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Notch3 is a major regulator of vascular tone in cerebral and tail resistance arteries

Arteriosclerosis, thrombosis, and vascular biology, 2008-12, Vol.28 (12), p.2216

2008

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Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia

Nature (London), 1996-10, Vol.383 (6602), p.707-710

1996

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Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel disease

The Journal of clinical investigation, 2010-02, Vol.120 (2), p.433-445

2010

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Splice site mutation causing a seven amino acid Notch3 in-frame deletion in CADASIL

Neurology, 2000-05, Vol.54 (9), p.1874

2000

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The ectodomain of the Notch3 receptor accumulates within the cerebrovasculature of CADASIL patients

The Journal of clinical investigation, 2000-03, Vol.105 (5), p.597-605

2000

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J022 Effecteurs du récepteur Notch3 dans la cellule musculaire lisse des petites artères

Archives of cardiovascular diseases, 2009, Vol.102, p.S110-S110

2009

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Skin biopsy immunostaining with a Notch3 monoclonal antibody for CADASIL diagnosis

The Lancet (British edition), 2001-12, Vol.358 (9298), p.2049-2051

2001

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Lésions de la substance blanche dans la maladie Cadasil : une altération de l’homéostasie ionique et hydrique ?

Revue neurologique, 2014-04, Vol.170, p.A81-A81

2014

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A Human Homolog of Bacterial Acetolactate Synthase Genes Maps within the CADASIL Critical Region

Genomics (San Diego, Calif.), 1996-12, Vol.38 (2), p.192-198

1996

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Krit1/cerebral cavernous malformation 1 mRNA is preferentially expressed in neurons and epithelial cells in embryo and adult

Mechanisms of development, 2002-09, Vol.117 (1), p.363-367

2002

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Notch3 Mutations in Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL), a Mendelian Condition Causing Stroke and Vascular Dementia

Annals of the New York Academy of Sciences, 1997-09, Vol.826 (1), p.213-217

1997

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CADASIL: genetics and physiopathology

Bulletin de l'Académie nationale de médecine, 2000, Vol.184 (7), p.1535-42; discussion 1542-4

2000

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Notch3 is a major regulator of vascular tone in cerebral and tail resistance arteries

Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia

Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel disease

Splice site mutation causing a seven amino acid Notch3 in-frame deletion in CADASIL

The ectodomain of the Notch3 receptor accumulates within the cerebrovasculature of CADASIL patients

J022 Effecteurs du récepteur Notch3 dans la cellule musculaire lisse des petites artères

Skin biopsy immunostaining with a Notch3 monoclonal antibody for CADASIL diagnosis

Lésions de la substance blanche dans la maladie Cadasil : une altération de l’homéostasie ionique et hydrique ?

A Human Homolog of Bacterial Acetolactate Synthase Genes Maps within the CADASIL Critical Region

Krit1/cerebral cavernous malformation 1 mRNA is preferentially expressed in neurons and epithelial cells in embryo and adult

Notch3 Mutations in Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL), a Mendelian Condition Causing Stroke and Vascular Dementia

CADASIL: genetics and physiopathology

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