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The pore-forming subunit MCU of the mitochondrial Ca2+ uniporter is required for normal glucose-stimulated insulin secretion in vitro and in vivo in mice

Diabetologia, 2020-07, Vol.63 (7), p.1368-1381

2020

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Gi/o protein-coupled receptor inhibition of beta-cell electrical excitability and insulin secretion depends on Na+/K+ ATPase activation

Nature communications, 2022-10, Vol.13 (1), p.6461-6461, Article 6461

2022

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TALK-1-mediated alterations of β-cell mitochondrial function and insulin secretion impair glucose homeostasis on a diabetogenic diet

Cell reports (Cambridge), 2024-01, Vol.43 (1), p.113673-113673, Article 113673

2024

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The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults

eLife, 2024-05, Vol.12

2024

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Osteopontin activates the diabetes-associated potassium channel TALK-1 in pancreatic β-cells

PloS one, 2017-04, Vol.12 (4), p.e0175069-e0175069

2017

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Glucose-mediated inhibition of calcium-activated potassium channels limits α-cell calcium influx and glucagon secretion

American journal of physiology: endocrinology and metabolism, 2019-04, Vol.316 (4), p.E646-E659

2019

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Selective Small Molecule Activators of TREK‑2 Channels Stimulate Dorsal Root Ganglion c‑Fiber Nociceptor Two-Pore-Domain Potassium Channel Currents and Limit Calcium Influx

ACS chemical neuroscience, 2017-03, Vol.8 (3), p.558-568

2017

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A KCNK16 mutation causing TALK-1 gain of function is associated with maturity-onset diabetes of the young

JCI insight, 2021-07, Vol.6 (13)

2021

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TALK-1 reduces delta-cell endoplasmic reticulum and cytoplasmic calcium levels limiting somatostatin secretion

Molecular metabolism (Germany), 2018-03, Vol.9, p.84-97

2018

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Lactate activation of α-cell KATP channels inhibits glucagon secretion by hyperpolarizing the membrane potential and reducing Ca2+ entry

Molecular metabolism (Germany), 2020-12, Vol.42, p.101056-101056, Article 101056

2020

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Neuronal L-Type Calcium Channel Signaling to the Nucleus Requires a Novel CaMKIIα-Shank3 Interaction

The Journal of neuroscience, 2020-03, Vol.40 (10), p.2000-2014

2020

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Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca2+-Regulated Genes

Diabetes (New York, N.Y.), 2017-08, Vol.66 (8), p.2175-2187

2017

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Glucose-Dependent miR-125b Is a Negative Regulator of β-Cell Function

Diabetes (New York, N.Y.), 2022-07, Vol.71 (7), p.1525-1545

2022

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Tetraspanin‐7 regulation of L‐type voltage‐dependent calcium channels controls pancreatic β‐cell insulin secretion

The Journal of physiology, 2020-11, Vol.598 (21), p.4887-4905

2020

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Liraglutide increases islet Ca2+ oscillation frequency and insulin secretion by activating hyperpolarization‐activated cyclic nucleotide‐gated channels

Diabetes, obesity & metabolism, 2022-09, Vol.24 (9), p.1741-1752

2022

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TRPM7 is a crucial regulator of pancreatic endocrine development and high-fat-diet-induced β-cell proliferation

Development (Cambridge), 2021-08, Vol.148 (16)

2021

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Liraglutide increases islet Ca 2+ oscillation frequency and insulin secretion by activating hyperpolarization-activated cyclic nucleotide-gated channels

Diabetes, obesity & metabolism, 2022-09, Vol.24 (9), p.1741-1752

2022

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Cytokine-mediated changes in K+ channel activity promotes an adaptive Ca2+ response that sustains β-cell insulin secretion during inflammation

Scientific reports, 2018-01, Vol.8 (1), p.1-15, Article 1158

2018

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TALK-1 channels control β cell endoplasmic reticulum Ca 2+ homeostasis

Science signaling, 2017-09, Vol.10 (497)

2017

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Fibronectin Binding to the Treponema pallidum Adhesin Protein Fragment rTp0483 on Functionalized Self-Assembled Monolayers

Bioconjugate chemistry, 2012-02, Vol.23 (2), p.184-195

2012

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Preparing recombinant "Split AEP" for protein labeling

Methods in enzymology, 2023, Vol.690, p.501-540

2023

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2-Point site Voronoi diagrams

Discrete Applied Mathematics, 2002-10, Vol.122 (1), p.37-54

2002

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The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults

eLife, 2024-05, Vol.12

2024

Volltextzugriff (PDF)

Osteopontin activates the diabetes-associated potassium channel TALK-1 in pancreatic [beta]-cells

PloS one, 2017-04, Vol.12 (4), p.e0175069

2017

Volltextzugriff (PDF)

G i/o protein-coupled receptor inhibition of beta-cell electrical excitability and insulin secretion depends on Na + /K + ATPase activation

Nature communications, 2022-10, Vol.13 (1), p.6461

2022

Volltextzugriff (PDF)

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The pore-forming subunit MCU of the mitochondrial Ca2+ uniporter is required for normal glucose-stimulated insulin secretion in vitro and in vivo in mice

Gi/o protein-coupled receptor inhibition of beta-cell electrical excitability and insulin secretion depends on Na+/K+ ATPase activation

TALK-1-mediated alterations of β-cell mitochondrial function and insulin secretion impair glucose homeostasis on a diabetogenic diet

The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults

Osteopontin activates the diabetes-associated potassium channel TALK-1 in pancreatic β-cells

Glucose-mediated inhibition of calcium-activated potassium channels limits α-cell calcium influx and glucagon secretion

Selective Small Molecule Activators of TREK‑2 Channels Stimulate Dorsal Root Ganglion c‑Fiber Nociceptor Two-Pore-Domain Potassium Channel Currents and Limit Calcium Influx

A KCNK16 mutation causing TALK-1 gain of function is associated with maturity-onset diabetes of the young

TALK-1 reduces delta-cell endoplasmic reticulum and cytoplasmic calcium levels limiting somatostatin secretion

Lactate activation of α-cell KATP channels inhibits glucagon secretion by hyperpolarizing the membrane potential and reducing Ca2+ entry

Neuronal L-Type Calcium Channel Signaling to the Nucleus Requires a Novel CaMKIIα-Shank3 Interaction

Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca2+-Regulated Genes

Glucose-Dependent miR-125b Is a Negative Regulator of β-Cell Function

Tetraspanin‐7 regulation of L‐type voltage‐dependent calcium channels controls pancreatic β‐cell insulin secretion

Liraglutide increases islet Ca2+ oscillation frequency and insulin secretion by activating hyperpolarization‐activated cyclic nucleotide‐gated channels

TRPM7 is a crucial regulator of pancreatic endocrine development and high-fat-diet-induced β-cell proliferation

Liraglutide increases islet Ca 2+ oscillation frequency and insulin secretion by activating hyperpolarization-activated cyclic nucleotide-gated channels

Cytokine-mediated changes in K+ channel activity promotes an adaptive Ca2+ response that sustains β-cell insulin secretion during inflammation

TALK-1 channels control β cell endoplasmic reticulum Ca 2+ homeostasis

Fibronectin Binding to the Treponema pallidum Adhesin Protein Fragment rTp0483 on Functionalized Self-Assembled Monolayers

Preparing recombinant "Split AEP" for protein labeling

2-Point site Voronoi diagrams

The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults

Osteopontin activates the diabetes-associated potassium channel TALK-1 in pancreatic [beta]-cells

G i/o protein-coupled receptor inhibition of beta-cell electrical excitability and insulin secretion depends on Na + /K + ATPase activation

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