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The structure and mode of action of different botulinum toxins

European journal of neurology, 2006-12, Vol.13 (s4), p.1-9

2006

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Porphyrin derivatives as potent and selective blockers of neuronal Kv1 channels

Chemical communications (Cambridge, England), 2015-01, Vol.51 (6), p.1066-1069

2015

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Innocuous full-length botulinum neurotoxin targets and promotes the expression of lentiviral vectors in central and autonomic neurons

Gene therapy, 2011-07, Vol.18 (7), p.656-665

2011

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Functional Repair of Motor Endplates after Botulinum Neurotoxin Type A Poisoning: Biphasic Switch of Synaptic Activity between Nerve Sprouts and their Parent Terminals

Proceedings of the National Academy of Sciences - PNAS, 1999-03, Vol.96 (6), p.3200-3205

1999

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Inactivation properties of voltage-gated K + channels altered by presence of β-subunit

Nature (London), 1994-05, Vol.369 (6478), p.289-294

1994

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Glial heterogeneity in expression of the inwardly rectifying K+ channel, Kir4.1, in adult rat CNS

Glia, 2000-06, Vol.30 (4), p.362-372

2000

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Primary Structure of a β Subunit of α-Dendrotoxin-Sensitive K+Channels from Bovine Brain

Proceedings of the National Academy of Sciences - PNAS, 1994-03, Vol.91 (5), p.1637-1641

1994

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Predominant expression of Kv1.3 voltage-gated K+ channel subunit in rat prostate cancer cell lines: electrophysiological, pharmacological and molecular characterisation

Pflügers Archiv, 2003-08, Vol.446 (5), p.559-571

2003

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Evaluation of the therapeutic usefulness of botulinum neurotoxin B, C1, E, and F compared with the long lasting type A. Basis for distinct durations of inhibition of exocytosis in central neurons

The Journal of biological chemistry, 2003-01, Vol.278 (2), p.1363-1371

2003

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A role for the interchain disulfide or its participating thiols in the internalization of botulinum neurotoxin A revealed by a toxin derivative that binds to ecto-acceptors and inhibits transmitter release intracellularly

The Journal of biological chemistry, 1993-10, Vol.268 (28), p.20838-20844

1993

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Central Action of Dendrotoxin: Selective Reduction of a Transient K Conductance in Hippocampus and Binding to Localized Acceptors

Proceedings of the National Academy of Sciences - PNAS, 1986-01, Vol.83 (2), p.493-497

1986

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Subunit Composition of Kv1 Channels in Human CNS

Journal of neurochemistry, 1999-08, Vol.73 (2), p.849-858

1999

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Oligomeric properties of .alpha.-dendrotoxin-sensitive potassium ion channels purified from bovine brain

Biochemistry (Easton), 1992-11, Vol.31 (45), p.11084-11088

1992

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Importance of Two Adjacent C-terminal Sequences of SNAP-25 in Exocytosis from Intact and Permeabilized Chromaffin Cells Revealed by Inhibition with Botulinum Neurotoxins A and E

Biochemistry (Easton), 1997-03, Vol.36 (11), p.3061-3067

1997

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Tetanus toxin and botulinum toxins type A and B inhibit glutamate, gamma-aminobutyric acid, aspartate, and met-enkephalin release from synaptosomes. Clues to the locus of action

The Journal of biological chemistry, 1992-10, Vol.267 (30), p.21338-21343

1992

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Botulinum A Like Type B and Tetanus Toxins Fulfils Criteria for Being a Zinc‐Dependent Protease

Journal of neurochemistry, 1993-12, Vol.61 (6), p.2338-2341

1993

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Dendrotoxin acceptor from bovine synaptic plasma membranes. Binding properties, purification and subunit composition of a putative constituent of certain voltage-activated K+ channels

Biochemical journal, 1989-02, Vol.257 (3), p.899-903

1989

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Prominent location of A K+ channel containing the α subunit Kv 1.2 in the basket cell nerve terminals of rat cerebellum

Neuroscience, 1993-12, Vol.57 (4), p.1039-1045

1993

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Productive and non-productive binding of botulinum neurotoxin A to motor nerve endings are distinguished by its heavy chain

Journal of neuroscience research, 1996-05, Vol.44 (3), p.263-271

1996

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Production of seizures and brain damage in rats by α-dendrotoxin, a selective K + channel blocker

Neuroscience letters, 1992-05, Vol.139 (1), p.34-40

1992

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Evidence for the Induction of Repetitive Action Potentials in Synaptosomes by K+‐Channel Inhibitors: An Analysis of Plasma Membrane Ion Fluxes

Journal of neurochemistry, 1996-07, Vol.67 (1), p.389-397

1996

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Mast cell degranulating peptide and dendrotoxin selectively inhibit a fast-activating potassium current and bind to common neuronal proteins

Neuroscience, 1987-12, Vol.23 (3), p.893-902

1987

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Episodic Ataxia Type-1 Mutations in the Kv1.1 Potassium Channel Display Distinct Folding and Intracellular Trafficking Properties

The Journal of biological chemistry, 2001-12, Vol.276 (52), p.49427-49434

2001

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Botulinum Neurotoxin C1 Cleaves both Syntaxin and SNAP-25 in Intact and Permeabilized Chromaffin Cells: Correlation with Its Blockade of Catecholamine Release

Biochemistry (Easton), 1996-02, Vol.35 (8), p.2630-2636

1996

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Protein Kinase B Stimulates the Translocation of GLUT4 but Not GLUT1 or Transferrin Receptors in 3T3-L1 Adipocytes by a Pathway Involving SNAP-23, Synaptobrevin-2, and/or Cellubrevin

The Journal of biological chemistry, 1999-10, Vol.274 (40), p.28087-28095

1999

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The structure and mode of action of different botulinum toxins

Porphyrin derivatives as potent and selective blockers of neuronal Kv1 channels

Innocuous full-length botulinum neurotoxin targets and promotes the expression of lentiviral vectors in central and autonomic neurons

Functional Repair of Motor Endplates after Botulinum Neurotoxin Type A Poisoning: Biphasic Switch of Synaptic Activity between Nerve Sprouts and their Parent Terminals

Inactivation properties of voltage-gated K + channels altered by presence of β-subunit

Glial heterogeneity in expression of the inwardly rectifying K+ channel, Kir4.1, in adult rat CNS

Primary Structure of a β Subunit of α-Dendrotoxin-Sensitive K+Channels from Bovine Brain

Predominant expression of Kv1.3 voltage-gated K+ channel subunit in rat prostate cancer cell lines: electrophysiological, pharmacological and molecular characterisation

Evaluation of the therapeutic usefulness of botulinum neurotoxin B, C1, E, and F compared with the long lasting type A. Basis for distinct durations of inhibition of exocytosis in central neurons

A role for the interchain disulfide or its participating thiols in the internalization of botulinum neurotoxin A revealed by a toxin derivative that binds to ecto-acceptors and inhibits transmitter release intracellularly

Central Action of Dendrotoxin: Selective Reduction of a Transient K Conductance in Hippocampus and Binding to Localized Acceptors

Subunit Composition of Kv1 Channels in Human CNS

Oligomeric properties of .alpha.-dendrotoxin-sensitive potassium ion channels purified from bovine brain

Importance of Two Adjacent C-terminal Sequences of SNAP-25 in Exocytosis from Intact and Permeabilized Chromaffin Cells Revealed by Inhibition with Botulinum Neurotoxins A and E

Tetanus toxin and botulinum toxins type A and B inhibit glutamate, gamma-aminobutyric acid, aspartate, and met-enkephalin release from synaptosomes. Clues to the locus of action

Botulinum A Like Type B and Tetanus Toxins Fulfils Criteria for Being a Zinc‐Dependent Protease

Dendrotoxin acceptor from bovine synaptic plasma membranes. Binding properties, purification and subunit composition of a putative constituent of certain voltage-activated K+ channels

Prominent location of A K+ channel containing the α subunit Kv 1.2 in the basket cell nerve terminals of rat cerebellum

Productive and non-productive binding of botulinum neurotoxin A to motor nerve endings are distinguished by its heavy chain

Production of seizures and brain damage in rats by α-dendrotoxin, a selective K + channel blocker

Evidence for the Induction of Repetitive Action Potentials in Synaptosomes by K+‐Channel Inhibitors: An Analysis of Plasma Membrane Ion Fluxes

Mast cell degranulating peptide and dendrotoxin selectively inhibit a fast-activating potassium current and bind to common neuronal proteins

Episodic Ataxia Type-1 Mutations in the Kv1.1 Potassium Channel Display Distinct Folding and Intracellular Trafficking Properties

Botulinum Neurotoxin C1 Cleaves both Syntaxin and SNAP-25 in Intact and Permeabilized Chromaffin Cells: Correlation with Its Blockade of Catecholamine Release

Protein Kinase B Stimulates the Translocation of GLUT4 but Not GLUT1 or Transferrin Receptors in 3T3-L1 Adipocytes by a Pathway Involving SNAP-23, Synaptobrevin-2, and/or Cellubrevin

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