Predictors for mortality in patients with antineutrophil cytoplasmic autoantibody-associated vasculitis: a study of 398 Chinese patients

Journal of rheumatology, 2014-09, Vol.41 (9), p.1849-1855

2014

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The BVAS is an independent predictor of cardiovascular events and cardiovascular disease-related mortality in patients with ANCA-associated vasculitis: A study of 504 cases in a single Chinese center

Seminars in arthritis and rheumatism, 2018-02, Vol.47 (4), p.524-529

2018

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Effect of total alkaloids from Commelina communis L. on lung damage by influenza virus infection

Microbiology and immunology, 2010-12, Vol.54 (12), p.754-757

2010

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High mobility group box 1 contributes to anti-neutrophil cytoplasmic antibody-induced neutrophils activation through receptor for advanced glycation end products (RAGE) and Toll-like receptor 4

Arthritis research & therapy, 2015-03, Vol.17 (1), p.64-64, Article 64

2015

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HMGB 1 contributes to glomerular endothelial cell injury in ANCA ‐associated vasculitis through enhancing endothelium–neutrophil interactions

Journal of cellular and molecular medicine, 2017-07, Vol.21 (7), p.1351-1360

2017

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High-mobility group box 1 potentiates antineutrophil cytoplasmic antibody-inducing neutrophil extracellular traps formation

Predictors of Treatment Resistance and Relapse in Antineutrophil Cytoplasmic Antibody–Associated Vasculitis: A Study of 439 Cases in a Single Chinese Center

Glutathione Peroxidase 4 Is a Predictor of Diabetic Kidney Disease Progression in Type 2 Diabetes Mellitus

Association between serum uric acid level and mortality in China

Dapagliflozin Ameliorates Diabetic Kidney Disease via Upregulating Crry and Alleviating Complement Over-activation in db/db Mice

Clinical and pathological characteristics of DKD patients with early-onset type 2 diabetes

High mobility group box-1 contributes to anti-myeloperoxidase antibody-induced glomerular endothelial cell injury through a moesin-dependent route

HMGB1 contributes to glomerular endothelial cell injury in ANCA‐associated vasculitis through enhancing endothelium–neutrophil interactions

C3c deposition predicts worse renal outcomes in patients with biopsy‐proven diabetic kidney disease in type 2 diabetes mellitus

Unstably controlled systolic blood pressure trajectories are associated with markers for kidney damage in prediabetic population: results from the INDEED cohort study

Urinary levels of high mobility group box-1 are associated with disease activity in antineutrophil cytoplasmic autoantibody-associated vasculitis

Clinical and Pathological Characteristics of Patients With Nonproteinuric Diabetic Nephropathy

Deficiency of C3a receptor attenuates the development of diabetic nephropathy

Association of Circulating Level of High Mobility Group Box 1 With Disease Activity in Antineutrophil Cytoplasmic Autoantibody–Associated Vasculitis

Persistent hematuria in patients with antineutrophil cytoplasmic antibody-associated vasculitis during clinical remission: chronic glomerular lesion or low-grade active renal vasculitis?

Association of HLA Genes with Clinical Outcomes of ANCA-Associated Vasculitis

Dapagliflozin Alleviates Diabetic Kidney Disease via Hypoxia Inducible Factor 1α/Heme Oxygenase 1-Mediated Ferroptosis

Myeloperoxidase-ANCA-positive granulomatosis with polyangiitis is a distinct subset of ANCA-associated vasculitis: A retrospective analysis of 455 patients from a single center in China

Annexin A1 alleviates kidney injury by promoting the resolution of inflammation in diabetic nephropathy

Associations of urinary epidermal growth factor and monocyte chemotactic protein-1 with kidney involvement in patients with diabetic kidney disease

Predictors for mortality in patients with antineutrophil cytoplasmic autoantibody-associated vasculitis: a study of 398 Chinese patients

The BVAS is an independent predictor of cardiovascular events and cardiovascular disease-related mortality in patients with ANCA-associated vasculitis: A study of 504 cases in a single Chinese center

Effect of total alkaloids from Commelina communis L. on lung damage by influenza virus infection

High mobility group box 1 contributes to anti-neutrophil cytoplasmic antibody-induced neutrophils activation through receptor for advanced glycation end products (RAGE) and Toll-like receptor 4

HMGB 1 contributes to glomerular endothelial cell injury in ANCA ‐associated vasculitis through enhancing endothelium–neutrophil interactions

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