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Copy number polymorphism in Fcgr3 predisposes to glomerulonephritis in rats and humans

Nature, 2006-02, Vol.439 (7078), p.851-855

2006

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inositol 5-phosphatase SHIP2 regulates endocytic clathrin-coated pit dynamics

The Journal of cell biology, 2010-08, Vol.190 (3), p.307-315

2010

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Requirement for Class II Phosphoinositide 3-Kinase C2α in Maintenance of Glomerular Structure and Function

Molecular and Cellular Biology, 2011-01, Vol.31 (1), p.63-80

2011

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Regulation of Neuron Survival through an Intersectin-Phosphoinositide 3′-Kinase C2β-AKT Pathway

Molecular and Cellular Biology, 2007-11, Vol.27 (22), p.7906-7917

2007

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Recruitment of the Class II Phosphoinositide 3-Kinase C2β to the Epidermal Growth Factor Receptor: Role of Grb2

Molecular and Cellular Biology, 2001-10, Vol.21 (19), p.6660-6667

2001

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Haptoglobin Genotype-Dependent Anti-Inflammatory Signaling in CD163+ Macrophages

International journal of inflammation, 2013-01, Vol.2013, p.980327-7

2013

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Hyperglycemia Causes Renal Cell Damage via CCN2-Induced Activation of the TrkA Receptor

Diabetes (New York, N.Y.), 2012-09, Vol.61 (9), p.2280-2288

2012

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Predictive Value of PTEN and AR Coexpression of Sustained Responsiveness to Hormonal Therapy in Prostate Cancer—A Pilot Study

Neoplasia (New York, N.Y.), 2008-09, Vol.10 (9), p.949-953

2008

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Phosphorylation of Both EGFR and ErbB2 Is a Reliable Predictor of Prostate Cancer Cell Proliferation in Response to EGF

Neoplasia (New York, N.Y.), 2004-11, Vol.6 (6), p.846-853

2004

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Class II Phosphoinositide 3-Kinases Are Downstream Targets of Activated Polypeptide Growth Factor Receptors

Molecular and Cellular Biology, 2000-06, Vol.20 (11), p.3817-3830

2000

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Androgen-Independent Prostate Cancer: Potential Role of Androgen and ErbB Receptor Signal Transduction Crosstalk

Neoplasia (New York, N.Y.), 2003-03, Vol.5 (2), p.99-109

2003

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Overexpression of a rat kinase-deficient phosphoinositide 3-kinase, Vps34p, inhibits cathepsin D maturation

Biochemical journal, 2001-02, Vol.353 (Pt 3), p.655-661

2001

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Insulin-feedback via PI3K-C2α activated PKBα/Akt1 is required for glucose-stimulated insulin secretion

The FASEB journal, 2010-06, Vol.24 (6), p.1824-1837

2010

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Elevated Akt Phosphorylation as an Indicator of Renal Tubular Epithelial Cell Stress

The Journal of biological chemistry, 2002-09, Vol.277 (37), p.33943-33949

2002

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The N-terminus of phosphoinositide 3-kinase-C2β regulates lipid kinase activity and binding to clathrin

Journal of cellular physiology, 2006-03, Vol.206 (3), p.586-593

2006

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The Class II Phosphoinositide 3-Kinase C2α Is Activated by Clathrin and Regulates Clathrin-Mediated Membrane Trafficking

Molecular cell, 2001-02, Vol.7 (2), p.443-449

2001

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Ca2+-regulated Pool of Phosphatidylinositol-3-phosphate Produced by Phosphatidylinositol 3-Kinase C2α on Neurosecretory Vesicles

Molecular biology of the cell, 2008-12, Vol.19 (12), p.5593-5603

2008

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Hyperglycemia Causes Renal Cell Damage via CCN2-Induced Activation of the TrkA Receptor: Implications for Diabetic Nephropathy

Diabetes (New York, N.Y.), 2012-09, Vol.61 (9), p.2280-2288

2012

Volltextzugriff (PDF)

Phosphatidylinositol 3-Kinase C2α Is Essential for ATP-dependent Priming of Neurosecretory Granule Exocytosis

Molecular biology of the cell, 2005-10, Vol.16 (10), p.4841-4851

2005

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Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility

Nature genetics, 2008-05, Vol.40 (5), p.553-559

2008

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Mannose receptor interacts with Fc receptors and is critical for the development of crescentic glomerulonephritis in mice

The Journal of clinical investigation, 2010-05, Vol.120 (5), p.1469-1478

2010

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The class II phosphoinositide 3-kinase PI3K-C2β regulates cell migration by a PtdIns(3)P dependent mechanism

Journal of cellular physiology, 2005-12, Vol.205 (3), p.452-462

2005

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The N-terminus of phosphoinositide 3-kinase-C2beta regulates lipid kinase activity and binding to clathrin

Journal of cellular physiology, 2006-03, Vol.206 (3), p.586-593

2006

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The Class II Phosphoinositide 3-Kinase PI3K-C2α Is Concentrated in the Trans-Golgi Network and Present in Clathrin-coated Vesicles

The Journal of biological chemistry, 2000-04, Vol.275 (16), p.11943-11950

2000

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Epidermal growth factor stimulates translocation of the class II phosphoinositide 3-kinase PI3K-C2β to the nucleus

Biochemical journal, 2009-08, Vol.422 (1), p.53-60

2009

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Copy number polymorphism in Fcgr3 predisposes to glomerulonephritis in rats and humans

inositol 5-phosphatase SHIP2 regulates endocytic clathrin-coated pit dynamics

Requirement for Class II Phosphoinositide 3-Kinase C2α in Maintenance of Glomerular Structure and Function

Regulation of Neuron Survival through an Intersectin-Phosphoinositide 3′-Kinase C2β-AKT Pathway

Recruitment of the Class II Phosphoinositide 3-Kinase C2β to the Epidermal Growth Factor Receptor: Role of Grb2

Haptoglobin Genotype-Dependent Anti-Inflammatory Signaling in CD163+ Macrophages

Hyperglycemia Causes Renal Cell Damage via CCN2-Induced Activation of the TrkA Receptor

Predictive Value of PTEN and AR Coexpression of Sustained Responsiveness to Hormonal Therapy in Prostate Cancer—A Pilot Study

Phosphorylation of Both EGFR and ErbB2 Is a Reliable Predictor of Prostate Cancer Cell Proliferation in Response to EGF

Class II Phosphoinositide 3-Kinases Are Downstream Targets of Activated Polypeptide Growth Factor Receptors

Androgen-Independent Prostate Cancer: Potential Role of Androgen and ErbB Receptor Signal Transduction Crosstalk

Overexpression of a rat kinase-deficient phosphoinositide 3-kinase, Vps34p, inhibits cathepsin D maturation

Insulin-feedback via PI3K-C2α activated PKBα/Akt1 is required for glucose-stimulated insulin secretion

Elevated Akt Phosphorylation as an Indicator of Renal Tubular Epithelial Cell Stress

The N-terminus of phosphoinositide 3-kinase-C2β regulates lipid kinase activity and binding to clathrin

The Class II Phosphoinositide 3-Kinase C2α Is Activated by Clathrin and Regulates Clathrin-Mediated Membrane Trafficking

Ca2+-regulated Pool of Phosphatidylinositol-3-phosphate Produced by Phosphatidylinositol 3-Kinase C2α on Neurosecretory Vesicles

Hyperglycemia Causes Renal Cell Damage via CCN2-Induced Activation of the TrkA Receptor: Implications for Diabetic Nephropathy

Phosphatidylinositol 3-Kinase C2α Is Essential for ATP-dependent Priming of Neurosecretory Granule Exocytosis

Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility

Mannose receptor interacts with Fc receptors and is critical for the development of crescentic glomerulonephritis in mice

The class II phosphoinositide 3-kinase PI3K-C2β regulates cell migration by a PtdIns(3)P dependent mechanism

The N-terminus of phosphoinositide 3-kinase-C2beta regulates lipid kinase activity and binding to clathrin

The Class II Phosphoinositide 3-Kinase PI3K-C2α Is Concentrated in the Trans-Golgi Network and Present in Clathrin-coated Vesicles

Epidermal growth factor stimulates translocation of the class II phosphoinositide 3-kinase PI3K-C2β to the nucleus

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