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Fatty acid binding protein 4 regulates pancreatic cancer cell proliferation via activation of nuclear factor E2-related factor 2

Surgery for obesity and related diseases, 2022-04, Vol.18 (4), p.485-493

2022

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Novel Bifunctional Natriuretic Peptides as Potential Therapeutics

The Journal of biological chemistry, 2008-12, Vol.283 (50), p.35003-35009

2008

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A Familial Mutation Renders Atrial Natriuretic Peptide Resistant to Proteolytic Degradation

The Journal of biological chemistry, 2009-07, Vol.284 (29), p.19196-19202

2009

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A human skeletal overgrowth mutation increases maximal velocity and blocks desensitization of guanylyl cyclase-B

Bone (New York, N.Y.), 2013-10, Vol.56 (2), p.375-382

2013

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Guanylyl cyclase (GC)-A and GC-B activities in ventricles and cardiomyocytes from failed and non-failed human hearts: GC-A is inactive in the failed cardiomyocyte

Journal of molecular and cellular cardiology, 2012-03, Vol.52 (3), p.727-732

2012

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A functional screen provides evidence for a conserved, regulatory, juxtamembrane phosphorylation site in guanylyl cyclase a and B

PloS one, 2012-05, Vol.7 (5), p.e36747

2012

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Natriuretic Peptides, Their Receptors, and Cyclic Guanosine Monophosphate-Dependent Signaling Functions

Endocrine reviews, 2006-02, Vol.27 (1), p.47-72

2006

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Antibody tracking demonstrates cell type-specific and ligand-independent internalization of guanylyl cyclase a and natriuretic peptide receptor C

Molecular pharmacology, 2011-07, Vol.80 (1), p.155-162

2011

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Renal hyporesponsiveness to atrial natriuretic peptide in congestive heart failure results from reduced atrial natriuretic peptide receptor concentrations

American journal of physiology. Renal physiology, 2007-05, Vol.292 (5), p.F1636-F1644

2007

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Biodesign of a renal-protective peptide based on alternative splicing of B-type natriuretic peptide

Proceedings of the National Academy of Sciences - PNAS, 2009-07, Vol.106 (27), p.11282-11287

2009

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Natriuretic peptides: their structures, receptors, physiologic functions and therapeutic applications

Handbook of experimental pharmacology, 2009 (191), p.341

2009

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Guanylyl cyclase‐A phosphorylation decreases cardiac hypertrophy and improves systolic function in male, but not female, mice

The FASEB journal, 2022-01, Vol.36 (1), p.e22069-n/a

2022

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Dendroaspis natriuretic peptide and the designer natriuretic peptide, CD-NP, are resistant to proteolytic inactivation

Journal of molecular and cellular cardiology, 2011-07, Vol.51 (1), p.67-71

2011

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Novel enhancers of guanylyl cyclase‐A activity acting via allosteric modulation

British journal of pharmacology, 2023-12, Vol.180 (24), p.3254-3270

2023

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Lipid peroxidation products induce carbonyl stress, mitochondrial dysfunction, and cellular senescence in human and murine cells

Aging cell, 2024-10, p.e14367

2024

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Regulation of the Natriuretic Peptide Receptor 2 (Npr2) by Phosphorylation of Juxtamembrane Serine and Threonine Residues Is Essential for Bifurcation of Sensory Axons

The Journal of neuroscience, 2018-11, Vol.38 (45), p.9768-9780

2018

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Skeletal overgrowth-causing mutations mimic an allosterically activated conformation of guanylyl cyclase-B that is inhibited by 2,4,6,-trinitrophenyl ATP

The Journal of biological chemistry, 2017-06, Vol.292 (24), p.10220-10229

2017

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A Glutamate-Substituted Mutant Mimics the Phosphorylated and Active Form of Guanylyl Cyclase-A

Molecular pharmacology, 2017-07, Vol.92 (1), p.67-74

2017

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Dephosphorylation of juxtamembrane serines and threonines of the NPR2 guanylyl cyclase is required for rapid resumption of oocyte meiosis in response to luteinizing hormone

Developmental biology, 2016-01, Vol.409 (1), p.194-201

2016

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ProBNP(1-108) is resistant to degradation and activates guanylyl cyclase-A with reduced potency

Clinical chemistry (Baltimore, Md.), 2011-09, Vol.57 (9), p.1272-1278

2011

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Catalytically Active Guanylyl Cyclase B Requires Endoplasmic Reticulum-mediated Glycosylation, and Mutations That Inhibit This Process Cause Dwarfism

The Journal of biological chemistry, 2016-05, Vol.291 (21), p.11385-11393

2016

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Insulin-degrading Enzyme Modulates the Natriuretic Peptide-mediated Signaling Response

The Journal of biological chemistry, 2011-02, Vol.286 (6), p.4670-4679

2011

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Differential Regulation of Membrane Guanylyl Cyclases in Congestive Heart Failure: Natriuretic Peptide Receptor (NPR)-B, Not NPR-A, Is the Predominant Natriuretic Peptide Receptor in the Failing Heart

Endocrinology (Philadelphia), 2007-07, Vol.148 (7), p.3518-3522

2007

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Human B-type natriuretic peptide is not degraded by meprin A

Biochemical pharmacology, 2010-10, Vol.80 (7), p.1007-1011

2010

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ProBNP1-108 Is Resistant to Degradation and Activates Guanylyl Cyclase-A with Reduced Potency

Clinical chemistry (Baltimore, Md.), 2011-09, Vol.57 (9), p.1272-1278

2011

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Fatty acid binding protein 4 regulates pancreatic cancer cell proliferation via activation of nuclear factor E2-related factor 2

Novel Bifunctional Natriuretic Peptides as Potential Therapeutics

A Familial Mutation Renders Atrial Natriuretic Peptide Resistant to Proteolytic Degradation

A human skeletal overgrowth mutation increases maximal velocity and blocks desensitization of guanylyl cyclase-B

Guanylyl cyclase (GC)-A and GC-B activities in ventricles and cardiomyocytes from failed and non-failed human hearts: GC-A is inactive in the failed cardiomyocyte

A functional screen provides evidence for a conserved, regulatory, juxtamembrane phosphorylation site in guanylyl cyclase a and B

Natriuretic Peptides, Their Receptors, and Cyclic Guanosine Monophosphate-Dependent Signaling Functions

Antibody tracking demonstrates cell type-specific and ligand-independent internalization of guanylyl cyclase a and natriuretic peptide receptor C

Renal hyporesponsiveness to atrial natriuretic peptide in congestive heart failure results from reduced atrial natriuretic peptide receptor concentrations

Biodesign of a renal-protective peptide based on alternative splicing of B-type natriuretic peptide

Natriuretic peptides: their structures, receptors, physiologic functions and therapeutic applications

Guanylyl cyclase‐A phosphorylation decreases cardiac hypertrophy and improves systolic function in male, but not female, mice

Dendroaspis natriuretic peptide and the designer natriuretic peptide, CD-NP, are resistant to proteolytic inactivation

Novel enhancers of guanylyl cyclase‐A activity acting via allosteric modulation

Lipid peroxidation products induce carbonyl stress, mitochondrial dysfunction, and cellular senescence in human and murine cells

Regulation of the Natriuretic Peptide Receptor 2 (Npr2) by Phosphorylation of Juxtamembrane Serine and Threonine Residues Is Essential for Bifurcation of Sensory Axons

Skeletal overgrowth-causing mutations mimic an allosterically activated conformation of guanylyl cyclase-B that is inhibited by 2,4,6,-trinitrophenyl ATP

A Glutamate-Substituted Mutant Mimics the Phosphorylated and Active Form of Guanylyl Cyclase-A

Dephosphorylation of juxtamembrane serines and threonines of the NPR2 guanylyl cyclase is required for rapid resumption of oocyte meiosis in response to luteinizing hormone

ProBNP(1-108) is resistant to degradation and activates guanylyl cyclase-A with reduced potency

Catalytically Active Guanylyl Cyclase B Requires Endoplasmic Reticulum-mediated Glycosylation, and Mutations That Inhibit This Process Cause Dwarfism

Insulin-degrading Enzyme Modulates the Natriuretic Peptide-mediated Signaling Response

Differential Regulation of Membrane Guanylyl Cyclases in Congestive Heart Failure: Natriuretic Peptide Receptor (NPR)-B, Not NPR-A, Is the Predominant Natriuretic Peptide Receptor in the Failing Heart

Human B-type natriuretic peptide is not degraded by meprin A

ProBNP1-108 Is Resistant to Degradation and Activates Guanylyl Cyclase-A with Reduced Potency

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